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K L Davies Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Street, Cambridge, UK

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J Miles Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Street, Cambridge, UK

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E J Camm Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Street, Cambridge, UK
The Ritchie Centre, Hudson Institute of Medical Research, Clayton, Australia

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D J Smith Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Street, Cambridge, UK

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P Barker MRC Metabolic Diseases Unit, Mouse Biochemistry Laboratory, Cambridge Biomedical Campus, Cambridge, UK

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K Taylor Endocrine Laboratory, Blood Sciences, Cambridge University Hospitals NHS Foundation Trust, Hills Road, Cambridge, UK

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A J Forhead Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Street, Cambridge, UK
Department of Biological and Medical Sciences, Oxford Brookes University, Oxford, UK

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A L Fowden Department of Physiology, Development and Neuroscience, University of Cambridge, Downing Street, Cambridge, UK

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) However, relatively little is known about the long-term effects of naturally occurring increments in fetal cortisol concentrations of the magnitude seen in response to prenatal environmental challenges known to programme adult metabolic and endocrine

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-23-0326
Volume/Issue:
Volume 260: Issue 3
Open access
Zengmin Wang Department of Pediatrics, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong, PR China
Division of Pediatric Endocrinology, University of Alabama at Birmingham, Birmingham, Alabama, USA

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Gail J Mick Division of Pediatric Endocrinology, University of Alabama at Birmingham, Birmingham, Alabama, USA

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Rongrong Xie Division of Pediatric Endocrinology, University of Alabama at Birmingham, Birmingham, Alabama, USA
Department of Endocrinology, Children’s Hospital of Soochow University, Suzhou, Jiangsu, PR China

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Xudong Wang Division of Pediatric Endocrinology, University of Alabama at Birmingham, Birmingham, Alabama, USA

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Xuemei Xie Division of Pediatric Endocrinology, University of Alabama at Birmingham, Birmingham, Alabama, USA

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Guimei Li Department of Pediatrics, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, Shandong, PR China

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Kenneth L McCormick Division of Pediatric Endocrinology, University of Alabama at Birmingham, Birmingham, Alabama, USA

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Introduction Circulating adrenal-derived cortisol, which generated intracellularly from cortisone in various tissues, contributes to maintain blood glucose homeostasis. Only the active 11-hydroxy derivatives (cortisol and corticosterone), and

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0006
Volume/Issue:
Volume 229: Issue 1
Free access
Simon Travers Université Paris-Saclay, Inserm, Physiologie et Physiopathologie Endocriniennes, Le Kremlin-Bicêtre, France
Service de Génétique Moléculaire, Pharmacogénétique et Hormonologie, Hôpital de Bicêtre, Assistance Publique-Hôpitaux de Paris, Le Kremlin Bicêtre, France

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Laetitia Martinerie Université Paris-Saclay, Inserm, Physiologie et Physiopathologie Endocriniennes, Le Kremlin-Bicêtre, France
Service d’Endocrinologie Pédiatrique, Hôpital Robert Debré, Assistance Publique Hôpitaux de Paris, Paris, France
Université de Paris, Faculté de Santé, UFR de Médecine, Paris, France
PremUp Fondation, Paris, France

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Qiong-Yao Xue Université Paris-Saclay, Inserm, Physiologie et Physiopathologie Endocriniennes, Le Kremlin-Bicêtre, France
Service de Génétique Moléculaire, Pharmacogénétique et Hormonologie, Hôpital de Bicêtre, Assistance Publique-Hôpitaux de Paris, Le Kremlin Bicêtre, France

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Julie Perrot Université Paris-Saclay, Inserm, Physiologie et Physiopathologie Endocriniennes, Le Kremlin-Bicêtre, France

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Say Viengchareun Université Paris-Saclay, Inserm, Physiologie et Physiopathologie Endocriniennes, Le Kremlin-Bicêtre, France

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Kathleen M Caron Department of Cell Biology and Physiology, School of Medicine, University of North Carolina, Chapel Hill, North Carolina, USA

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Elizabeth S Blakeney Department of Cell Biology and Physiology, School of Medicine, University of North Carolina, Chapel Hill, North Carolina, USA

