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Juliano C da Silveira Department of Veterinary Medicine, Faculty of Animal Sciences and Food Engineering, University of Sao Paulo, Pirassununga, Sao Paulo, Brazil

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Ana Clara F C M de Ávila Department of Veterinary Medicine, Faculty of Animal Sciences and Food Engineering, University of Sao Paulo, Pirassununga, Sao Paulo, Brazil

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Hannah L Garrett Department of Biomedical Sciences, College of Veterinary and Biomedical Sciences, Animal Reproduction and Biotechnology Laboratory, Colorado State University, Fort Collins, Colorado, USA

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Jason E Bruemmer Department of Biomedical Sciences, College of Veterinary and Biomedical Sciences, Animal Reproduction and Biotechnology Laboratory, Colorado State University, Fort Collins, Colorado, USA

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Quinton A Winger Department of Biomedical Sciences, College of Veterinary and Biomedical Sciences, Animal Reproduction and Biotechnology Laboratory, Colorado State University, Fort Collins, Colorado, USA

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Gerrit J Bouma Department of Biomedical Sciences, College of Veterinary and Biomedical Sciences, Animal Reproduction and Biotechnology Laboratory, Colorado State University, Fort Collins, Colorado, USA

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identified that are highly conserved between species (e.g., Supplementary Table 1, see section on supplementary data given at the end of this article). MicroRNAs in gamete maturation Taking advantage of conditional gene targeting approaches in mice

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-17-0200
Volume/Issue:
Volume 236: Issue 1
Free access
Andrea Vasconsuelo Departamento de Biología, Bioquímica y Farmacia, Universidad Nacional del Sur, San Juan 670, 8000 Bahía Blanca, Argentina

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Lorena Milanesi Departamento de Biología, Bioquímica y Farmacia, Universidad Nacional del Sur, San Juan 670, 8000 Bahía Blanca, Argentina

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Ricardo Boland Departamento de Biología, Bioquímica y Farmacia, Universidad Nacional del Sur, San Juan 670, 8000 Bahía Blanca, Argentina

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New York Academy of Sciences 278 93 – 123 . Biswas G Guha M Avadhani NG 2005 Mitochondria-to-nucleus stress signaling in mammalian cells: nature of nuclear gene targets, transcription regulation, and induced resistance to

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0250
Volume/Issue:
Volume 196: Issue 2
Free access
Gordon Moody
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Pedro J Beltran
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Petia Mitchell
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Elaina Cajulis
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Young-Ah Chung
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David Hwang Oncology Research Therapeutic Area, David Geffen School of Medicine, USC Davis School of Gerontology, Amgen, Inc., One Amgen Center Drive, Thousand Oaks, California 91320, USA

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Richard Kendall
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Robert Radinsky
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Pinchas Cohen Oncology Research Therapeutic Area, David Geffen School of Medicine, USC Davis School of Gerontology, Amgen, Inc., One Amgen Center Drive, Thousand Oaks, California 91320, USA

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Frank J Calzone
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.2337/diabetes.50.5.1110 ) Yakar S Pennisi P Kim CH Zhao H Toyoshima Y Gavrilova O LeRoith D 2005 Studies involving the GH–IGF axis: lessons from IGF-I and IGF-I receptor gene targeting mouse models . Journal of Endocrinological Investigation

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0306
Volume/Issue:
Volume 221: Issue 1
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Jonathan M Mudry Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery

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Julie Massart Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery

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Ferenc L M Szekeres Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery

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Anna Krook Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery
Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery

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(C) TWIST1 ( n =5) or (D) TWIST2 ( n =6). Results are mean± s.e.m. of fold of control. *** P <0.001; ** P <0.01; * P <0.05. o.e., overexpression. mRNA levels of a number of gene targets of Twist were determined using RT-PCR. As expected from

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0474
Volume/Issue:
Volume 224: Issue 3
Free access
Hong-Wei Wang The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA

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Michelle Muguira The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA

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Wei-Dong Liu The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA

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Tao Zhang The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA

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Chiachen Chen The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA

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Rebecca Aucoin The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA

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Mary B Breslin The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA

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Michael S Lan The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA

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et al . 2005 ). In contrast to the positive regulation of insulin gene expression, negative regulation was not as frequently reported. Nkx2.2 is an islet-enriched transcription factor belonging to the NK2 family. Gene targeting experiments have shown

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0001
Volume/Issue:
Volume 198: Issue 1
Free access
Hong Lan Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Galya Vassileva Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Aaron Corona Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Li Liu Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Hana Baker Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Andrei Golovko Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Susan J Abbondanzo Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Weiwen Hu Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Shijun Yang Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Yun Ning Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Robert A Del Vecchio Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Frederique Poulet Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Maureen Laverty Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Eric L Gustafson Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Joseph A Hedrick Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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Timothy J Kowalski Cardiovascular and Metabolic Disease Research, Drug Safety,, Departments of

