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Jacob C Garza Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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Chung Sub Kim Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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Jing Liu Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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Wei Zhang Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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Xin-Yun Lu Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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PVN relevant to MC4R regulation of food intake is thyroid-releasing hormone (TRH) neurons. It has been shown that a large proportion of TRH neurons express MC4R ( Liu et al . 2003 ) and receive input from POMC and AgRP neurons ( Fekete et al . 2000

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0009
Volume/Issue:
Volume 197: Issue 3
Free access
D C Ferguson Department of Physiology and Pharmacology, College of Veterinary Medicine, University of Georgia, Athens, Georgia 30602, USA

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Z Caffall Department of Physiology and Pharmacology, College of Veterinary Medicine, University of Georgia, Athens, Georgia 30602, USA

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M Hoenig Department of Physiology and Pharmacology, College of Veterinary Medicine, University of Georgia, Athens, Georgia 30602, USA

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, albeit with a normal baseline TSH concentration ( Daminet et al. 2003 ). It has been suggested that measurement of the serum response of TSH to administered TRH would be useful to ascertain whether pituitary sensitivity is altered in obesity. However

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0064
Volume/Issue:
Volume 194: Issue 2
Free access
Juriaan R Metz Department Organismal Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1, 6525 ED Nijmegen, The Netherlands
Cell Biology and Immunology Group, Wageningen University, Marijkeweg 40, 6709 PG Wageningen, The Netherlands

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Mark O Huising Department Organismal Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1, 6525 ED Nijmegen, The Netherlands
Cell Biology and Immunology Group, Wageningen University, Marijkeweg 40, 6709 PG Wageningen, The Netherlands

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Karin Leon Department Organismal Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1, 6525 ED Nijmegen, The Netherlands
Cell Biology and Immunology Group, Wageningen University, Marijkeweg 40, 6709 PG Wageningen, The Netherlands

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B M Lidy Verburg-van Kemenade Department Organismal Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1, 6525 ED Nijmegen, The Netherlands
Cell Biology and Immunology Group, Wageningen University, Marijkeweg 40, 6709 PG Wageningen, The Netherlands

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Gert Flik Department Organismal Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1, 6525 ED Nijmegen, The Netherlands
Cell Biology and Immunology Group, Wageningen University, Marijkeweg 40, 6709 PG Wageningen, The Netherlands

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stimulated with 1×10 −7 M thyrotrophin-releasing hormone (TRH; Biotrend, Köln, Germany) and head kidneys were stimulated with 5×10 −8 M human ACTH (Sigma). Five- or fifteen-minute fractions were collected, immediately stored at −20 °C and analysed for α

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06640
Volume/Issue:
Volume 191: Issue 1
Free access
Pryscila D S Teixeira Departamento de Fisiologia e Biofisica, Instituto de Ciencias Biomedicas, Universidade de Sao Paulo, Sao Paulo, Brazil

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Angela M Ramos-Lobo Departamento de Fisiologia e Biofisica, Instituto de Ciencias Biomedicas, Universidade de Sao Paulo, Sao Paulo, Brazil

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Mariana Rosolen Tavares Departamento de Fisiologia e Biofisica, Instituto de Ciencias Biomedicas, Universidade de Sao Paulo, Sao Paulo, Brazil

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Frederick Wasinski Departamento de Fisiologia e Biofisica, Instituto de Ciencias Biomedicas, Universidade de Sao Paulo, Sao Paulo, Brazil

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Renata Frazao Departamento de Anatomia, Instituto de Ciencias Biomedicas, Universidade de Sao Paulo, Sao Paulo, Brazil

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Jose Donato Jr Departamento de Fisiologia e Biofisica, Instituto de Ciencias Biomedicas, Universidade de Sao Paulo, Sao Paulo, Brazil

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Expression Assay (Applied Biosystems) Trh tcgtgctaactggtatcccc cccaaatctcccctctcttc Analysis of innervation of ARH neurons To determine possible sexually dimorphic differences in the developmental timing of the innervation of ARH

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-20-0076
Volume/Issue:
Volume 249: Issue 3
Free access
S C P Dutra
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E G Moura
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A L Rodrigues
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P C Lisboa
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I Bonomo
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F P Toste
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M C F Passos
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that these animals had central leptin resistance and lower hypothalamic OB-Rb expression in adulthood ( Toste et al . 2006 b ). Leptin has a well-known stimulatory role upon thyroid function, increasing thyrotrophin-releasing hormone (TRH) and

