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Stanford Diabetes Research Center, Stanford University, Stanford, California, USA
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course, this now fits perfectly with our understanding of the differences in the underlying pathophysiology of glucokinase (GCK) vs transcription factor (HNF1A/HNF4A/HNF1B) MODY. At the time of writing this article, the number of genetic subtypes
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Department of Endocrinology and Diabetes, Westmead Hospital, Sydney, New South Wales, Australia
Garvan Institute of Medical Research, Sydney, New South Wales, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, New South Wales, Australia
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was positively correlated with increasing donor body mass index (BMI). As mentioned earlier, in non-diabetic humans, as BMI increases, so does β-cell mass. Another example of this is the ectopic expression of the transcription factor paired box 4 (PAX4
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transcription factor basic leucine zipper transcription factor 2 (BACH2). First identified as a key regulator of Tregs, it was thought to be yet another argument of a deficient immune system in T1D ( Roychoudhuri et al. 2013 ). Later, it was shown that BACH2
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Institute for Molecular Medicine Finland (FIMM), Helsinki University, Helsinki, Finland
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Institute for Molecular Medicine Finland (FIMM), Helsinki University, Helsinki, Finland
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) Grant SF Thorleifsson G Reynisdottir I Benediktsson R Manolescu A Sainz J Helgason A Stefansson H Emilsson V Helgadottir A 2006 Variant of transcription factor 7-like 2 (TCF7L2) gene confers risk of type 2 diabetes . Nature Genetics
