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Syed Jalal Khundmiri Division of Nephrology and Hypertension, Department of Physiology and Biophysics, Department of Medicine
Division of Nephrology and Hypertension, Department of Physiology and Biophysics, Department of Medicine

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physiological and pathophysiological concentrations of cardiotonic steroids on renal salt reabsorption ( Aizman et al . 2001 , Abramowitz et al . 2003 , Aizman & Aperia 2003 , Contreras et al . 2006 , Khundmiri et al . 2006 , 2007 , 2014 , Aperia 2007

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-13-0613
Volume/Issue:
Volume 222: Issue 1
Free access
Louise J N Jensen Medical Department M and Medical Research Laboratories, Clinical Institute, Aarhus University Hospital, DK-8000 Aarhus C, Denmark
Stark Diabetes Center, Division of Endocrinology, University of Texas Medical Branch, Galvestone, Texas, USA
Renal Unit, Department of Internal Medicine, Ghent University Hospital, Ghent, Belgium
Department of Cell Biology, Institute of Anatomy, Aarhus University, Aarhus, Denmark

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Larry Denner Medical Department M and Medical Research Laboratories, Clinical Institute, Aarhus University Hospital, DK-8000 Aarhus C, Denmark
Stark Diabetes Center, Division of Endocrinology, University of Texas Medical Branch, Galvestone, Texas, USA
Renal Unit, Department of Internal Medicine, Ghent University Hospital, Ghent, Belgium
Department of Cell Biology, Institute of Anatomy, Aarhus University, Aarhus, Denmark

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Bieke F Schrijvers Medical Department M and Medical Research Laboratories, Clinical Institute, Aarhus University Hospital, DK-8000 Aarhus C, Denmark
Stark Diabetes Center, Division of Endocrinology, University of Texas Medical Branch, Galvestone, Texas, USA
Renal Unit, Department of Internal Medicine, Ghent University Hospital, Ghent, Belgium
Department of Cell Biology, Institute of Anatomy, Aarhus University, Aarhus, Denmark

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Ronald G Tilton Medical Department M and Medical Research Laboratories, Clinical Institute, Aarhus University Hospital, DK-8000 Aarhus C, Denmark
Stark Diabetes Center, Division of Endocrinology, University of Texas Medical Branch, Galvestone, Texas, USA
Renal Unit, Department of Internal Medicine, Ghent University Hospital, Ghent, Belgium
Department of Cell Biology, Institute of Anatomy, Aarhus University, Aarhus, Denmark

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Ruth Rasch Medical Department M and Medical Research Laboratories, Clinical Institute, Aarhus University Hospital, DK-8000 Aarhus C, Denmark
Stark Diabetes Center, Division of Endocrinology, University of Texas Medical Branch, Galvestone, Texas, USA
Renal Unit, Department of Internal Medicine, Ghent University Hospital, Ghent, Belgium
Department of Cell Biology, Institute of Anatomy, Aarhus University, Aarhus, Denmark

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Allan Flyvbjerg Medical Department M and Medical Research Laboratories, Clinical Institute, Aarhus University Hospital, DK-8000 Aarhus C, Denmark
Stark Diabetes Center, Division of Endocrinology, University of Texas Medical Branch, Galvestone, Texas, USA
Renal Unit, Department of Internal Medicine, Ghent University Hospital, Ghent, Belgium
Department of Cell Biology, Institute of Anatomy, Aarhus University, Aarhus, Denmark

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Introduction Diabetic nephropathy develops in 15–25% of all patients with type 1 diabetes and is one of the leading causes of end-stage renal failure. Accordingly, development of diabetic nephropathy is associated with a considerable

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06524
Volume/Issue:
Volume 188: Issue 3
Free access
Caiping Mao Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Rong Liu Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Le Bo Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Ningjing Chen Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Shigang Li Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Shuixiu Xia Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Jie Chen Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Dawei Li Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Lubo Zhang Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China
Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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Zhice Xu Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China
Institute for Fetology and Reproductive Medicine Center, Center for Prenatal Biology, First Hospital of Soochow University, Suzhou 215006, People's Republic of China

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humans, previous studies also demonstrated that the kidney may be affected in programming of renal and cardiovascular diseases ( do Carmo Pinho et al . 2003 , Bagby 2007 ). It is well known that high-salt diets (HSDs) are related to hypertension as well

