Search Results
Search for other papers by Diego Safian in
Google Scholar
PubMed
Search for other papers by Najoua Ryane in
Google Scholar
PubMed
Search for other papers by Jan Bogerd in
Google Scholar
PubMed
Reproduction and Developmental Biology Group, Institute of Marine Research, Nordnes, Bergen, Norway
Search for other papers by Rüdiger W Schulz in
Google Scholar
PubMed
-stimulating hormone (Fsh) are major modulators of vertebrate spermatogenesis. Upon gonadotropin signaling, somatic cells such as SCs, LCs and myoid cells produce paracrine factors modulating spermatogonial activity ( Oatley et al. 2009 , Ding et al. 2011 , Chen
Search for other papers by Yuxin Xie in
Google Scholar
PubMed
Search for other papers by Lianhe Chu in
Google Scholar
PubMed
Search for other papers by Yun Liu in
Google Scholar
PubMed
Search for other papers by Kathy W Y Sham in
Google Scholar
PubMed
College of Life Sciences, Northwest Normal University, Lanzhou, China
Search for other papers by Jianzhen Li in
Google Scholar
PubMed
Search for other papers by Christopher H K Cheng in
Google Scholar
PubMed
control of gonadotropic hormone stimulation of their receptors in the testes ( Pudney 1995 , Schulz et al . 2010 ). More specifically, both pituitary gonadotropins including luteinizing hormone (LH) and follicle-stimulating hormone (FSH) exert their
Search for other papers by Diego Crespo in
Google Scholar
PubMed
Search for other papers by Moline Severino Lemos in
Google Scholar
PubMed
Institute of Oceanography, Minjiang University, Fuzhou, People’s Republic of China
Search for other papers by Yu Ting Zhang in
Google Scholar
PubMed
Search for other papers by Diego Safian in
Google Scholar
PubMed
Search for other papers by Birgitta Norberg in
Google Scholar
PubMed
Search for other papers by Jan Bogerd in
Google Scholar
PubMed
Research Group Reproduction and Developmental Biology, Institute of Marine Research, Bergen, Norway
Search for other papers by Rüdiger W Schulz in
Google Scholar
PubMed
production ( Frungieri et al. 2015 ). In rat, FSH stimulated Sertoli cell PG production ( Jannini et al. 1994 ). In fetal mice, Sertoli cell-derived PGD 2 keeps spermatogonia in an undifferentiated state by autocrine stimulation of CYP26B1 activity that
Search for other papers by Se-Min Kim in
Google Scholar
PubMed
Search for other papers by Farhath Sultana in
Google Scholar
PubMed
Search for other papers by Steven Sims in
Google Scholar
PubMed
Search for other papers by Judit Gimenez-Roig in
Google Scholar
PubMed
Search for other papers by Victoria Laurencin in
Google Scholar
PubMed
Search for other papers by Anusha Pallapati in
Google Scholar
PubMed
Search for other papers by Satish Rojekar in
Google Scholar
PubMed
Search for other papers by Tal Frolinger in
Google Scholar
PubMed
Search for other papers by Weibin Zhou in
Google Scholar
PubMed
Search for other papers by Anisa Gumerova in
Google Scholar
PubMed
Search for other papers by Anne Macdonald in
Google Scholar
PubMed
Search for other papers by Vitaly Ryu in
Google Scholar
PubMed
Search for other papers by Daria Lizneva in
Google Scholar
PubMed
Search for other papers by Funda Korkmaz in
Google Scholar
PubMed
Search for other papers by Tony Yuen in
Google Scholar
PubMed
Search for other papers by Mone Zaidi in
Google Scholar
PubMed
Introduction Two gonadotropins secreted by the anterior pituitary gland, namely, follicle-stimulating hormone (FSH) and luteinizing hormone (LH), along with thyroid-stimulating hormone (TSH), as well as their respective receptors, exhibit
Shandong Provincial Key Laboratory of Endocrinology and Lipid Metabolism, Jinan, Shandong Province, China
Cheeloo College of Medicine, Shandong University, Jinan, Shandong Province, China
Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, Shandong Province, China
Search for other papers by Yan Wang in
Google Scholar
PubMed
