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Shaodong Guo Division of Molecular Cardiology, Department of Medicine, College of Medicine, Texas A&M University Health Science Center, Scott & White, Central Texas Veterans Health Care System, 1901 South 1st Street, Bldg. 205, Temple, Texas 76504, USA

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or α-cells; hyperglucagonemia; hyperglycemia Ins2-cre Talchai et al . (2012) Endothelium Irs1 −/− ∷ Irs2 −/− Reduced Akt and eNOS phosphorylation; impaired skeletal muscle glucose uptake; insulin resistance Tie2-cre Kubota et al . (2011

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-13-0327
Volume/Issue:
Volume 220: Issue 2
Free access
Shannon M Gifford Perinatal Research Laboratories, Department of Obstetrics and Gynecology, University of Wisconsin Madison, 7E Meriter Hospital/Park, 202 South Park Street, Madison, Wisconsin 53715, USA

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Fu-Xian Yi Perinatal Research Laboratories, Department of Obstetrics and Gynecology, University of Wisconsin Madison, 7E Meriter Hospital/Park, 202 South Park Street, Madison, Wisconsin 53715, USA

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Ian M Bird Perinatal Research Laboratories, Department of Obstetrics and Gynecology, University of Wisconsin Madison, 7E Meriter Hospital/Park, 202 South Park Street, Madison, Wisconsin 53715, USA

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( Baumgartner-Parzer & Waldhausl 2001 , Vallet & Wiel 2001 , Levi et al. 2002 ). In addition, the endothelium serves as a selective permeability barrier between the blood and tissue, including the brain ( Albert et al. 1997 ), and produces a variety of

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06773
Volume/Issue:
Volume 190: Issue 2
Free access
Lucy V Norling Barts and The London School of Medicine, The William Harvey Research Institute, Centre for Biochemical Pharmacology, Queen Mary University of London, Charterhouse Square, London EC1M 6BQ, UK

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Mauro Perretti Barts and The London School of Medicine, The William Harvey Research Institute, Centre for Biochemical Pharmacology, Queen Mary University of London, Charterhouse Square, London EC1M 6BQ, UK

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Dianne Cooper Barts and The London School of Medicine, The William Harvey Research Institute, Centre for Biochemical Pharmacology, Queen Mary University of London, Charterhouse Square, London EC1M 6BQ, UK

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( Flotte et al . 1983 ). Gal-3 expression has been found to be increased in neutrophils upon adhesion to the endothelium ( Gil et al . 2006 b ), which also coincided with a relocalisation of Gal-3 to the plasma membrane in endothelial cells ( Gil et al

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0512
Volume/Issue:
Volume 201: Issue 2
Free access
Morag J Young Cardiovascular Endocrinology, Department of Physiology, MIMR-PHI Institute, 27–31 Wright St, Clayton 3168, Australia
Cardiovascular Endocrinology, Department of Physiology, MIMR-PHI Institute, 27–31 Wright St, Clayton 3168, Australia

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Amanda J Rickard Cardiovascular Endocrinology, Department of Physiology, MIMR-PHI Institute, 27–31 Wright St, Clayton 3168, Australia
Cardiovascular Endocrinology, Department of Physiology, MIMR-PHI Institute, 27–31 Wright St, Clayton 3168, Australia

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numerous pathological conditions including atherosclerosis, inflammation and tissue remodelling. The endothelium expresses MR, GR and HSD2 (HSD11B2) all of which drive numerous physiological and pathological functions in the vascular wall. Clinical and

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-14-0471
Volume/Issue:
Volume 224: Issue 1
Free access
Marta Toral Departamento de Farmacología, Facultad de Farmacia, Granada, Spain

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Rosario Jimenez Departamento de Farmacología, Facultad de Farmacia, Granada, Spain
Instituto de Investigación Biosanitaria GRANADA, Hospitales Universitarios de Granada, Universidad de Granada, Granada, Spain