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Pascal Boileau Université Paris-Saclay, Inserm, Physiologie et Physiopathologie Endocriniennes, Le Kremlin-Bicêtre, France
Department of Neonatal Pediatrics, Centre Hospitalier Intercommunal de Poissy-Saint-Germain, Poissy, France
UFR des Sciences de la Santé, Simone Veil. Université Versailles St-Quentin en Yvelines, Montigny le Bretonneux, France

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Marc Lombès Université Paris-Saclay, Inserm, Physiologie et Physiopathologie Endocriniennes, Le Kremlin-Bicêtre, France
PremUp Fondation, Paris, France

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Eric Pussard Université Paris-Saclay, Inserm, Physiologie et Physiopathologie Endocriniennes, Le Kremlin-Bicêtre, France
Service de Génétique Moléculaire, Pharmacogénétique et Hormonologie, Hôpital de Bicêtre, Assistance Publique-Hôpitaux de Paris, Le Kremlin Bicêtre, France

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analysis by liquid chromatography coupled to tandem mass spectrometry (LC-MS/MS) revealed in this cohort a defect of both cortisol and aldosterone secretion in very preterm neonates. This defect was supported by a partial yet transient deficiency in 11β

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-20-0564
Volume/Issue:
Volume 251: Issue 1
Restricted access
Jason P Breves Department of Biology, Skidmore College, Saratoga Springs, New York, USA

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Mayu Inokuchi Hawai‘i Institute of Marine Biology, University of Hawai‘i at Mānoa, Kāne‘ohe, Hawai‘i, USA
Department of Aquatic Bioscience, Graduate School of Agricultural and Life Sciences, University of Tokyo, Bunkyo, Tokyo, Japan

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Yoko Yamaguchi Hawai‘i Institute of Marine Biology, University of Hawai‘i at Mānoa, Kāne‘ohe, Hawai‘i, USA

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Andre P Seale Hawai‘i Institute of Marine Biology, University of Hawai‘i at Mānoa, Kāne‘ohe, Hawai‘i, USA

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Bethany L Hunt Department of Biology, Skidmore College, Saratoga Springs, New York, USA

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Soichi Watanabe Department of Aquatic Bioscience, Graduate School of Agricultural and Life Sciences, University of Tokyo, Bunkyo, Tokyo, Japan

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Darren T Lerner Hawai‘i Institute of Marine Biology, University of Hawai‘i at Mānoa, Kāne‘ohe, Hawai‘i, USA
University of Hawai‘i Sea Grant College Program, University of Hawai‘i at Mānoa, Honolulu, Hawai‘i, USA

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Toyoji Kaneko Department of Aquatic Bioscience, Graduate School of Agricultural and Life Sciences, University of Tokyo, Bunkyo, Tokyo, Japan

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E Gordon Grau Hawai‘i Institute of Marine Biology, University of Hawai‘i at Mānoa, Kāne‘ohe, Hawai‘i, USA

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promotes phenotypes associated with FW acclimation, whereas growth hormone (Gh) supports phenotypes associated with SW acclimation ( Hirano 1986 , McCormick 2001 ). Cortisol, under the control of adrenocorticotropic hormone, can seemingly support both FW

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0162
Volume/Issue:
Volume 230: Issue 3
Free access
Raymond W M Kwong Department of Biology, University of Ottawa, 30 Marie Curie, Ottawa, Ontario, Canada K1N 6N5

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Steve F Perry Department of Biology, University of Ottawa, 30 Marie Curie, Ottawa, Ontario, Canada K1N 6N5

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Introduction Cortisol is the major corticosteroid in fish, and its effects on transcellular ion transport are well documented (see review by McCormick (2001) ). Increasing evidence suggests that cortisol may also regulate paracellular permeability

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0574
Volume/Issue:
Volume 217: Issue 3
Free access
K L Davies Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK

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E J Camm Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK
The Ritchie Centre, Hudson Institute of Medical Research, Clayton, Australia

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D J Smith Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK

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O R Vaughan Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK
Institute for Women’s Health, University College London, London, UK

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A J Forhead Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK
Department of Biological and Medical Sciences, Oxford Brookes University, Oxford, UK

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A J Murray Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK

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A L Fowden Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK

