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Rocío Fuente Institute of Physiology, University of Zurich, and National Center of Competence in Research NNCR Kidney, Zurich, CH, Switzerland
Division of Pediatrics, University of Oviedo, Oviedo, Spain
Department of Pediatrics, Hospital Universitario Central, Oviedo, Spain

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Eva-Maria Pastor-Arroyo Institute of Physiology, University of Zurich, and National Center of Competence in Research NNCR Kidney, Zurich, CH, Switzerland

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Nicole Gehring Institute of Physiology, University of Zurich, and National Center of Competence in Research NNCR Kidney, Zurich, CH, Switzerland

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Patricia Oro Carbajosa Division of Pediatrics, University of Oviedo, Oviedo, Spain

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Laura Alonso-Durán Division of Pediatrics, University of Oviedo, Oviedo, Spain

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Ivan Zderic AO Research Institute Davos, AO Foundation, Clavadelerstrasse, Davos Platz, Switzerland

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James Tapia-Dean Department of Pediatrics, Hospital Universitario Central, Oviedo, Spain

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Ahmad Kamal Hamid Institute of Physiology, University of Zurich, and National Center of Competence in Research NNCR Kidney, Zurich, CH, Switzerland

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Carla Bettoni Institute of Physiology, University of Zurich, and National Center of Competence in Research NNCR Kidney, Zurich, CH, Switzerland

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Fernando Santos Division of Pediatrics, University of Oviedo, Oviedo, Spain
Department of Pediatrics, Hospital Universitario Central, Oviedo, Spain

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Carsten A Wagner Institute of Physiology, University of Zurich, and National Center of Competence in Research NNCR Kidney, Zurich, CH, Switzerland

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Isabel Rubio-Aliaga Institute of Physiology, University of Zurich, and National Center of Competence in Research NNCR Kidney, Zurich, CH, Switzerland

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Introduction Fibroblast growth factor 23 (FGF23) together with parathyroid hormone (PTH), calcitriol, and αKlotho are the key regulators of phosphate homeostasis ( Hu et al. 2013 ). FGF23 is mainly synthetized in osteocytes and osteoblasts

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-23-0025
Volume/Issue:
Volume 259: Issue 1
Restricted access
Erin Faught Department of Biological Sciences, University of Calgary, Calgary, Alberta, Canada

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Mathilakath M Vijayan Department of Biological Sciences, University of Calgary, Calgary, Alberta, Canada

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Introduction Both intrauterine and early postnatal environments program various physiological systems in vertebrates. Major factors affecting fetal growth include maternal nutrition and fetal stress. Excess glucocorticoids (GCs) are of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0358
Volume/Issue:
Volume 244: Issue 2
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Caiyun Sun State Key Laboratory of Biocontrol, Institute of Aquatic Economic Animals and Guangdong Provincial Key Laboratory for Aquatic Economic Animals, School of Life Sciences, Sun Yat-Sen University, No. 135, XinGang West Road, Guangzhou 510275, People's Republic of China

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Da Duan State Key Laboratory of Biocontrol, Institute of Aquatic Economic Animals and Guangdong Provincial Key Laboratory for Aquatic Economic Animals, School of Life Sciences, Sun Yat-Sen University, No. 135, XinGang West Road, Guangzhou 510275, People's Republic of China

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Bo Li State Key Laboratory of Biocontrol, Institute of Aquatic Economic Animals and Guangdong Provincial Key Laboratory for Aquatic Economic Animals, School of Life Sciences, Sun Yat-Sen University, No. 135, XinGang West Road, Guangzhou 510275, People's Republic of China

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Chaobin Qin State Key Laboratory of Biocontrol, Institute of Aquatic Economic Animals and Guangdong Provincial Key Laboratory for Aquatic Economic Animals, School of Life Sciences, Sun Yat-Sen University, No. 135, XinGang West Road, Guangzhou 510275, People's Republic of China

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Jirong Jia State Key Laboratory of Biocontrol, Institute of Aquatic Economic Animals and Guangdong Provincial Key Laboratory for Aquatic Economic Animals, School of Life Sciences, Sun Yat-Sen University, No. 135, XinGang West Road, Guangzhou 510275, People's Republic of China

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Bin Wang State Key Laboratory of Biocontrol, Institute of Aquatic Economic Animals and Guangdong Provincial Key Laboratory for Aquatic Economic Animals, School of Life Sciences, Sun Yat-Sen University, No. 135, XinGang West Road, Guangzhou 510275, People's Republic of China

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Haiyan Dong State Key Laboratory of Biocontrol, Institute of Aquatic Economic Animals and Guangdong Provincial Key Laboratory for Aquatic Economic Animals, School of Life Sciences, Sun Yat-Sen University, No. 135, XinGang West Road, Guangzhou 510275, People's Republic of China

