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Jennifer Chen
Jennifer Chen
The Westmead Institute for Medical Research, The University of Sydney Westmead Hospital, New South Wales, Australia
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Jenny E Gunton
Jenny E Gunton
The Westmead Institute for Medical Research, The University of Sydney Westmead Hospital, New South Wales, Australia
Department of Endocrinology and Diabetes, Westmead Hospital, Sydney, New South Wales, Australia
Garvan Institute of Medical Research, Sydney, New South Wales, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, New South Wales, Australia
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Department of Endocrinology and Diabetes, Westmead Hospital, Sydney, New South Wales, Australia
Garvan Institute of Medical Research, Sydney, New South Wales, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, New South Wales, Australia
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increased β-cell mass and proliferation compared to mice that consumed a normal diet ( Peyot et al. 2010 ). β-cell mass increases during pregnancy, mostly mediated by prolactin and placental lactogen. This is more prominent in mice, compared to humans
Jennifer M Ikle
Jennifer M Ikle
Division of Endocrinology, Department of Pediatrics, Stanford University School of Medicine, Stanford, California, USA
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Anna L Gloyn
Anna L Gloyn
Division of Endocrinology, Department of Pediatrics, Stanford University School of Medicine, Stanford, California, USA
Stanford Diabetes Research Center, Stanford University, Stanford, California, USA
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Stanford Diabetes Research Center, Stanford University, Stanford, California, USA
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. 2014 ). The exception to this is during pregnancy, where glucose-lowering medications may be considered in the case of increased fetal growth observed on ultrasound ( Chakera et al. 2015 ). Hepatocyte nuclear factor 1 alpha (HNF1A-MODY, MODY3