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study in fetal sheep has demonstrated that the natural prepartum cortisol surge closely parallels the increase in mitochondrial OXPHOS capacity of skeletal muscle towards term ( Davies et al. 2020 ). However, whether these changes are the direct

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-21-0171
Volume/Issue:
Volume 251: Issue 1
Open access
Lesley A Hill Departments of Cellular and Physiological Sciences and Obstetrics and Gynaecology, The University of British Columbia, Vancouver, British Columbia, Canada

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Dimitra A Vassiliadi Endocrine Unit, Second Department of Internal Medicine-Research Institute and Diabetes Center, Attiko University Hospital, Athens, Greece

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Ioanna Dimopoulou Endocrine Unit, Second Department of Internal Medicine-Research Institute and Diabetes Center, Attiko University Hospital, Athens, Greece

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Anna J Anderson BHF Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom

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Luke D Boyle BHF Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom

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Alixe H M Kilgour BHF Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom

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Roland H Stimson BHF Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom

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Yoan Machado Department of Biochemistry and Molecular Biology, The University of British Columbia, Vancouver, British Columbia, Canada

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Christopher M Overall Department of Biochemistry and Molecular Biology, The University of British Columbia, Vancouver, British Columbia, Canada

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Brian R Walker BHF Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom
Institute of Genetic Medicine, Newcastle University, Newcastle upon Tyne, United Kingdom

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John G Lewis Canterbury Health Laboratories, Christchurch, New Zealand

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Geoffrey L Hammond Departments of Cellular and Physiological Sciences and Obstetrics and Gynaecology, The University of British Columbia, Vancouver, British Columbia, Canada

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(RIAs) correlate well with measurements of its cortisol-binding capacity, except in samples containing CBG variants with abnormal steroid-binding properties ( Robinson & Hammond 1985 , Smith et al. 1992 , Emptoz-Bonneton et al. 2000 , Perogamvros

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0479
Volume/Issue:
Volume 240: Issue 1
Open access
Timothy J Cole Department of Biochemistry and Molecular Biology, Monash University, Melbourne, Victoria, Australia
Centre for Endocrinology and Metabolism, Hudson Institute of Medical Research, Monash Medical Centre, Clayton, Victoria, Australia

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Morag J Young Centre for Endocrinology and Metabolism, Hudson Institute of Medical Research, Monash Medical Centre, Clayton, Victoria, Australia
Department of Molecular and Translational Research, Monash University, Melbourne, Victoria, Australia

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Introduction The mineralocorticoid receptor (MR) is an intracellular steroid hormone receptor, and a member of the nuclear receptor superfamily, that mediates the physiological action of two important adrenal steroids, aldosterone and cortisol

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-17-0155
Volume/Issue:
Volume 234: Issue 1
Free access
Yusuke Kumai
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Nicholas J Bernier Department of Biology, Department of Integrative Biology, University of Ottawa, 30 Marie Curie, Ottawa, Ontario, Canada K1N 6N5

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Steve F Perry
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is tightly regulated by several hormones, including prolactin ( Pickford & Phillips 1959 , Breves et al . 2010 ), cortisol ( Laurent & Perry 1990 , Lin et al . 2011 , Kumai et al . 2012 a ), stanniocalcin ( Tseng et al . 2009 ), vitamin D ( Lin

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0374
Volume/Issue:
Volume 220: Issue 3
Free access
Neele S Dellschaft Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK
Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK

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Marie-Cecile Alexandre-Gouabau Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK

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David S Gardner Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK

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Jean-Philippe Antignac Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK

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Duane H Keisler Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK

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Helen Budge Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK

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Michael E Symonds Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK

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Sylvain P Sebert Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK
Early Life Research Unit, INRA and University of Nantes, School of Veterinary Medicine and Science, Institute of Health Sciences, Oniris, Department of Animal Science, Academic Division of Child Health, Obstetrics and Gynaecology, School of Medicine, Queen's Medical Centre, The University of Nottingham, Nottingham NG7 2UH, UK

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stresses in early life and plays a central role in the regulation of energy homeostasis ( Adam et al . 2008 ). Cortisol can influence blood pressure as a consequence of regulating gene expression of arginine vasopressin ( AVP ) and corticotropin

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0600
Volume/Issue:
Volume 224: Issue 2
Free access