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Jay H Lo Department of Molecular and Cell Biology, University of Connecticut, 91 N. Eagleville Road, U-3125, Storrs, Connecticut 06269, USA

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Pinwen Peter Chiou Department of Molecular and Cell Biology, University of Connecticut, 91 N. Eagleville Road, U-3125, Storrs, Connecticut 06269, USA

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C M Lin Department of Molecular and Cell Biology, University of Connecticut, 91 N. Eagleville Road, U-3125, Storrs, Connecticut 06269, USA

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Thomas T Chen Department of Molecular and Cell Biology, University of Connecticut, 91 N. Eagleville Road, U-3125, Storrs, Connecticut 06269, USA

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factors in liver function and development. Part II: the C/EBPs and D site-binding protein in cell cycle control, carcinogenesis, circadian gene regulation, liver regeneration, apoptosis, and liver-specific gene regulation. Pharmacological Reviews 56

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0085
Volume/Issue:
Volume 194: Issue 2
Free access
Pia Kiilerich Institute of Biology and
Department of Biochemistry and Molecular Biology, University of Southern Denmark, Odense, Campusvej 55, DK-5230 Odense M, Denmark

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Karsten Kristiansen Institute of Biology and
Department of Biochemistry and Molecular Biology, University of Southern Denmark, Odense, Campusvej 55, DK-5230 Odense M, Denmark

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Steffen S Madsen Institute of Biology and
Department of Biochemistry and Molecular Biology, University of Southern Denmark, Odense, Campusvej 55, DK-5230 Odense M, Denmark

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requirement to investigate the signaling pathways of GR and MR in fish. The knowledge of osmoregulatory gene regulation by cortisol in fish gill is scarce and little is known about exact cortisol signaling mechanisms through GR and MR. This is the

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0185
Volume/Issue:
Volume 194: Issue 2
Free access
Nariaki Fujimoto Department of Developmental Biology, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Radiation Responses, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Xenobiotic Metabolism and Molecular Toxicology, Institute of Pharmaceutical Sciences, Hiroshima University School of Medicine, Kasumi 1-2-3, Minami-ku, Hiroshima 734-8551, Japan

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Yukimi Akimoto Department of Developmental Biology, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Radiation Responses, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Xenobiotic Metabolism and Molecular Toxicology, Institute of Pharmaceutical Sciences, Hiroshima University School of Medicine, Kasumi 1-2-3, Minami-ku, Hiroshima 734-8551, Japan

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Tomoharu Suzuki Department of Developmental Biology, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Radiation Responses, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Xenobiotic Metabolism and Molecular Toxicology, Institute of Pharmaceutical Sciences, Hiroshima University School of Medicine, Kasumi 1-2-3, Minami-ku, Hiroshima 734-8551, Japan

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Shigeyuki Kitamura Department of Developmental Biology, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Radiation Responses, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Xenobiotic Metabolism and Molecular Toxicology, Institute of Pharmaceutical Sciences, Hiroshima University School of Medicine, Kasumi 1-2-3, Minami-ku, Hiroshima 734-8551, Japan

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Shigeru Ohta Department of Developmental Biology, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Radiation Responses, Research Institute for Radiation Biology and Medicine, Hiroshima University, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8553, Japan
Department of Xenobiotic Metabolism and Molecular Toxicology, Institute of Pharmaceutical Sciences, Hiroshima University School of Medicine, Kasumi 1-2-3, Minami-ku, Hiroshima 734-8551, Japan

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has delineated the main mouse prostatic secretion pattern for the first time. The data will be useful for studying androgen-dependent gene regulation in the prostate, and may also provide markers for studying functional differentiation of prostate

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06733
Volume/Issue:
Volume 190: Issue 3
Free access
A M Solomon Department of Endocrinology, Royal Free Hospital and Medical School, Pond Street, London NW3 2PF, UK

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P M G Bouloux Department of Endocrinology, Royal Free Hospital and Medical School, Pond Street, London NW3 2PF, UK

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measure glucocorticoid-dependent transactivation and transrepression of gene regulation. Journal of Steroid Biochemistry and Molecular Biology 88 191 –201. Carel JC 2005 Growth hormone in Turner syndrome: twenty years

Article Type:
Review Article
DOI:
https://doi.org/10.1677/joe.1.06837
Volume/Issue:
Volume 191: Issue 2
Free access
Craig L Doig School of Clinical and Experimental Medicine, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Birmingham B15 2TT, UK

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Jamila Bashir School of Clinical and Experimental Medicine, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Birmingham B15 2TT, UK

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Agnieszka E Zielinska School of Clinical and Experimental Medicine, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Birmingham B15 2TT, UK

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Mark S Cooper School of Clinical and Experimental Medicine, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Birmingham B15 2TT, UK

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Paul M Stewart School of Clinical and Experimental Medicine, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Birmingham B15 2TT, UK