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Sebastián Montoro-Molina Departamento de Ciencias de la Salud, Universidad de Jaén, Jaén, Spain

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Miguel Romero Departamento de Farmacología, Facultad de Farmacia, Granada, Spain
Instituto de Investigación Biosanitaria GRANADA, Hospitales Universitarios de Granada, Universidad de Granada, Granada, Spain

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Rosemary Wangensteen Departamento de Ciencias de la Salud, Universidad de Jaén, Jaén, Spain

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Juan Duarte Departamento de Farmacología, Facultad de Farmacia, Granada, Spain
Instituto de Investigación Biosanitaria GRANADA, Hospitales Universitarios de Granada, Universidad de Granada, Granada, Spain

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Félix Vargas Instituto de Investigación Biosanitaria GRANADA, Hospitales Universitarios de Granada, Universidad de Granada, Granada, Spain
Departamento de Fisiología, Facultad de Medicina, Granada, Spain

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be rate limiting for NO synthesis. In addition, human umbilical vein endothelium also requires the activity of the cationic and neutral amino acid transport system y + L for NO synthesis ( Arancibia-Garavilla et al. 2003 ). After observing that L

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0229
Volume/Issue:
Volume 239: Issue 1
Free access
Yunshan Hu Australian Centre for Blood Diseases, Eastern Clinical Research Unit, Department of Diabetes and Endocrinology, Biotechnology Division, Monash University, 6th Floor Burnett Tower, 89 Commercial Road, Prahran, Melbourne, Victoria, Australia 3181
Australian Centre for Blood Diseases, Eastern Clinical Research Unit, Department of Diabetes and Endocrinology, Biotechnology Division, Monash University, 6th Floor Burnett Tower, 89 Commercial Road, Prahran, Melbourne, Victoria, Australia 3181

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Hong Bin Liu Australian Centre for Blood Diseases, Eastern Clinical Research Unit, Department of Diabetes and Endocrinology, Biotechnology Division, Monash University, 6th Floor Burnett Tower, 89 Commercial Road, Prahran, Melbourne, Victoria, Australia 3181
Australian Centre for Blood Diseases, Eastern Clinical Research Unit, Department of Diabetes and Endocrinology, Biotechnology Division, Monash University, 6th Floor Burnett Tower, 89 Commercial Road, Prahran, Melbourne, Victoria, Australia 3181

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Richard W Simpson Australian Centre for Blood Diseases, Eastern Clinical Research Unit, Department of Diabetes and Endocrinology, Biotechnology Division, Monash University, 6th Floor Burnett Tower, 89 Commercial Road, Prahran, Melbourne, Victoria, Australia 3181
Australian Centre for Blood Diseases, Eastern Clinical Research Unit, Department of Diabetes and Endocrinology, Biotechnology Division, Monash University, 6th Floor Burnett Tower, 89 Commercial Road, Prahran, Melbourne, Victoria, Australia 3181

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Anthony E Dear Australian Centre for Blood Diseases, Eastern Clinical Research Unit, Department of Diabetes and Endocrinology, Biotechnology Division, Monash University, 6th Floor Burnett Tower, 89 Commercial Road, Prahran, Melbourne, Victoria, Australia 3181
Australian Centre for Blood Diseases, Eastern Clinical Research Unit, Department of Diabetes and Endocrinology, Biotechnology Division, Monash University, 6th Floor Burnett Tower, 89 Commercial Road, Prahran, Melbourne, Victoria, Australia 3181

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( Bar-Tana 2001 ). PPARγ is expressed most abundantly in adipose tissue, pancreatic β cells, vascular endothelium and macrophages ( Dubois et al . 2000 , Willson et al . 2001 ). Some evidence suggests that the effects of TZDs are mediated independent