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0139
Volume/Issue:
Volume 218: Issue 1
Free access
Félix Vargas
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Isabel Rodríguez-Gómez
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Pablo Vargas-Tendero
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Eugenio Jimenez Departamento de Fisiología, Departamento de Bioquímica y Biología Molecular e Inmunología, Facultad de Medicina, Universidad de Granada, E-18012 Granada, Spain

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Mercedes Montiel Departamento de Fisiología, Departamento de Bioquímica y Biología Molecular e Inmunología, Facultad de Medicina, Universidad de Granada, E-18012 Granada, Spain

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of life, when an increase in renal renin content ( Bouhnik et al . 1981 ) and PRC ( Jiménez et al . 1984 ) was observed, suggesting the operation of distinct mechanisms according to the age at which the thyroid hormone deficit is produced. In vitro

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-11-0349
Volume/Issue:
Volume 213: Issue 1
Free access
Hitesh Soni Department of Physiology, University of Tennessee Health Science Center, Memphis, Tennessee 38163, USA

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Adebowale Adebiyi Department of Physiology, University of Tennessee Health Science Center, Memphis, Tennessee 38163, USA

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paracrine fashion to modulate cardiovascular and renal homeostasis. Thus, alterations in expression, function, and regulation of these mediators and their receptors underlie pathophysiological mechanisms of a variety of cardiovascular and renal diseases

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0556
Volume/Issue:
Volume 217: Issue 3
Free access
Keerati Wanchai Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
School of Medicine, Mae Fah Luang University, Chiang Rai, Thailand

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Sakawdaurn Yasom Department of Microbiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Wannipa Tunapong Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Titikorn Chunchai Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Parameth Thiennimitr Department of Microbiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Chaiyavat Chaiyasut Faculty of Pharmacy, Chiang Mai University, Chiang Mai, Thailand

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Anchalee Pongchaidecha Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Varanuj Chatsudthipong Department of Physiology, Faculty of Science, Mahidol University, Bangkok, Thailand

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Siriporn Chattipakorn Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
Department of Oral Biology and Diagnostic Sciences, Faculty of Dentistry, Chiang Mai University, Chiang Mai, Thailand

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Nipon Chattipakorn Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

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Anusorn Lungkaphin Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand
Center for Research and Development of Natural Products for Health, Chiang Mai University, Chiang Mai, Thailand

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development of renal injury and dysfunction ( Kopple & Feroze 2011 ). A number of studies have shown that increased BMI is associated with a decline in renal function and increased risk of chronic kidney disease (CKD) ( Culleton et al. 1999 , Wang et al

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-17-0471
Volume/Issue:
Volume 237: Issue 1
Free access
Weixia Han Department of Pathology, Hebei Medical University, Shijiazhuang, China
Hebei Key Laboratory of Kidney Diseases, Shijiazhuang, China

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Chen Wang Department of Pathology, Second Hospital, Shanxi Medical University, Taiyuan, China

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Zhifen Yang Department of Pathology, Hebei Medical University, Shijiazhuang, China

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Lin Mu Department of Pathology, Hebei Medical University, Shijiazhuang, China

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Ming Wu Department of Pathology, Hebei Medical University, Shijiazhuang, China

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Nan Chen Department of Pathology, Hebei Medical University, Shijiazhuang, China

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Chunyang Du Department of Pathology, Hebei Medical University, Shijiazhuang, China

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Huijun Duan Department of Pathology, Hebei Medical University, Shijiazhuang, China
Hebei Key Laboratory of Kidney Diseases, Shijiazhuang, China

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Yonghong Shi Department of Pathology, Hebei Medical University, Shijiazhuang, China
Hebei Key Laboratory of Kidney Diseases, Shijiazhuang, China

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Introduction Diabetic nephropathy (DN) is a microvascular complication of diabetes mellitus, accounting for almost 50% of all end-stage renal disease cases. Renal fibrosis is a major characteristic of DN and an important predictor of renal

