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Hamza Amine, Yacir Benomar, Adil Haimeur, Hafida Messaouri, Nadia Meskini, and Mohammed Taouis

during a high fat diet in rats ( Storlien et al . 1991 ). Indeed, the substitution of omega-3 fatty acids (w-3 FAs) from fish oil for other types of lipids prevents insulin resistance ( Kraegen et al . 1991 , Jucker et al . 1999 ) and has been

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Tanja Jene, Inigo Ruiz de Azua, Annika Hasch, Jennifer Klüpfel, Julia Deuster, Mirjam Maas, Cora H Nijboer, Beat Lutz, Marianne B Müller, and Michael A van der Kooij

diabetes ( Kelly & Ismail 2015 ). Therefore, we hypothesize that the observed metabolic disturbances induced by CSD represents a prodromal phase where the contribution of a nutritional challenge, in the form of a high-fat diet, could lead to the progression

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Eliana H Akamine, Anderson C Marçal, João Paulo Camporez, Mara S Hoshida, Luciana C Caperuto, Estela Bevilacqua, and Carla R O Carvalho

to analyze the effect of high-fat diet-induced obesity on the insulin signaling in the ovary. We also verified if insulin signaling impairment is time dependent in relation to the period during which the ovary was submitted to adverse effects of

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Xiao-Bing Cui, Jun-Na Luan, Jianping Ye, and Shi-You Chen

demonstrate that high-fat diet (HFD) dramatically induces RGC32 expression in the adipose tissue. RGC32 deficiency attenuates HFD-induced obesity and insulin resistance in mice. The beneficial effect of RGC32 deficiency is due to the decreased adipose tissue

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Eriko Inoue, Toshihiro Ichiki, Kotaro Takeda, Hirohide Matsuura, Toru Hashimoto, Jiro Ikeda, Aya Kamiharaguchi, and Kenji Sunagawa

regulate normal signaling through the insulin receptor/IRS1 axis ( Hotamisligil et al . 1996 ). Mice lacking chemokine receptor-2 (CCR2), a receptor for MCP1, are partly protected against developing high-fat diet (HFD)-induced insulin resistance and

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Bo Ahrén, Maria Sörhede Winzell, and Giovanni Pacini

basis, total energy 16.1 kJ/g) or a high-fat diet (fat 60%, carbohydrate 20%, protein 20% on an energy basis, total energy 22.0 kJ/g) for 8 weeks. The main difference between the two diets is that the high-fat diet has a high content of lard (317 g

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Shibin Ding, Ying Fan, Nana Zhao, Huiqin Yang, Xiaolei Ye, Dongliang He, Xin Jin, Jian Liu, Chong Tian, Hongyu Li, Shunqing Xu, and Chenjiang Ying

be really safe. In recent years, life styles dominated by an increase in the consumption of high-fat diets have contributed to the accelerated diabetes epidemic ( Hu 2011 ), and consumption of fat-rich products favors co-exposure to BPA and HFD

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Zhiguo Liu, Chun Yan Lim, Michelle Yu-Fah Su, Stephanie Li Ying Soh, Guanghou Shui, Markus R Wenk, Kevin L Grove, George K Radda, Weiping Han, and Xiaoqiu Xiao

, Habbout et al . 2012 ). CPO manipulation results in leptin resistance specifically in the arcuate nucleus of hypothalamus during the early postnatal period that persists into adulthood. When fed with a high-fat diet (HFD), CPO mice exhibit hyperphagia and

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Rumana Yasmeen, Qiwen Shen, Aejin Lee, Jacob H Leung, Devan Kowdley, David J DiSilvestro, Lu Xu, Kefeng Yang, Andrei Maiseyeu, Naresh C Bal, Muthu Periasamy, Paolo Fadda, and Ouliana Ziouzenkova

mice (WT) were fed a high-fat diet (HF, 45% kcal from fat, D12451, Research Diet Inc.) for 30 days. Then, mice were randomly assigned into: A control group injected with 0.1 mL sterile phosphate buffered saline (PBS) into both epididymal iAb fat

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Sara Baldassano, Antonella Amato, Francesco Cappello, Francesca Rappa, and Flavia Mulè

, Ørskov et al . 2005 , Guan et al . 2006 ). Reports by several laboratories indicate that high-fat diet (HFD) maintenance promotes the capacity of the gastrointestinal tract to digest and absorb fats, through pronounced effects on the intestinal