Shandong Provincial Key Laboratory of Endocrinology and Lipid Metabolism, Jinan, Shandong Province, China
Cheeloo College of Medicine, Shandong University, Jinan, Shandong Province, China
Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, Shandong Province, China
Search for other papers by Mengqi Zhang in
Google Scholar
PubMed
Shandong Provincial Key Laboratory of Endocrinology and Lipid Metabolism, Jinan, Shandong Province, China
Cheeloo College of Medicine, Shandong University, Jinan, Shandong Province, China
Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, Shandong Province, China
Search for other papers by Zhikun Huan in
Google Scholar
PubMed
Shandong Provincial Key Laboratory of Endocrinology and Lipid Metabolism, Jinan, Shandong Province, China
Cheeloo College of Medicine, Shandong University, Jinan, Shandong Province, China
Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, Shandong Province, China
Search for other papers by Shanshan Shao in
Google Scholar
PubMed
Shandong Provincial Key Laboratory of Endocrinology and Lipid Metabolism, Jinan, Shandong Province, China
Cheeloo College of Medicine, Shandong University, Jinan, Shandong Province, China
Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, Shandong Province, China
Search for other papers by Xiujuan Zhang in
Google Scholar
PubMed
Search for other papers by Dehuan Kong in
Google Scholar
PubMed
Shandong Provincial Key Laboratory of Endocrinology and Lipid Metabolism, Jinan, Shandong Province, China
Cheeloo College of Medicine, Shandong University, Jinan, Shandong Province, China
Department of Endocrinology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, Shandong Province, China
Search for other papers by Jin Xu in
Google Scholar
PubMed
of OA have been linked to the decrease in estrogen that occurs with the onset of menopause ( Martin-Millan & Castaneda 2013 ). Although FSH levels increase in parallel, the effect of FSH on cartilage and chondrocytes has never been explored. FSH is
Search for other papers by Juliana I Candelaria in
Google Scholar
PubMed
Search for other papers by Maria B Rabaglino in
Google Scholar
PubMed
Search for other papers by Anna C Denicol in
Google Scholar
PubMed
Introduction Ovarian antral follicles require gonadotropin stimulation for development and ovulation whereas preantral follicles seem capable of development in the absence of follicle-stimulating hormone (FSH) and luteinizing hormone (LH
Monash Institute for Medical Research, Monash University, Clayton, Victoria, 3168, Australia
Center for Reproductive Biology, School of Molecular Biosciences, Washington State University, Pullman, Washington, 99164, USA
The Australian Research Council Centre of Excellence in Biotechnology and Development
Search for other papers by Sarah J Meachem in
Google Scholar
PubMed
Monash Institute for Medical Research, Monash University, Clayton, Victoria, 3168, Australia
Center for Reproductive Biology, School of Molecular Biosciences, Washington State University, Pullman, Washington, 99164, USA
The Australian Research Council Centre of Excellence in Biotechnology and Development
Search for other papers by Saleela M Ruwanpura in
Google Scholar
PubMed
Monash Institute for Medical Research, Monash University, Clayton, Victoria, 3168, Australia
Center for Reproductive Biology, School of Molecular Biosciences, Washington State University, Pullman, Washington, 99164, USA
The Australian Research Council Centre of Excellence in Biotechnology and Development
Search for other papers by Jessica Ziolkowski in
Google Scholar
PubMed
Monash Institute for Medical Research, Monash University, Clayton, Victoria, 3168, Australia
Center for Reproductive Biology, School of Molecular Biosciences, Washington State University, Pullman, Washington, 99164, USA
The Australian Research Council Centre of Excellence in Biotechnology and Development
Search for other papers by Jacquelyn M Ague in
Google Scholar
PubMed
