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Sun-O Ka, Mi-Young Song, Eun Ju Bae, and Byung-Hyun Park

cytokines in the liver and adipose tissue and an increase in the number of CLSs. Notably, mS1KO mice on a HFD showed a decrease in hepatic triglycerides and steatosis and smaller adipocyte size compared with WT mice. These findings are in contrast to those

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Patrice D Cani, Catherine A Daubioul, Brigitte Reusens, Claude Remacle, Grégory Catillon, and Nathalie M Delzenne

.) ( Delzenne 2003 , Delzenne et al. 2003 ). We have shown in previous studies that oligofructose – a short chain fructan obtained from inulin -decreases steatosis by reducing spontaneous food intake in obese Zucker Fa/Fa rats ( Daubioul et al. 2002

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Alia H Sukkar, Aaron M Lett, Gary Frost, and Edward S Chambers

hepatic steatosis by dietary supplementation of SCFAs over a period of 12 weeks. One of the possible underlying mechanisms was the decrease in peroxisome proliferator-activated receptor gamma ( Pparg ) activity in the liver. Pparg increases energy

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Greg M Kowalski, Michael J Kraakman, Shaun A Mason, Andrew J Murphy, and Clinton R Bruce

:10.1152/ajpendo.90996.2008 ) Meex RC Hoy AJ Morris A Brown RD Lo JC Burke M Goode RJ Kingwell BA Kraakman MJ Febbraio MA 2015 Fetuin B is a secreted hepatocyte factor linking steatosis to impaired glucose

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Nailliw Z Preite, Bruna P P do Nascimento, Cynthia R Muller, Anna Laura V Américo, Talita S Higa, Fabiana S Evangelista, Carmen L Lancellotti, Felipe dos Santos Henriques, Miguel Luiz Batista Jr, Antonio C Bianco, and Miriam O Ribeiro

feeding on a HFD resulted in a severe macrovesicular liver steatosis, with triglyceride accumulation in large hepatocyte vacuoles but similar in WT and ARβ 3 KO ( Fig. 5C , D and E ). HFD-induced glucose intolerance was similar in both groups ( Fig. 6

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I Sadaf Farooqi and Stephen O'Rahilly

et al . 1999 ). In those who survive, obesity continues in adult life, with hepatic steatosis ( von Schnurbein et al . 2013 ) and hyperinsulinaemia consistent with the severity of obesity. Some adults have developed type 2 diabetes in the third to

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Julie Rodriguez and Nathalie M Delzenne

in obesity, which are summarized in Fig. 2 . Due to the high number of reviews focusing on the impact of prebiotics on the liver steatosis and related metabolic disorders, we chose to present the existing literature related to the effects on the

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Fabio Arturo Iannotti and Vincenzo Di Marzo

the liver – increased de novo lipogenesis and insulin resistance, hepatic steatosis and dyslipidemia; (3) in the white adipose tissue – increased energy storage capacity, adipogenesis, lipogenesis, insulin resistance and leptin release, decreased

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Milos Mraz and Martin Haluzik

migration ( Gerhardt et al . 2001 , Christiansen et al . 2005 ). Overexpression of MCP1 leads to ATM infiltration, IR, and liver steatosis without increasing body weight, while deletion of MCP1 or macrophage CCR2 reduces ATM numbers in AT and improves

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Haijiang Wu, Xinna Deng, Yonghong Shi, Ye Su, Jinying Wei, and Huijun Duan

inhibitor ameliorates hepatic steatosis by improving ER stress and inflammation in a type 2 diabetic mouse model . PLoS ONE 10 e0120711 . ( doi:10.1371/journal.pone.0120711 ) Knutti D Kaul A Kralli A 2000 A tissue-specific coactivator of