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Yasminye D Pettway
Yasminye D Pettway
Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee, USA
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Diane C Saunders
Diane C Saunders
Department of Medicine, Division of Diabetes, Endocrinology and Metabolism, Vanderbilt University Medical Center, Nashville, Tennessee, USA
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Marcela Brissova
Marcela Brissova
Department of Medicine, Division of Diabetes, Endocrinology and Metabolism, Vanderbilt University Medical Center, Nashville, Tennessee, USA
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, transplantation of human islets ameliorates the previously reported negative effects of interrupted glucagon signaling, making this a suitable model for the study of normal human α cell physiology. Adaptations to physiologic states Aging Islet
Rui Gao
Rui Gao
Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK
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Samuel Acreman
Samuel Acreman
Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK
Department of Physiology, Institute of Neuroscience and Physiology, Metabolic Research Unit, University of Gothenburg, Göteborg, Sweden
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Department of Physiology, Institute of Neuroscience and Physiology, Metabolic Research Unit, University of Gothenburg, Göteborg, Sweden
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Jinfang Ma
Jinfang Ma
Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK
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Fernando Abdulkader
Fernando Abdulkader
Department of Physiology and Biophysics, Institute of Biomedical Sciences, University of Sao Paulo, Sao Paulo, Brazil
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Anna Wendt
Anna Wendt
Department of Clinical Sciences Malmö, Islet Cell Exocytosis, Lund University Diabetes Centre, Lund University, Malmö, Sweden
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Quan Zhang
Quan Zhang
Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford, Churchill Hospital, Oxford, UK
CNC - Center for Neuroscience and Cell Biology, CIBB - Centre for Innovative Biomedicine and Biotechnology, University of Coimbra, Coimbra, Portugal
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CNC - Center for Neuroscience and Cell Biology, CIBB - Centre for Innovative Biomedicine and Biotechnology, University of Coimbra, Coimbra, Portugal
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) work in concert to stimulate or inhibit glucose counter-regulation ( Sherwin 2008 ). Previous hypoglycaemia impairs VMH glucose sensing by multiple adaptations, including reduced VMH K ATP -channel activity ( McCrimmon et al. 2005 ), increased