Monash Institute for Medical Research, Monash University, Clayton, Victoria, 3168, Australia
Center for Reproductive Biology, School of Molecular Biosciences, Washington State University, Pullman, Washington, 99164, USA
The Australian Research Council Centre of Excellence in Biotechnology and Development
Search for other papers by Michael K Skinner in
Google Scholar
PubMed
Monash Institute for Medical Research, Monash University, Clayton, Victoria, 3168, Australia
Center for Reproductive Biology, School of Molecular Biosciences, Washington State University, Pullman, Washington, 99164, USA
The Australian Research Council Centre of Excellence in Biotechnology and Development
Search for other papers by Kate L Loveland in
Google Scholar
PubMed
spermatogenic wave are under investigation and appear to be influenced by a complex network of interacting signals. It is known that pituitary-derived follicle stimulating hormone (FSH) exerts its effects on the developing germ cells indirectly, as only the
Search for other papers by Marie M Devillers in
Google Scholar
PubMed
Search for other papers by Florence Petit in
Google Scholar
PubMed
Search for other papers by Victoria Cluzet in
Google Scholar
PubMed
Search for other papers by Charlotte M François in
Google Scholar
PubMed
Search for other papers by Frank Giton in
Google Scholar
PubMed
Search for other papers by Ghislaine Garrel in
Google Scholar
PubMed
Search for other papers by Joëlle Cohen-Tannoudji in
Google Scholar
PubMed
Search for other papers by Céline J Guigon in
Google Scholar
PubMed
Introduction In mammals, the infantile period is characterized by a transient elevation in the levels of the two gonadotropins LH (luteinizing hormone) and FSH (follicle-stimulating hormone) shortly after birth ( Döhler & Wuttke 1975
Prince Henry's Institute of Medical Research, Department of Obstetrics and Gynaecology, Monash Institute of Medical Research and ARC Centre of Excellence in Biotechnology and Development, Clayton, Victoria 3168, Australia
Search for other papers by Saleela M Ruwanpura in
Google Scholar
PubMed
Prince Henry's Institute of Medical Research, Department of Obstetrics and Gynaecology, Monash Institute of Medical Research and ARC Centre of Excellence in Biotechnology and Development, Clayton, Victoria 3168, Australia
Search for other papers by Robert I McLachlan in
Google Scholar
PubMed
Search for other papers by Peter G Stanton in
Google Scholar
PubMed
Search for other papers by Kate L Loveland in
Google Scholar
PubMed
Search for other papers by Sarah J Meachem in
Google Scholar
PubMed
endocrine signals including follicle-stimulating hormone (FSH) and luteinizing hormone (LH)/testosterone (T). A key role for FSH in regulating the size of the Sertoli cell population in early postnatal life has been attributed to its stimulatory effect on
Search for other papers by Xin Li in
Google Scholar
PubMed
College of Veterinary Medicine, Yangzhou University, Yangzhou, Jiangsu, People’s Republic of China
Search for other papers by Hongjiao Li in
Google Scholar
PubMed
Search for other papers by Di Zhang in
Google Scholar
PubMed
Search for other papers by Guojin Xu in
Google Scholar
PubMed
Search for other papers by Jinglin Zhang in
Google Scholar
PubMed
College of Veterinary Medicine, Yangzhou University, Yangzhou, Jiangsu, People’s Republic of China
Joint International Research Laboratory of Agriculture and Agri-Product Safety, The Ministry of Education of China, Yangzhou University, Yangzhou, Jiangsu, People’s Republic of China
Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, Yangzhou, Jiangsu, People’s Republic of China
Search for other papers by Sheng Cui in
Google Scholar
PubMed
& Elamraoui 1995 , Kelberman et al. 2009 ). Pituitary gonadotroph secretes luteinizing hormone (LH) and follicle-stimulating hormone (FSH) and regulates mammalian reproductive process through hypothalamus-pituitary-gonadal (HPG) axis ( Christensen et al