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Wensheng Li State Key Laboratory of Biocontrol, Institute of Aquatic Economic Animals and Guangdong Provincial Key Laboratory for Aquatic Economic Animals, School of Life Sciences, Sun Yat-Sen University, No. 135, XinGang West Road, Guangzhou 510275, People's Republic of China

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. 2006 ); ii) hUII is an autocrine/paracrine growth factor in porcine renal epithelial cells ( Matsushita et al . 2003 ) and involved in the pathogenesis of vascular remodeling ( Zhang et al . 2008 ); and iii) UII can stimulate the proliferation of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0282
Volume/Issue:
Volume 220: Issue 1
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Almas R Juma Department of Physiology, Department of Clinical Sciences, Department of Human Genetics, Anatomy and Microbiology, School of Life Sciences, La Trobe University, Bundoora, Victoria 3086, Australia

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Pauliina E Damdimopoulou Department of Physiology, Department of Clinical Sciences, Department of Human Genetics, Anatomy and Microbiology, School of Life Sciences, La Trobe University, Bundoora, Victoria 3086, Australia

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Sylvia V H Grommen Department of Physiology, Department of Clinical Sciences, Department of Human Genetics, Anatomy and Microbiology, School of Life Sciences, La Trobe University, Bundoora, Victoria 3086, Australia

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Wim J M Van de Ven Department of Physiology, Department of Clinical Sciences, Department of Human Genetics, Anatomy and Microbiology, School of Life Sciences, La Trobe University, Bundoora, Victoria 3086, Australia

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Bert De Groef Department of Physiology, Department of Clinical Sciences, Department of Human Genetics, Anatomy and Microbiology, School of Life Sciences, La Trobe University, Bundoora, Victoria 3086, Australia

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PLAG1 in the endocrine systems coordinating growth and reproduction in particular. Molecular biology of PLAG1 Gene and protein structure of PLAG1 The PLAG1 gene is located on human chromosome 8q12. Although the predominant PLAG1 transcript is 7.3 kb

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-15-0449
Volume/Issue:
Volume 228: Issue 2
Free access
Claire L Wood Division of Developmental Biology, Roslin Institute, University of Edinburgh, Edinburgh, UK

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Ondrej Soucek Department of Paediatrics, 2nd Faculty of Medicine, Charles University in Prague and Motol University Hospital, Prague, Czech Republic
Department of Women’s and Children’s Health, Karolinska Institutet and Pediatric Endocrinology Unit, Karolinska University Hospital, Stockholm, Sweden

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Sze C Wong Developmental Endocrinology Research Group, School of Medicine, University of Glasgow, Glasgow, UK

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Farasat Zaman Department of Women’s and Children’s Health, Karolinska Institutet and Pediatric Endocrinology Unit, Karolinska University Hospital, Stockholm, Sweden

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Colin Farquharson Division of Developmental Biology, Roslin Institute, University of Edinburgh, Edinburgh, UK

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Lars Savendahl Department of Women’s and Children’s Health, Karolinska Institutet and Pediatric Endocrinology Unit, Karolinska University Hospital, Stockholm, Sweden

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S Faisal Ahmed Developmental Endocrinology Research Group, School of Medicine, University of Glasgow, Glasgow, UK

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-induced osteoporosis (GIO) and growth retardation. Osteoporosis is characterized by a reduction in bone mass and loss of bone microarchitecture, leading to impaired bone strength and increased fracture risk ( Reinwald & Burr 2008 ). GIO is the most prevalent type of

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-17-0361
Volume/Issue:
Volume 236: Issue 1
Free access
Alexander Hennebry AgResearch Ltd, Hamilton, New Zealand

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Jenny Oldham AgResearch Ltd, Hamilton, New Zealand

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Tea Shavlakadze School of Anatomy, Physiology & Human Biology, The University of Western Australia, Crawley, Western Australia, Australia

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Miranda D Grounds School of Anatomy, Physiology & Human Biology, The University of Western Australia, Crawley, Western Australia, Australia

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Philip Sheard Department of Physiology, University of Otago, Dunedin, New Zealand

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Marta L Fiorotto USDA/ARS Children’s Nutrition Research Center, Baylor College of Medicine, Houston, Texas, USA

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Shelley Falconer AgResearch Ltd, Hamilton, New Zealand

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Heather K Smith Department of Exercise Sciences, University of Auckland, Auckland, New Zealand

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Carole Berry AgResearch Ltd, Hamilton, New Zealand

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Ferenc Jeanplong AgResearch Ltd, Hamilton, New Zealand

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Jeremy Bracegirdle AgResearch Ltd, Hamilton, New Zealand

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Kenneth Matthews AgResearch Ltd, Hamilton, New Zealand

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Gina Nicholas AgResearch Ltd, Hamilton, New Zealand

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Mônica Senna-Salerno AgResearch Ltd, Hamilton, New Zealand

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Trevor Watson AgResearch Ltd, Hamilton, New Zealand

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Christopher D McMahon AgResearch Ltd, Hamilton, New Zealand

