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Michiyo Tanikawa, Hwa-Yong Lee, Kikuko Watanabe, Magdalena Majewska, Dariusz J Skarzynski, Soo-Bong Park, Dong-Seok Lee, Choon-Keun Park, Tomas J Acosta, and Kiyoshi Okuda

House Co., London, UK) for 24 h. Oxytocin (OT, 100 nM; Teikoku Hormone MFG Co., Tokyo, Japan) and tumor necrosis factor-α (TNF, 0.06 nM; Dainippon Pharmaceutical Co., Ltd) was used. The concentrations of OT and TNF were based on a previous study

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David M Golding, Daniel J Rees, Jennifer R Davies, Dinko Relkovic, Hannah V Furby, Irina A Guschina, Anna L Hopkins, Jeffrey S Davies, James L Resnick, Anthony R Isles, and Timothy Wells

possible that proportionate hyperphagia may arise from impairment of the anorexic oxytocin circuitry. Although we have yet to quantify oxytocin expression in PWS-IC del mice, a reduction in the population of parvocellular oxytocin neurons has previously

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Z Zhang, P H Bisschop, E Foppen, H C van Beeren, A Kalsbeek, A Boelen, and E Fliers


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David R Grattan

levels in the blood at all times that prolactin secretion is high. The same is probably true for a large number of factors that have been investigated as putative ‘prolactin releasing hormones’, including thyrotropin-releasing hormone, oxytocin, galanin

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Christian Vollmer, Ingo Schwartges, Silke Naber, Christopher Beck, Inge Bauer, and Olaf Picker

). Although the relation between increased vasopressin plasma levels and the effect of the V 1A receptors might indicate an effect of vasopressin, we cannot exclude the effect of other possible ligands, e.g. oxytocin, on the receptor. However, vasopressin has

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A F Roy, Y Benomar, V Bailleux, C M Vacher, A Aubourg, A Gertler, J Djiane, and M Taouis

hormones that show significant plasma level changes during these periods. During pregnancy and lactation, other hormones may also contribute to hyperphagia. The lack of central oxytocin action may partly contribute to maternal hyperphagia ( Douglas et al

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Rafaela Fadoni Alponti, Patricia Lucio Alves, and Paulo Flavio Silveira

preference for substrates containing the N-terminal Cys ( Alponti et al . 2015 ), such as vasopressin and oxytocin, but also acts on ANG III, Lys-bradykinin, and bradykinin ( Keller 2004 , Ruster & Wolf 2013 ), all of which with important interaction with

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Ágnes Domokos, Zsuzsa Mergl, István Barna, Gábor B Makara, and Dóra Zelena

elevation of the oxytocin levels in di/di rats could – at least partly – compensate its HPA axis stimulatory role as oxytocin may act on pituitary V1b receptors to stimulate ACTH release ( Schlosser et al . 1994 ). The role of AVP in opiate

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A L Griffiths, K M Marshall, J Senior, C Fleming, and D F Woodward

the uterine horn appears to be working in synchrony to aid the expulsion of the litter. Oestrogen is thought to modulate several factors which contribute to the contractile state of the uterus during parturition, including oxytocin production

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Dóra Zelena, Ágnes Domokos, Subodh Kumar Jain, Ryan Jankord, and Ludmila Filaretova

rats led to a similar conclusion ( Popova et al . 2002 ). It is possible that oxytocin compensates for the loss of AVP in this process as it is also stimulated by osmotic challenges ( Schlosser et al . 1994 ). However, the control procedure (volume