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Chiung-Zuan Chiu, Bao-Wei Wang, and Kou-Gi Shyu

, Zhang et al . 2007 ). Reactive oxygen species (ROS) are involved in the UII-induced cardiomyocyte hypertrophy ( Liu et al . 2009 ). One study has demonstrated that the generation of ROS is involved in UII-induced cell proliferation, tyrosine

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Laura Marroqui, Eva Tudurí, Paloma Alonso-Magdalena, Iván Quesada, Ángel Nadal, and Reinaldo Sousa dos Santos

cellular processes. For instance, mitochondria are implicated in hormone secretion ( Chow et al. 2017 ), generation of reactive oxygen species (ROS) ( Brand 2016 ) and cell death ( Fulda et al. 2010 ). Such involvement in a wide variety of processes

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S Ö Íşeri, G Şener, M Yüksel, G Contuk, Ş Çetinel, N Gedik, and B Ç Yeğen

lactate dehydrogenase (LDH) levels. Serum LDH activity was measured as a marker of systemic tissue injury. The gastric tissue samples were immediately taken and stored at −70 °C. Formation of reactive oxygen species in tissue samples was monitored by using

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Chan-Juan Ma, Ai-Fang Nie, Zhi-Jian Zhang, Zhi-Guo Zhang, Li Du, Xiao-Ying Li, and Guang Ning

 min, and then loaded onto SDS–PAGE. Measurement of intracellular reactive oxygen species For analysis of intracellular reactive oxygen species (ROS), the redox-sensitive fluorescent probe DCFH-DA was used, as described previously ( Kuang et al . 2009

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Yeon-Ju Lee, Se-Hui Jung, JongYun Hwang, Sohee Jeon, Eun-Taek Han, Won Sun Park, Seok-Ho Hong, Young-Myeong Kim, and Kwon-Soo Ha

.26331 ) 10.1002/mds.26331 Shin YJ Hyon JY Kim S Koh JW Kwon SI Wee WR 2011 Cysteamine suppresses human peripheral blood mononuclear cells – human corneal endothelial cell reaction via reactive oxygen species reduction

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Eun-Kyung Choi, Woon-Ki Kim, Ok-Joo Sul, Yun-Kyung Park, Eun-Sook Kim, Jae-Hee Suh, Rina Yu, and Hye-Seon Choi

′ and GAPDH, 5′-acccagaagactgtggatgg-3′ and 5′-cacattgggggtaggaacac-3′. Intracellular reactive oxygen species detection The intracellular formation of reactive oxygen species (ROS) was detected using the fluorescence probe, 2′,7′-dichlorofluorescein

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Paola Venditti, Angela Bari, Lisa Di Stefano, and Sergio Di Meo

increase enhances tetravalent and univalent oxygen reduction, leading to higher oxygen consumption and reactive oxygen species (ROS) production respectively ( Fernández & Videla 1993 , Venditti et al . 2003 ). Studies on the effects of tri

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P Venditti, G Chiellini, L Di Stefano, G Napolitano, R Zucchi, A Columbano, T S Scanlan, and S Di Meo

phosphorylation, and mitochondria also represent the main source of reactive oxygen species (ROS), the above-mentioned mechanisms can be understood by studying the effects of TRβ activation on mitochondrial population characteristics. Thus, in the present study

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Ana B Ropero, Pablo Juan-Picó, Alex Rafacho, Esther Fuentes, F Javier Bermúdez-Silva, Enrique Roche, Ivan Quesada, Fernando Rodríguez de Fonseca, and Angel Nadal

, growing evidence points to the existence of a non-genomic effect of PPAR ligands via a PPAR-independent pathway. These non-genomic actions include mitochondrial effects, rapid reactive oxygen species (ROS) formation, MAPK activation, and expression of

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Jin Sook Yoon, Hyun Jung Lee, Min Kyung Chae, Sang Yeul Lee, and Eun Jig Lee

( Eckstein et al . 2003 ). Cigarette smoke is considered to act, in part, by enhancing the generation of reactive oxygen species (ROS) and increasing oxidative stress in the closed bony orbital environment, either through direct contact with the sinus and