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Introduction Two factors play critical but opposing roles in regulating the growth and size of skeletal muscle. The growth hormone, insulin-like growth factor-1 (GH-IGF1) axis stimulates growth and accounts for 83% of postnatal growth and the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-17-0032
Volume/Issue:
Volume 234: Issue 2
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Julian C Lui Section on Growth and Development, Eunice Kennedy Shriver National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland, USA

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Introduction Longitudinal bone growth is driven by a process called endochondral ossification, in which growing cartilage is gradually remodeled into newly formed bone ( Kronenberg 2003 ). During embryonic development, this process begins with

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-20-0045
Volume/Issue:
Volume 246: Issue 1
Free access
Ryoko Yamamoto Orthodontics and Craniofacial Developmental Biology, Oral Growth and Developmental Biology, Department of Molecular Genetics, Hiroshima University Graduate School of Biomedical Sciences, Hiroshima 734-8553, Japan

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Tomoko Minamizaki Orthodontics and Craniofacial Developmental Biology, Oral Growth and Developmental Biology, Department of Molecular Genetics, Hiroshima University Graduate School of Biomedical Sciences, Hiroshima 734-8553, Japan

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Yuji Yoshiko Orthodontics and Craniofacial Developmental Biology, Oral Growth and Developmental Biology, Department of Molecular Genetics, Hiroshima University Graduate School of Biomedical Sciences, Hiroshima 734-8553, Japan

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Hirotaka Yoshioka Orthodontics and Craniofacial Developmental Biology, Oral Growth and Developmental Biology, Department of Molecular Genetics, Hiroshima University Graduate School of Biomedical Sciences, Hiroshima 734-8553, Japan

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Kazuo Tanne Orthodontics and Craniofacial Developmental Biology, Oral Growth and Developmental Biology, Department of Molecular Genetics, Hiroshima University Graduate School of Biomedical Sciences, Hiroshima 734-8553, Japan

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Jane E Aubin Orthodontics and Craniofacial Developmental Biology, Oral Growth and Developmental Biology, Department of Molecular Genetics, Hiroshima University Graduate School of Biomedical Sciences, Hiroshima 734-8553, Japan

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Norihiko Maeda Orthodontics and Craniofacial Developmental Biology, Oral Growth and Developmental Biology, Department of Molecular Genetics, Hiroshima University Graduate School of Biomedical Sciences, Hiroshima 734-8553, Japan

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-resistant rickets suggest the existence of additional phosphaturic factor(s). Recently, intensive studies of several such putative factors (e.g. matrix extracellular phosphoglycoprotein, dentin matrix protein 1 (DMP1), and fibroblast growth factor 23 (FGF23) have

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-10-0058
Volume/Issue:
Volume 206: Issue 3
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Ryan Paul University of Auckland Waikato Clinical School, Hamilton, New Zealand

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Kim Whiteman ManukaMed LP, Hamilton, New Zealand

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Shelley J Falconer AgResearch Ltd, Hamilton, New Zealand

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Jenny M Oldham AgResearch Ltd, Hamilton, New Zealand

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Ferenc Jeanplong AgResearch Ltd, Hamilton, New Zealand

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Kenneth G Matthews AgResearch Ltd, Hamilton, New Zealand

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Heather K Smith Department of Exercise Sciences, University of Auckland, Auckland Mail Centre, Auckland, New Zealand

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Mark Thomas ManukaMed LP, Hamilton, New Zealand

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Trevor Watson AgResearch Ltd, Hamilton, New Zealand

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Christopher D McMahon ManukaMed LP, Hamilton, New Zealand
AgResearch Ltd, Hamilton, New Zealand

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Introduction Male mammals generally have a larger body size and muscle mass than females and, in mice, this arises from a faster rate of growth of muscle and bone before epiphysial closure ( Griffin & Goldspink 1973 , Callewaert et al

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-20-0485
Volume/Issue:
Volume 248: Issue 2
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Patricia Forcinito
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Anenisia C Andrade Developmental Endocrinology Branch, Department of Women's and Children's Health, Program in Developmental Endocrinology and Genetics, Eunice Kennedy Shriver National Institute of Child Health and Human Development, National Institutes of Health, CRC, Room 1-3330, 10 Center Drive, MSC-1103, Bethesda, Maryland 20892-1103, USA

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Gabriela P Finkielstain
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Jeffrey Baron
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Ola Nilsson Developmental Endocrinology Branch, Department of Women's and Children's Health, Program in Developmental Endocrinology and Genetics, Eunice Kennedy Shriver National Institute of Child Health and Human Development, National Institutes of Health, CRC, Room 1-3330, 10 Center Drive, MSC-1103, Bethesda, Maryland 20892-1103, USA

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Julian C Lui
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Introduction Mammalian body length is primarily determined by longitudinal bone growth that occurs at the growth plate, which is a thin layer of cartilage found near the ends of long bones. The process of longitudinal bone growth occurs by

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-10-0302
Volume/Issue:
Volume 208: Issue 1
Free access