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Rosalia C M Simmen, Dustin M Brown, Charles M Quick, Iad Alhallak, Tyler Rose, Shi J Liu, and Angela S Kelley

endometriosis: link to ovarian cancer . Endocrinology 626 – 638 . ( https://doi.org/10.1210/en.2018-00794 ) 30657901 Burke SD Dong H Hazan AD Croy BA 2007 Aberrant endometrial features of pregnancy in diabetic NOD mice . Diabetes 2919 – 2926

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Sebastian Weise, Susan Kralisch, Grit Sommer, Ulrike Lossner, Matthias Bluher, Michael Stumvoll, and Mathias Fasshauer

). Consistent with these findings, Thomas and co-workers have shown that IL-1 receptor-deficient non-obese diabetic mice were partially protected from the development of T2DM ( Thomas et al . 2004 ). Furthermore, blockage of IL-1 signaling with anakinra, a

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James M Evans, Laura S Morris, and Julian R Marchesi

gut microbiota plays a role in the development of type 1 diabetes ( Wen et al . 2008 ). In this study, the islets of Langherans of germ-free, specific pathogen-free and altered Schaedler flora ( Dewhirst et al . 1999 ) Myd88 knockout NOD mice were

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José Edgar Nicoletti-Carvalho, Tatiane C Araújo Nogueira, Renata Gorjão, Carla Rodrigues Bromati, Tatiana S Yamanaka, Antonio Carlos Boschero, Licio Augusto Velloso, Rui Curi, Gabriel Forato Anhê, and Silvana Bordin

al . 2003 , Choi et al . 2004 ), and to increase cell death in primary islets treated with FFAs ( Ellingsgaard et al . 2008 ). Paradoxically, it was observed that the incidence of T1DM in non-obese diabetic (NOD) mice decreased when the mice were

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Binbin Guan, Wenyi Li, Fengying Li, Yun Xie, Qicheng Ni, Yanyun Gu, Xiaoying Li, Qidi Wang, Hongli Zhang, and Guang Ning

T Romano LC Zou B O’Donnell CP ,. 2011 Glucose stimulates human beta cell replication in vivo in islets transplanted into NOD-severe combined immunodeficiency (SCID) mice. Diabetologia 54 572 – 582 . ( doi:10.1007/s00125

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Y Wang, L P Wu, J Fu, H J Lv, X Y Guan, L Xu, P Chen, C Q Gao, P Hou, M J Ji, and B Y Shi

priority for all scientific researchers. We have shown that GD could be prevented by the induction of immunotolerance to TSHR in neonatal BALB/c mice and that this is an effective method of prevention for GD ( Wu et al . 2011 ). This innovation could

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G Burnstock and I Novak

centres of islets to replace the lost β-cells in both STZ-diabetic rats and NOD mice ( Coutinho-Silva et al . 2003 , 2007 ). A study has shown that P2X 7 receptors were expressed in β-cells (and also α-cells) and receptors were down-regulated in type 2

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J A Tamblyn, M Hewison, C L Wagner, J N Bulmer, and M D Kilby

NOD mice . Diabetes 51 1367 – 1374 . ( doi:10.2337/diabetes.51.5.1367 ) Griffin MD Lutz WH Phan VA Bachman LA McKean DJ Kumar R 2000 Potent inhibition of dendritic cell differentiation and maturation by vitamin D analogs

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Jorge N Artaza and Keith C Norris

-induced apoptosis in non-obese diabetic (NOD) mice and protects against diabetes . Clinical and Experimental Immunology 112 181 – 187 . Dusso AS Brown AJ 1998 Mechanism of vitamin D action and its regulation . American Journal of Kidney Diseases 32

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C Giuliani, M Saji, I Bucci, G Fiore, M Liberatore, D S Singer, F Monaco, L D Kohn, and G Napolitano

Major histocompatibility complex class I-deficient NOD-Β 2 m null mice are diabetes and insulinitis resistant. Diabetes 43 505 –509. Singer DS , Kohn LD, Zinger H & Mozes E 1994 Methimazole prevents induction of