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Ekaterine Tskitishvili, Christel Pequeux, Carine Munaut, Renaud Viellevoye, Michelle Nisolle, Agnes Noël and Jean-Michel Foidart

hypoxic–ischemic encephalopathy (HIE) remains a serious condition that causes significant mortality and morbidity in near-term and term newborns; also, it does occur in premature infants as well ( Volpe 2001 ). The brain damage due to hypoxia and ischemia

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Dipali Goyal, Sean W Limesand and Ravi Goyal

include maternal food excess and deprivation, hypoxia, drug addiction, alcohol and emotional stress ( Limesand et al. 2006 , Jansson & Powell 2007 , Gluckman et al. 2008 , Hanson & Gluckman 2008 , Borengasser et al. 2013 , Goyal & Longo 2013

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Ingrid Segers, Tom Adriaenssens, Sandra Wathlet and Johan Smitz

( Hernandez-Gonzalez et al . 2006 ) Human CXCR4 expression in cumulus cells is negatively correlated with embryo cleaving capacity ( van Montfoort et al . 2008 ) Induced by hypoxia and EGFR signaling ( Phillips et al . 2005 ) Syndecan 4 ( Sdc4

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Barry N Madison, Patrick T K Woo and Nicholas J Bernier

gene expression. Previously, we have shown that hypoxia exposure can increase the gene expression of POA crf in rainbow trout ( Bernier & Craig 2005 ). At the pituitary level, peak C. salmositica infection was associated with a significant

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Marion Walser, Linus Schiöler, Jan Oscarsson, Maria A I Åberg, Ruth Wickelgren, Johan Svensson, Jörgen Isgaard and N David Åberg

were the rate-limiting enzymes of the haeme synthesis delta-aminolevulinate synthase 1 and 2 ( Alas1 , Alas2 ) and the hypoxia-inducible factor 1-alpha ( HIF1a ), which functions as a master transcriptional regulator of the adaptive response to hypoxia

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Local impact of thyroid hormone inactivation

Deiodinases: the balance of thyroid hormone

Monica Dentice and Domenico Salvatore

transforming growth factor β (TGFβ), acting via a Smad-dependent pathway, in diverse human cell types including fetal and adult fibroblasts, hemangioma cells, fetal epithelia, and skeletal muscle myoblasts ( Huang et al . 2005 ). Hypoxia stimulates D3 activity

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Kevin M Sargent, Renee M McFee, Renata Spuri Gomes and Andrea S Cupp

's expression as well as VEGFA upregulating upstream factors in the arachidonic pathway to further enhance COX2 activity. COX1 is thought to be constitutively expressed while most of the effects of hypoxia and VEGFA may be through stimulation of the inducible

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Harman S Mattu and Harpal S Randeva

. Leptin delays mitochondrial permeability transition pore (MPTP) opening (possibly determining protection against I/R injury) ( Halestrap et al . 2004 ). Leptin also protects cardiomyocytes from hypoxia-induced damage ( Erkasap et al . 2006 ). JAK

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Kenichiro Watanabe, Takahiro Nemoto, Shigeo Akira, Toshiyuki Takeshita and Tamotsu Shibasaki

, adrenal, uterus, ovary, and testis ( Hsu & Hsueh 2001 , Yamauchi et al . 2005 , Lee et al . 2011 ). It has been reported that expression of Ucn2 mRNA in various tissues is controlled by stress, inflammation, and hypoxia ( Tanaka et al . 2003 , Tao

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Motoi Sohmiya and Yuzuru Kato

with a molecular mass of 34 000 Da. EPO secretion was strongly stimulated by hypoxia ( Goldberg et al. 1989 ). EPO secretion is regulated endocrinologically. Thyroid hormones and adrenocortical hormone enhance EPO secretion ( Peschle et al. 1978