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Centre for Endocrinology and Metabolism, Hudson Institute of Medical Research, Monash Medical Centre, Clayton, Victoria, Australia
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Department of Molecular and Translational Research, Monash University, Melbourne, Victoria, Australia
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No DOC/salt cardiac fibrosis, no macrophage recruitment Rickard et al . (2014) VE-Cad-Cre Obesity-induced dysfunction blunted inflammation Schafer et al . (2013) , Jia et al . (2015) Cardiomyocyte MLC2a-Cre Improved cardiac
Escola Superior de Saúde, Universidade do Algarve, Faro, Portugal
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modifications in the number, size and pigment content of aggregates of macrophages, the melanomacrophage centers (MMCs) of the piscine innate immune system ( Fournie et al. 2001 , Agius & Roberts 2003 , De Vico et al. 2008 ), and this is the predominant
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work, we established a male mouse model to observe the influence of iron on oxidative stress and bone resorption in vivo ( Jia et al . 2012 ). In addition, we treated a murine macrophage cell line, RAW264.7 with ferric ions to test their activity
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shown to attenuate obesity-induced chronic inflammation in adipose tissue by repressing the inflammatory responses of macrophages ( Ribas et al . 2011 ). Previous studies reported that estrogen acted on the CNS, especially on the hypothalamus
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Institute of Biomedicine, University of Barcelona, Barcelona, Spain
Centro de Investigación Biomédica en Red de Obesidad y Nutrición (CIBEROBN), Carlos III Health Institute, Madrid, Spain
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Institute of Biomedicine, University of Barcelona, Barcelona, Spain
Centro de Investigación Biomédica en Red de Obesidad y Nutrición (CIBEROBN), Carlos III Health Institute, Madrid, Spain
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Department of Endocrinology and Nutrition, Hospital Clinic of Barcelona, Barcelona, Spain
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM), Carlos III Health Institute, Madrid, Spain
Department of Medicine, Faculty of Medicine and Health Sciences, University of Barcelona, Barcelona, Spain
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macrophages in white adipose tissue Sections of 5 µm from the paraffin-embedded epididymal adipose tissue were deparaffinized and rehydrated in water. A pre-treatment using proteinase K was performed, and slides were blocked in 5% donkey serum for 1 h at
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stimulated the activation of signalling molecules ERK/MAPK and AKT. On the other hand, C5a and C5adR both inhibited the secretion of the inflammatory molecule, macrophage migration inhibitory factor (MIF) yet stimulated the secretion of ACTH. These data
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aortic root, starting at the appearance of the tricuspid valves. Collagen content of the lesions was determined after Masson's Trichrome staining (Sigma Diagnostics). Sections were stained immunohistochemically for the presence of macrophages using a MOMA
Department of Experimental Physiology, Department of Internal Medicine III and Institute of Physiology, Department of Biological Chemistry, Endocrine Unit, Experimental Research Centre, University of Athens Medical School, Athens, Greece
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Department of Experimental Physiology, Department of Internal Medicine III and Institute of Physiology, Department of Biological Chemistry, Endocrine Unit, Experimental Research Centre, University of Athens Medical School, Athens, Greece
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and SR-BI ( Miyazaki et al . 2002 ). Macrophage SR-A, the product of the MSR1 gene, is a multifunctional, multiligand pattern recognition receptor with roles in innate immunity, apoptotic cell clearance and age-related degenerative pathologies, such
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BK21 PLUS Project, Yonsei University College of Dentistry, Seoul, Korea
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BK21 PLUS Project, Yonsei University College of Dentistry, Seoul, Korea
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. Examples are autoimmune arthritis, osteoporosis, periodontitis, osteopetrosis and bone tumors ( Takayanagi 2007 , Zaidi 2007 ). Multinucleated mature osteoclasts originate from bone marrow-derived monocytes/macrophages (BMMs) through two essential factors
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Ralph H Johnson Veterans Affairs Medical Center, Division of Endocrinology, Department of Biostatistics, College of Dental Medicine
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Ralph H Johnson Veterans Affairs Medical Center, Division of Endocrinology, Department of Biostatistics, College of Dental Medicine
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, studies have shown that macrophages isolated from CD14 knockout mice are unable to produce TNFα, IL-1β, and interferon-inducible protein-10 in response to LPS and these mice have an early death in pneumococcal infection ( Vogel et al . 1998 ). Clearly