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0536
Volume/Issue:
Volume 241: Issue 1
Free access
Isabel Rodríguez-Gómez
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Inmaculada Banegas Departamento de Fisiología, Departamento de Ciencias de la Salud, Departamento de Farmacología, Departamento de Anatomía Patológica e Instituto de Biomedicina Regenerativa (IBIMER), Facultad de Medicina, Universidad de Granada, 18012 Granada, Spain

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Rosemary Wangensteen Departamento de Fisiología, Departamento de Ciencias de la Salud, Departamento de Farmacología, Departamento de Anatomía Patológica e Instituto de Biomedicina Regenerativa (IBIMER), Facultad de Medicina, Universidad de Granada, 18012 Granada, Spain

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Andrés Quesada Departamento de Fisiología, Departamento de Ciencias de la Salud, Departamento de Farmacología, Departamento de Anatomía Patológica e Instituto de Biomedicina Regenerativa (IBIMER), Facultad de Medicina, Universidad de Granada, 18012 Granada, Spain

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Rosario Jiménez Departamento de Fisiología, Departamento de Ciencias de la Salud, Departamento de Farmacología, Departamento de Anatomía Patológica e Instituto de Biomedicina Regenerativa (IBIMER), Facultad de Medicina, Universidad de Granada, 18012 Granada, Spain

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Mercedes Gómez-Morales Departamento de Fisiología, Departamento de Ciencias de la Salud, Departamento de Farmacología, Departamento de Anatomía Patológica e Instituto de Biomedicina Regenerativa (IBIMER), Facultad de Medicina, Universidad de Granada, 18012 Granada, Spain

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Francisco O'Valle Departamento de Fisiología, Departamento de Ciencias de la Salud, Departamento de Farmacología, Departamento de Anatomía Patológica e Instituto de Biomedicina Regenerativa (IBIMER), Facultad de Medicina, Universidad de Granada, 18012 Granada, Spain

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Juan Duarte Departamento de Fisiología, Departamento de Ciencias de la Salud, Departamento de Farmacología, Departamento de Anatomía Patológica e Instituto de Biomedicina Regenerativa (IBIMER), Facultad de Medicina, Universidad de Granada, 18012 Granada, Spain

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Félix Vargas
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published on the relationship between TH and renal capillary density. With this background, the objective of this study was to analyse the effects of a chronic excess or deficiency of circulating TH on cardiac and renal capillary densities and on glomerular

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0208
Volume/Issue:
Volume 216: Issue 1
Free access
Johan Svensson Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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Åsa Tivesten Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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Klara Sjögren Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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Olle Isaksson Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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Göran Bergström Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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Subburaman Mohan Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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Johan Mölne Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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Jörgen Isgaard Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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Claes Ohlsson Department of Internal Medicine, Sahlgrenska University Hospital, Gröna Stråket 8, SE-413 45 Göteborg, Sweden
Department of Clinical Physiology, Göteborg University, Göteborg, Sweden
Musculoskeletal Disease Center, Jerry L Pettis Memorial VA Medical Center, Loma Linda, California, USA
Department of Pathology, Sahlgrenska University Hospital, Göteborg, Sweden

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). In diabetic as well as non diabetic end-stage renal failure, there are marked abnormalities in the GH/IGF-I system ( Jain et al. 1998 , Rabkin & Schaefer 2004 ). In humans with diabetes mellitus (DM) type I, circulating IGF-I is low and

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0024
Volume/Issue:
Volume 193: Issue 3
Free access
Alaa E S Abdel-Razik Faculty of Life Sciences, University of Manchester, 1.124 Stopford Building, Oxford Road, Manchester M13 9PT, UK

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Ellen J Forty Faculty of Life Sciences, University of Manchester, 1.124 Stopford Building, Oxford Road, Manchester M13 9PT, UK

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Richard J Balment Faculty of Life Sciences, University of Manchester, 1.124 Stopford Building, Oxford Road, Manchester M13 9PT, UK

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Nick Ashton Faculty of Life Sciences, University of Manchester, 1.124 Stopford Building, Oxford Road, Manchester M13 9PT, UK

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. 2002 ). Furthermore, recent evidence now suggests that UTS may play a role in regulating renal function in mammals. The kidney is a major source of UTS in humans ( Nothacker et al . 1999 ), primates, mice ( Elshourbagy et al . 2002 ) and rats ( Song

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0260
Volume/Issue:
Volume 198: Issue 3
Free access