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Fan He, Xiaozhen Liu, Ke Xiong, Sijin Chen, Long Zhou, Wenguo Cui, Guoqing Pan, Zong-Ping Luo, Ming Pei, and Yihong Gong

cultured on decellularized ECM, or on tissue culture polystyrene (TCPS), with or without melatonin. Production of intracellular reactive oxygen species (ROS), antioxidant enzyme activity, cellular proliferation, osteogenic differentiation, and matrix

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Catherine Garrel, Paul A Fowler, and Kaïs H Al-Gubory

Introduction Maintaining a physiological equilibrium between intracellular levels of antioxidants and the production of reactive oxygen species (ROS) is crucial for the survival of the developing organisms. Tightly controlled ROS generation appears

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Anna E Bollag, Tianyang Guo, Ke-Hong Ding, Vivek Choudhary, Xunsheng Chen, Qing Zhong, Jianrui Xu, Kanglun Yu, Mohamed E Awad, Mohammed Elsalanty, Maribeth H Johnson, Meghan E McGee-Lawrence, Wendy B Bollag, and Carlos M Isales

strongly with osteoporotic fractures, so as the worldwide population continues to age, fracture risk will increase proportionally. Reactive oxygen species (ROS) contribute to age-related bone loss that can predispose to fractures ( Almeida et al. 2007

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Bo Qian, Haiyan Wang, Xiuli Men, Wenjian Zhang, Hanqing Cai, Shiqing Xu, Yaping Xu, Liya Ye, Claes B Wollheim, and Jinning Lou

manufacturer's protocol then detected by micro plate fluorescence reader (Molecular Devices Corp, Sunnyvale, CA, USA). Reactive oxygen species (ROS) assay by flow cytometry and xanthine oxidase (XOD) measurement ROS formation was analyzed by flow cytometry

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E Mezosi, J Szabo, E V Nagy, A Borbely, E Varga, G Paragh, and Z Varga

treatment ( Fernandez & Videla 1995 ). The stimulation of reactive oxygen species generation by T 3 was demonstrated in euthyroid human subjects as well ( Magsino et al. 2000 ). Others found rapid stimulation of oxygen consumption in human mononuclear

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Wanpitak Pongkan, Hiranya Pintana, Sivaporn Sivasinprasasn, Thidarat Jaiwongkam, Siriporn C Chattipakorn, and Nipon Chattipakorn

. 2011 ). Cardiac mitochondrial function was determined by measuring mitochondrial reactive oxygen species (ROS) production, mitochondrial membrane potential change (ΔΨm), and mitochondrial swelling. Transmission electron microscopy (TEM) was also used to

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Jia Sun, Haiping Zhu, Xiaorong Wang, Qiuqi Gao, Zhuoying Li, and Huiya Huang

.1371/journal.pone.0055027 ) 10.1371/journal.pone.0055027 23359116 Susztak K Raff AC Schiffer M Böttinger EP 2006 Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy . Diabetes

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Baiyang You, Yaoshan Dun, Wenliang Zhang, Lingjun Jiang, Hui Li, Murong Xie, Yuan Liu, and Suixin Liu

with augmented reactive oxygen species (ROS) production and mitochondrial dysfunction, and incomplete lipid oxidation and increased lipid accumulation in skeletal muscle are considered as responsible for IR development ( Krebs & Roden 2004 , Anderson

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Mika Suzuki, Hiroshi Kobayashi, Yoshiko Tanaka, Naohiro Kanayama, and Toshihiko Terao

can indeed be classified into distinct subsets. These include stress-related, apoptosis-related, proteases, signaling molecules, ion channel, cytokines and growth factors, aging-related, hyaluronan metabolism and signaling, reactive oxygen species

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Jie Miao, Yacir Benomar, Sarah Al Rifai, Ghislaine Poizat, Laure Riffault, Delphine Crépin, and Mohammed Taouis

neurodegenerative diseases. Hence, by eliminating injured and aged mitochondria that constitute an important source of reactive oxygen species. Thus, in contrast with resistin action, autophagy reduces cellular inflammatory responses ( De Lavera et al . 2017