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0366
Volume/Issue:
Volume 195: Issue 2
Free access
Marco Aurélio Liberato Costa da Veiga Laboratório de Endocrinologia Molecular, Departamento de Fisiologia e Farmacologia, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21949-900, Brazil

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Flávia Fonseca Bloise Laboratório de Endocrinologia Molecular, Departamento de Fisiologia e Farmacologia, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21949-900, Brazil

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Ricardo Henrique Costa-e-Sousa Laboratório de Endocrinologia Molecular, Departamento de Fisiologia e Farmacologia, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21949-900, Brazil

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Luana Lopes Souza Laboratório de Endocrinologia Molecular, Departamento de Fisiologia e Farmacologia, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21949-900, Brazil

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Norma Aparecida dos Santos Almeida Laboratório de Endocrinologia Molecular, Departamento de Fisiologia e Farmacologia, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21949-900, Brazil

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Karen Jesus Oliveira Laboratório de Endocrinologia Molecular, Departamento de Fisiologia e Farmacologia, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21949-900, Brazil
Laboratório de Endocrinologia Molecular, Departamento de Fisiologia e Farmacologia, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21949-900, Brazil

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Carmen Cabanelas Pazos-Moura Laboratório de Endocrinologia Molecular, Departamento de Fisiologia e Farmacologia, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Rio de Janeiro 21949-900, Brazil

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the reduction in thyrotropic axis would be consequent to hypothalamic action of Δ9-THC, resulting in decreased thyrotropin-releasing hormone (TRH) release, although so far, direct evidence is lacking. More recently, it was shown that the synthetic

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0380
Volume/Issue:
Volume 199: Issue 2
Free access
B Anguiano Instituto de Neurobiología, UNAM-Juriquilla, Querétaro, México 76230

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R Rojas-Huidobro Instituto de Neurobiología, UNAM-Juriquilla, Querétaro, México 76230

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G Delgado Instituto de Neurobiología, UNAM-Juriquilla, Querétaro, México 76230

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C Aceves Instituto de Neurobiología, UNAM-Juriquilla, Querétaro, México 76230

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alveolar epithelium ( Varas et al. 2002 ). It is also known that hyperthyroidism eliminates the PRL pulse by suppressing thyrotropin-releasing hormone (TRH) synthesis, which is an important agonist of pituitary PRL release in late pregnancy ( Van

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.05711
Volume/Issue:
Volume 183: Issue 2
Free access
Kylie S Foo Departments of, Neuroscience, Microbiology, Molecular Medicine and Surgery

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Hanna Brauner Departments of, Neuroscience, Microbiology, Molecular Medicine and Surgery

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Claes-Göran Östenson Departments of, Neuroscience, Microbiology, Molecular Medicine and Surgery

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Christian Broberger Departments of, Neuroscience, Microbiology, Molecular Medicine and Surgery

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1869 , Laguesse 1893 ), e.g. somatostatin in δ-cells ( Luft et al . 1974 ) and thyrotrophin-releasing hormone (TRH) in β-cells ( Kawano et al . 1983 ). In some cases, the functional output of signalling via these peptides converges to a common effect

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0254
Volume/Issue:
Volume 204: Issue 3
Free access
Alvaro Souto Padron Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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Ruy Andrade Louzada Neto Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil
Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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Thiago Urgal Pantaleão Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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Maria Carolina de Souza dos Santos Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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Renata Lopes Araujo Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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Bruno Moulin de Andrade Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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Monique da Silva Leandro Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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João Pedro Saar Werneck de Castro Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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Andrea Claudia Freitas Ferreira Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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Denise Pires de Carvalho Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil

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4 . NIS, TPO, DUOX1, DUOX2, NOX4, D1, TSHR, and TRH expressions As shown in Fig. 5 A, B and C, 3,5-T2 administration significantly reduced thyroid iodide uptake, TPO iodide oxidation activity, and thyroid D1 activity together with the significantly

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0502
Volume/Issue:
Volume 221: Issue 3
Open access
Michael Muchow
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Ioannis Bossis
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Tom E Porter
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-releasing hormone (TRH) stimulates secretion of TSH-like bioactivity from pituitaries cultured in vitro ( Scanes 1974 ), and increases plasma levels of thyroxine (T 4 ) in vivo ( Kuhn et al. 1988 ). In addition to TRH, corticotropin-releasing hormone (CRH) has

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.05944
Volume/Issue:
Volume 184: Issue 2
Free access