Search Results
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Yanqiu Wang in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Zhou Jin in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Jiajun Sun in
Google Scholar
PubMed
Search for other papers by Xinxin Chen in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Pu Xie in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Yulin Zhou in
Google Scholar
PubMed
Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Key Laboratory for Endocrine Tumor, State Key Laboratory of Medical Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China
Search for other papers by Shu Wang in
Google Scholar
PubMed
, a CD14 + monocyte/macrophage-restricted receptor and an interferon-stimulated gene (ISG) that is upregulated by type I IFN, was increased in uGD patients at both the monocyte mRNA and serum levels. Inhibiting monocyte SIGLEC1 expression caused
Search for other papers by Farhana Naznin in
Google Scholar
PubMed
Division of Neurology, Department of Sports and Fitness, AMED-CREST, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, 5200 Kihara, Kiyotake, Miyazaki 889-1692, Japan
Search for other papers by Koji Toshinai in
Google Scholar
PubMed
Search for other papers by T M Zaved Waise in
Google Scholar
PubMed
Search for other papers by Cherl NamKoong in
Google Scholar
PubMed
Search for other papers by Abu Saleh Md Moin in
Google Scholar
PubMed
Search for other papers by Hideyuki Sakoda in
Google Scholar
PubMed
Division of Neurology, Department of Sports and Fitness, AMED-CREST, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, 5200 Kihara, Kiyotake, Miyazaki 889-1692, Japan
Search for other papers by Masamitsu Nakazato in
Google Scholar
PubMed
12 weeks. Expression of the ghrelin receptor in both the nodose ganglion and hypothalamus were downregulated in HFD-fed mice. We also investigated inflammation in the nodose ganglion and hypothalamus by performing immunohistochemistry of macrophages
Microbiology, Czech Academy of Sciences, Vídenská 1083, CZ-142 20 Prague 4, Czech Republic
Search for other papers by K Vagnerová in
Google Scholar
PubMed
Microbiology, Czech Academy of Sciences, Vídenská 1083, CZ-142 20 Prague 4, Czech Republic
Search for other papers by M Kverka in
Google Scholar
PubMed
Microbiology, Czech Academy of Sciences, Vídenská 1083, CZ-142 20 Prague 4, Czech Republic
Search for other papers by P Klusoňová in
Google Scholar
PubMed
Microbiology, Czech Academy of Sciences, Vídenská 1083, CZ-142 20 Prague 4, Czech Republic
Search for other papers by P Ergang in
Google Scholar
PubMed
Microbiology, Czech Academy of Sciences, Vídenská 1083, CZ-142 20 Prague 4, Czech Republic
Search for other papers by I Mikšík in
Google Scholar
PubMed
Microbiology, Czech Academy of Sciences, Vídenská 1083, CZ-142 20 Prague 4, Czech Republic
Search for other papers by H Tlaskalová-Hogenová in
Google Scholar
PubMed
Microbiology, Czech Academy of Sciences, Vídenská 1083, CZ-142 20 Prague 4, Czech Republic
Search for other papers by J Pácha in
Google Scholar
PubMed
localized in the cells of lamina propria ( Whorwood et al. 1994 ). This matches with the findings of 11HSD1 in fibroblasts ( Hammami & Siiteri 1991 ), macrophages ( Thieringer et al. 2001 ), and lymphocytes ( Zhang et al. 2005 ). Consistent with the
Search for other papers by Jessica R Mader in
Google Scholar
PubMed
Search for other papers by Zachary T Resch in
Google Scholar
PubMed
Search for other papers by Gary R McLean in
Google Scholar
PubMed
Search for other papers by Jakob H Mikkelsen in
Google Scholar
PubMed
Search for other papers by Claus Oxvig in
Google Scholar
PubMed
Search for other papers by Ronald J Marler in
Google Scholar
PubMed
Search for other papers by Cheryl A Conover in
Google Scholar
PubMed
mice. b Significant difference between 18-month-old non-diabetic and diabetic WT mice. Immunohistochemistry De-paraffinized sections of mouse kidney were stained for macrophages using F4/80 as primary antibody. Slides were rinsed with Tris
Search for other papers by Zhongyang Lu in
Google Scholar
PubMed
Search for other papers by Xiaoming Zhang in
Google Scholar
PubMed
Search for other papers by Yanchun Li in
Google Scholar
PubMed
Search for other papers by Junfei Jin in
Google Scholar
PubMed
Ralph H. Johnson Veterans Affairs Medical Center, Division of Endocrinology, Charleston, South Carolina 29401, USA
Search for other papers by Yan Huang in