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-10-0393
Volume/Issue:
Volume 208: Issue 1
Free access
Ian M Bird Perinatal Research Laboratories, Department of Obstetrics and Gynecology, University of Wisconsin–Madison, 7E Meriter Hospital/Park, 202 South Park Street, Madison, Wisconsin 53715, USA

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. References Boeldt DS Yi FX Bird IM 2011 Pregnancy adaptive programming of capacitative entry responses alters nitric oxide (NO) output in vascular endothelium: new insights into eNOS regulation through adaptive cell signaling . Journal of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-11-0216
Volume/Issue:
Volume 210: Issue 3
Free access
Rong Wan Jiangsu Provincial Key Lab of Cardiovascular Diseases and Molecular Intervention, Department of Pharmacology, Nanjing Medical University, Nanjing 210029, People's Republic of China

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Chao Zhu Jiangsu Provincial Key Lab of Cardiovascular Diseases and Molecular Intervention, Department of Pharmacology, Nanjing Medical University, Nanjing 210029, People's Republic of China

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Rui Guo Jiangsu Provincial Key Lab of Cardiovascular Diseases and Molecular Intervention, Department of Pharmacology, Nanjing Medical University, Nanjing 210029, People's Republic of China

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Lai Jin Jiangsu Provincial Key Lab of Cardiovascular Diseases and Molecular Intervention, Department of Pharmacology, Nanjing Medical University, Nanjing 210029, People's Republic of China

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Yunxin Liu Jiangsu Provincial Key Lab of Cardiovascular Diseases and Molecular Intervention, Department of Pharmacology, Nanjing Medical University, Nanjing 210029, People's Republic of China

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Li Li Jiangsu Provincial Key Lab of Cardiovascular Diseases and Molecular Intervention, Department of Pharmacology, Nanjing Medical University, Nanjing 210029, People's Republic of China

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Hao Zhang Jiangsu Provincial Key Lab of Cardiovascular Diseases and Molecular Intervention, Department of Pharmacology, Nanjing Medical University, Nanjing 210029, People's Republic of China

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Shengnan Li Jiangsu Provincial Key Lab of Cardiovascular Diseases and Molecular Intervention, Department of Pharmacology, Nanjing Medical University, Nanjing 210029, People's Republic of China

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affinity than testosterone and 15- to 30-fold greater affinity than adrenal androgens for androgen receptor (AR; Ikeda et al . 2012 ). Reportedly, DHT enhanced the binding of monocytes to the endothelium via increased expression of vascular cell adhesion

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0138
Volume/Issue:
Volume 218: Issue 3
Free access
Hong-Yo Kang Graduate Institute of Clinical Medical Sciences, Hormone Research Center, Kaohsiung Chang Gung Memorial Hospital, Chang Gung University College of Medicine, Kaohsiung, Taiwan

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testosterone on vascular reactivity and inflammation. The majority of animal studies support the hypothesis that the actions of testosterone on vascular relaxation are both endothelium-dependent and -independent vasodilatory effects. Endothelial

Article Type:
Article Commentary
DOI:
https://doi.org/10.1530/JOE-13-0052
Volume/Issue:
Volume 217: Issue 3
Free access
Félix Vargas
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Isabel Rodríguez-Gómez
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Pablo Vargas-Tendero
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Eugenio Jimenez Departamento de Fisiología, Departamento de Bioquímica y Biología Molecular e Inmunología, Facultad de Medicina, Universidad de Granada, E-18012 Granada, Spain

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Mercedes Montiel Departamento de Fisiología, Departamento de Bioquímica y Biología Molecular e Inmunología, Facultad de Medicina, Universidad de Granada, E-18012 Granada, Spain

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system, by removing the C-terminal histidyl-leucine residue from AI. ACE is mainly located in the pulmonary vascular endothelium but has also been found in kidney, liver, brain, and cardiovascular tissues, among others. ACE activity is influenced by

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-11-0349
Volume/Issue:
Volume 213: Issue 1
Free access