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Introduction Ischemic cell death associated with myocardial infarction (MI) prompts the recruitment and activation of immune cells to ensure repair ( Epelman & Mann 2012 , Frangogiannis 2012 ). Neutrophils are recruited early, removing
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Institute of Genetic Medicine, Newcastle University, Newcastle upon Tyne, United Kingdom
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(RCL) that is characteristic of the SERPIN structure ( Gettins & Olson 2016 ). While CBG is not known to inhibit proteases, its RCL is cleaved by neutrophil elastase (NE) ( Hammond et al. 1990 ), chymotrypsin ( Lewis & Elder 2014 ) and Pseudomonas
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mice ( Gabel et al . 2005 ). Neutrophils invade the myocardium during reperfusion and are an important source of oxidant stress causing injury ( Reimer et al . 1989 ). In vivo , the inhibition of myocardial adhesion molecule expression by oestrogen
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following intra-peritoneal injection of thioglycollate (0.2 mL, 10%) were collected by lavage with 5mL ice-cold PBS ( Coutinho et al . 2016 ). At 24 h, cells comprise mainly neutrophils and monocytes, whereas the population is predominantly macrophages at
The National Institute of Biotechnology in the Negev (NIBN), Ben-Gurion University, Beer-Sheva, Israel
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The National Institute of Biotechnology in the Negev (NIBN), Ben-Gurion University, Beer-Sheva, Israel
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The National Institute of Biotechnology in the Negev (NIBN), Ben-Gurion University, Beer-Sheva, Israel
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. 2010 ). In mouse models amenable to detailed time-course analyses, even short-term (3 days) high-fat feeding (HFF) induced neutrophil-dominated adipose tissue inflammation. This inflammatory response clearly occurred before the development of obesity
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counts. Three hundred cells per mouse were counted to determine the number of neutrophils. Histopathology of the lungs The left lung was fixed in 6% v/v paraformaldehyde at a constant hydrostatic pressure of 25 cm H 2 O for 24 h and embedded in
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adhesion molecule (ICAM)-2 regulates angiogenesis . Blood 106 1636 – 1643 . ( doi:10.1182/blood-2004-12-4716 ) Huang MT Larbi KY Scheiermann C Woodfin A Gerwin N Haskard DO Nourshargh S 2006 ICAM-2 mediates neutrophil transmigration
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now known that several endogenous proteases, including neutrophil elastase ( Hammond et al. 1990 ) and chymotrypsin ( Lewis & Elder 2014 ), and a protease (LasB) produced by the opportunistic bacterial pathogen, Pseudomonas aeruginosa ( Simard et
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. In contrast, chemerin treatment reduces the recruitment of neutrophil and macrophages to the inflammatory sites and the expression of proinflammatory cytokine ( Cash et al . 2008 , Luangsay et al . 2009 ). These studies suggest that chemerin acts
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State Key Laboratory of Marine Pollution (SKLMP) at City University of Hong Kong, Hong Kong SAR, People’s Republic of China
Department of Materials Science and Engineering, College of Science and Engineering, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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and neutrophil chemotaxis were upregulated in estrogen-treated males. Among the genes whose expression level in injured hearts were sexually dimorphic, and/or reciprocally regulated by E2 and tamoxifen, were interferon-gamma ( ifn-γ ), interferon