Google Scholar
PubMed
by endogenous ligands that are likely to be related to hyperlipidemia in Apoe −/− mice. Indeed, in vitro studies have shown that minimally modified LDL (mmLDL) is capable of inducing inflammatory cytokine production in macrophages by engaging TLR4
Search for other papers by Sébastien Desarzens in
Google Scholar
PubMed
Zurich Center of Integrative Human Physiology (ZIHP), University of Zurich, Zurich, Switzerland
National Center of Competence in Research ‘Kidney.CH’, Zurich, Switzerland
Search for other papers by Nourdine Faresse in
Google Scholar
PubMed
-inflammatory genes and macrophage infiltration into adipose tissue in obese mice ( Guo et al . 2008 , Hirata et al . 2009 , Lee et al . 2014 ). Conversely, selective activation of GR by dexamethasone decreases pro-inflammatory cytokine expression and macrophage
Search for other papers by Zhiguo Liu in
Google Scholar
PubMed
Search for other papers by Chun Yan Lim in
Google Scholar
PubMed
Search for other papers by Michelle Yu-Fah Su in
Google Scholar
PubMed
Search for other papers by Stephanie Li Ying Soh in
Google Scholar
PubMed
Search for other papers by Guanghou Shui in
Google Scholar
PubMed
Search for other papers by Markus R Wenk in
Google Scholar
PubMed
Search for other papers by Kevin L Grove in
Google Scholar
PubMed
Search for other papers by George K Radda in
Google Scholar
PubMed
Laboratory of Lipid and Glucose Metabolism, Laboratory of Metabolic Medicine, Division of Neuroscience, Department of Biochemistry, The First Affiliated Hospital of Chongqing Medical University, 1 Youyi Road, Yuzhong District, Chongqing 400016, People's Republic of China
Search for other papers by Weiping Han in
Google Scholar
PubMed
Laboratory of Lipid and Glucose Metabolism, Laboratory of Metabolic Medicine, Division of Neuroscience, Department of Biochemistry, The First Affiliated Hospital of Chongqing Medical University, 1 Youyi Road, Yuzhong District, Chongqing 400016, People's Republic of China
Search for other papers by Xiaoqiu Xiao in
Google Scholar
PubMed
local increases in the production of non-esterified fatty acids (NEFA). Through increased expression of pro-inflammatory cytokines and chemokines, and infiltration of macrophages and other immune cells into adipose tissue, obesity-induced inflammation is
Search for other papers by Yuichi Kikuchi in
Google Scholar
PubMed
Search for other papers by Muneharu Yamada in
Google Scholar
PubMed
Search for other papers by Toshihiko Imakiire in
Google Scholar
PubMed
Search for other papers by Taketoshi Kushiyama in
Google Scholar
PubMed
Search for other papers by Keishi Higashi in
Google Scholar
PubMed
Search for other papers by Naomi Hyodo in
Google Scholar
PubMed
Search for other papers by Kojiro Yamamoto in
Google Scholar
PubMed
Search for other papers by Takashi Oda in
Google Scholar
PubMed
Search for other papers by Shigenobu Suzuki in
Google Scholar
PubMed
Search for other papers by Soichiro Miura in
Google Scholar
PubMed
prevented hypertensive renal injuries by inhibiting expression of extracellular matrix genes, oxidative stress, cell proliferation, and macrophage infiltration in several hypertensive models ( Kanda et al. 2003 , Nishikimi et al. 2004 , Ishikawa et al
Search for other papers by Eddy Himpe in
Google Scholar
PubMed
Search for other papers by Céline Degaillier in
Google Scholar
PubMed
Search for other papers by Astrid Coppens in
Google Scholar
PubMed
Search for other papers by Ron Kooijman in
Google Scholar
PubMed
of inflammation by chemoattractants such as interleukin-8 (IL8). This inflammatory cytokine is produced by monocytes and macrophages, and is also one of the most abundant cytokines produced by neutrophils, which triggers degranulation and oxidative
Search for other papers by Ljupka Gligorovska in
Google Scholar
PubMed
Search for other papers by Biljana Bursać in
Google Scholar
PubMed
Search for other papers by Sanja Kovačević in
Google Scholar
PubMed
Search for other papers by Nataša Veličković in
Google Scholar
PubMed
Search for other papers by Gordana Matić in
Google Scholar
PubMed
Search for other papers by Ana Djordjevic in
Google Scholar
PubMed
-abdominal adiposity and dysfunction of the adipose tissue, resulting in the infiltration of immune cells and development of chronic low-grade inflammation ( Monteiro & Azevedo 2010 ). Macrophage migration inhibitory factor (MIF) is a pleiotropic ubiquitously