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Gareth G Lavery School of Clinical and Experimental Medicine, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Birmingham B15 2TT, UK

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muscle, adipocytes from p65 overexpression transgenic mice had elevated 11β-HSD1 at the mRNA and protein level, but show no evidence for direct gene regulation ( Lee et al . 2013 ). In this context, NF-κB was acting as a positive regulator in a model of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0494
Volume/Issue:
Volume 220: Issue 3
Open access
Marcelo A Christoffolete
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Márton Doleschall Human and Natural Sciences Center, Laboratory of Endocrine Neurobiology, Division of Endocrinology, Division of Endocrinology, Federal University of ABC, Santo Andre-SP 09210-370, Brazil

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Péter Egri Human and Natural Sciences Center, Laboratory of Endocrine Neurobiology, Division of Endocrinology, Division of Endocrinology, Federal University of ABC, Santo Andre-SP 09210-370, Brazil

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Zsolt Liposits Human and Natural Sciences Center, Laboratory of Endocrine Neurobiology, Division of Endocrinology, Division of Endocrinology, Federal University of ABC, Santo Andre-SP 09210-370, Brazil

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Ann Marie Zavacki Human and Natural Sciences Center, Laboratory of Endocrine Neurobiology, Division of Endocrinology, Division of Endocrinology, Federal University of ABC, Santo Andre-SP 09210-370, Brazil

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Antonio C Bianco Human and Natural Sciences Center, Laboratory of Endocrine Neurobiology, Division of Endocrinology, Division of Endocrinology, Federal University of ABC, Santo Andre-SP 09210-370, Brazil

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Balázs Gereben Human and Natural Sciences Center, Laboratory of Endocrine Neurobiology, Division of Endocrinology, Division of Endocrinology, Federal University of ABC, Santo Andre-SP 09210-370, Brazil

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and Development 9 1033 – 1045 . Wu Y Koenig RJ 2000 Gene regulation by thyroid hormone . Trends in Endocrinology and Metabolism 11 207 – 211 . Zavacki AM Lehmann JM Seol W Willson TM Kliewer SA Moore DD 1997 Activation

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0448
Volume/Issue:
Volume 205: Issue 2
Free access
K Alexander H Iwen Department of Internal Medicine I, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany

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Oezge Senyaman Department of Internal Medicine I, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany

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Arne Schwartz Department of Internal Medicine I, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany

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Maren Drenckhan Department of Internal Medicine I, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany

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Britta Meier Department of Internal Medicine I, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany

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Dirk Hadaschik Department of Internal Medicine I, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany

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Johannes Klein Department of Internal Medicine I, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany

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five independent experiments and a representative immunoblot is shown. Actin served as loading control. * P <0.05, ** P <0.01. p38 MAPK mediates UCP-1 induction by ACTH p38 MAPK pathways have been reported to mediate UCP-1 gene regulation. Using a

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0299
Volume/Issue:
Volume 196: Issue 3
Free access
Seiji Tsutsumi
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Xi Zhang
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Keiko Takata
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Kazuhiro Takahashi
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Richard H Karas Department of Obstetrics and Gynecology, Molecular Cardiology Research Institute, Yamagata University School of Medicine, 2-2-2 Iida-Nishi, Yamagata 990-9585, Japan

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Hirohisa Kurachi
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Michael E Mendelsohn Department of Obstetrics and Gynecology, Molecular Cardiology Research Institute, Yamagata University School of Medicine, 2-2-2 Iida-Nishi, Yamagata 990-9585, Japan

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between SRC-1 and CREB binding protein . Molecular and Cellular Biology 20 8720 – 8730 . Sanches R Nguyen D Rocha W White JH Mader S 2002 Diversity in the mechanisms of gene regulation by estrogen receptors . BioEssays 24 244 – 254

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Research Article
DOI:
https://doi.org/10.1677/JOE-07-0292
Volume/Issue:
Volume 199: Issue 2
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C E Waters
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A Stevens
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A White
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D W Ray
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peptide gene regulation (as in the ectopic ACTH syndrome) but also have relevance as a mechanism which could contribute to the malignant cell phenotype. Figure 1 Glucocorticoid regulation of the TAT3-luc reporter gene in COR L103 cells. (A) COR L

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.05804
Volume/Issue:
Volume 183: Issue 2
Free access
Matthias Haase Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Matthias Schott Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Stefan R Bornstein Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Ludwik K Malendowicz Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Werner A Scherbaum Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Holger S Willenberg Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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of SF-1, DAX1, and CYP17 in the human fetal adrenal gland: potential interactions in gene regulation. Molecular Endocrinology 15 57 –68. Ho MM & Vinson GP 1995 Endocrine control of the distribution of basic

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-06-0083
Volume/Issue:
Volume 192: Issue 2
Free access