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and Methods Construction of targeting vector and generation of Gpr119 −/− mice A gene-targeting vector was constructed using the sequence from GenBank accession number NC_000086 . DNA fragments corresponding to the 5′ and 3′ regions of the Gpr119

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0453
Volume/Issue:
Volume 201: Issue 2
Free access
Marta Labeur Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Damian Refojo Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Barbara Wölfel Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Johanna Stalla Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Vivian Vargas Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Marily Theodoropoulou Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Michael Buchfelder Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Marcelo Paez-Pereda Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Eduardo Arzt Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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Günter K Stalla Department of Neuroendocrinology, Department of Inflammatory Disorders of the CNS at the Max Planck Institute of Psychiatry, Department of Neurosurgery, Affectis Pharmaceuticals, Laboratorio de Fisiología y Biología Molecular, Department of Molecular Neurogenetics

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recruitment of the complex together with other transcription factors to the Pomc promoter, which may elicit transcriptional repression of Pomc , remains to be elucidated. At present, the genes targeted by IFNG in AtT-20 cells remain unknown. Microarray or

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0011
Volume/Issue:
Volume 199: Issue 2
Free access
C D Simmons
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J M P Pabona
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Z Zeng
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M C Velarde
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D Gaddy
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F A Simmen
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R C M Simmen Department of Physiology and Biophysics, Arkansas Children's Nutrition Center, University of Arkansas for Medical Sciences, 4301 W Markham Street, Little Rock, Arkansas 72202, USA

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S Inokuchi K Yamashita K Yamamoto M 2003 Functional analysis of basic transcription element binding protein by gene targeting technology . Molecular and Cellular Biology 23 2489 – 2500 . Mutter GL Baak JPA Fitzgerald JT Gray R

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0474
Volume/Issue:
Volume 205: Issue 2
Free access
Noelia Martínez-Sánchez Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain
CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain

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José M Moreno-Navarrete CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
Department of Diabetes, Endocrinology and Nutrition, Hospital de Girona ‘Dr Josep Trueta’, Institut D’investigació Biomèdica de Girona (IdIBGi) and University of Girona, Girona, Spain

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Cristina Contreras Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain
CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain

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Eva Rial-Pensado Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain
CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain

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Johan Fernø Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain
Department of Clinical Science, KG Jebsen Center for Diabetes Research, University of Bergen, Bergen, Norway

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Rubén Nogueiras Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain
CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain

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Carlos Diéguez Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain
CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain

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José-Manuel Fernández-Real CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
Department of Diabetes, Endocrinology and Nutrition, Hospital de Girona ‘Dr Josep Trueta’, Institut D’investigació Biomèdica de Girona (IdIBGi) and University of Girona, Girona, Spain

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Miguel López Department of Physiology, CIMUS, University of Santiago de Compostela-Instituto de Investigación Sanitaria, Santiago de Compostela, Spain
CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain

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M 2015 Surgery-induced weight loss is associated with the down­regulation of genes targeted by microRNAs in adipose tissue . Journal of Clinical Endocrinology and Metabolism 100 E1467 – E1476 . ( doi:10.1210/jc.2015-2357 ) Pearce EN

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0425
Volume/Issue:
Volume 232: Issue 2
Open access
S Khan Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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D E W Livingstone Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK
Centre for Discovery Brain Science, University of Edinburgh, Hugh Robson Building, Edinburgh, UK

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A Zielinska College of Medical and Dental Sciences, University of Birmingham, Birmingham, UK

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C L Doig Department of Biosciences, School of Science & Technology, Nottingham Trent University, Nottingham, UK

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D F Cobice Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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C L Esteves Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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J T Y Man Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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N Z M Homer Mass Spectrometry Core Laboratory, Edinburgh Clinical Research Facility, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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J R Seckl Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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C L MacKay SIRCAMS, School of Chemistry, University of Edinburgh, Joseph Black Building, King's Buildings, Edinburgh, UK

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S P Webster Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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G G Lavery Department of Biosciences, School of Science & Technology, Nottingham Trent University, Nottingham, UK

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K E Chapman Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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B R Walker Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK
Clinical & Translational Research Institute, Newcastle University, International Centre for Life, Central Parkway, Newcastle upon Tyne, UK

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R Andrew Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK
Mass Spectrometry Core Laboratory, Edinburgh Clinical Research Facility, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK

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Cederquist C Rosen ED , 2013 Lessons on conditional gene targeting in mouse adipose tissue . Diabetes 62 864 – 874 . ( https://doi.org/10.2337/db12-1089 ) Liu J Wang L Zhang A Di W Zhang X Wu L Yu J Zha J Lv S Cheng P , 2011

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-23-0034
Volume/Issue:
Volume 258: Issue 3
Open access