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Sandra Ghelardoni, Grazia Chiellini, Sabina Frascarelli, Alessandro Saba and Riccardo Zucchi

investigation, we observed that T 1 AM, such as T 3 and T 4 , can be taken up and accumulated in hepatocytes. In HepG2 cell culture, these compounds were quickly absorbed because their presence was detected in cell lysate after a few minutes of infusion. At the

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Xue Jiang, Jia Xiao, Mulan He, Ani Ma and Anderson O L Wong

shed light on the role of SOCS in GH functions in fish model, type II SOCS, including Socs1–3 and Cish, were cloned in grass carp and their gene copy number and tissue expression, especially in the liver, were determined. Using carp hepatocytes, the

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Erin Faught, Lynsi Henrickson and Mathilakath M Vijayan

the possibility that this hormone may modulate exosomal Hsp70 expression using trout hepatocytes in primary culture ( Faught & Vijayan 2016 ). Materials and methods Experimental fish In vivo heat shock study was carried out with juvenile

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Norio Masumoto, Chise Tateno, Asato Tachibana, Rie Utoh, Yoshio Morikawa, Takashi Shimada, Hiroyuki Momisako, Toshiyuki Itamoto, Toshimasa Asahara and Katsutoshi Yoshizato

Introduction Using rodents as experimental animals, it has been shown that differentiated hepatocytes can re-enter into the cell cycle when stimulated by growth factors, cytokines, and hormones ( Taub 1996 , Michalopoulos

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Teresa Priego, Miriam Granado, Estibaliz Castillero, Ana Isabel Martín, M Ángeles Villanúa and Asunción López-Calderón

in the liver ( Granado et al. 2006 ). Taking into account that a direct inhibitory effect of cytokines on IGF-I gene expression in hepatocyte cultures has been reported ( Wolf et al. 1996 , Thissen & Verniers 1997 ), the inhibitory effect of LPS

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O A Sukocheva and D O Carpenter

. 1999 , Di Fulvio et al. 2000 , Shih et al. 2004 ) – established survival regulator of Fas-induced apoptosis ( Gibson et al. 1999 ). However, the effects of THs on Fas-induced apoptosis in hepatocytes have not yet been addressed. Death

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Abigail Wolf Greenstein, Neena Majumdar, Peng Yang, Papasani V Subbaiah, Rhonda D Kineman and Jose Cordoba-Chacon

Introduction Non-alcoholic fatty liver disease (NAFLD) is defined as excessive accumulation of fat (steatosis) within hepatocytes that is independent of alcohol intake. NAFLD increases the risk of diabetes, non-alcoholic steatohepatitis (NASH

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A Catizone, G Ricci, J Del Bravo and M Galdieri

. 1991 , Boitani et al. 1995 , Huleihel & Lunenfeld 2004 , Kubota et al. 2004 , Puglisi et al. 2004 ). Hepatocyte growth factor (HGF), originally identified as a mitogenic factor for hepatocytes ( Nakamura et al. 1984

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Rob Zachow and Mehmet Uzumcu

The hepatocyte growth factor (HGF) system HGF is an 87 kDa growth factor first identified and characterized due to its angiogenic and proliferative effects in primary epithelial cell cultures ( Bussolino et al . 1992 ). This pioneered a number of

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A L Pierce, H Fukada and W W Dickhoff

are pituitary GH and liver-derived endocrine insulin-like growth factor-I (IGF-I). IGF-I circulates bound to a number of IGF-binding proteins (IGFBPs), which modulate its growth-stimulatory effects. GH stimulates the production of IGF-I by hepatocytes