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Lucy V Norling
Lucy V Norling
Barts and The London School of Medicine, The William Harvey Research Institute, Centre for Biochemical Pharmacology, Queen Mary University of London, Charterhouse Square, London EC1M 6BQ, UK
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Mauro Perretti
Mauro Perretti
Barts and The London School of Medicine, The William Harvey Research Institute, Centre for Biochemical Pharmacology, Queen Mary University of London, Charterhouse Square, London EC1M 6BQ, UK
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Dianne Cooper
Dianne Cooper
Barts and The London School of Medicine, The William Harvey Research Institute, Centre for Biochemical Pharmacology, Queen Mary University of London, Charterhouse Square, London EC1M 6BQ, UK
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functions involved in immune and inflammatory responses and tumour development have been reported for galectins over the last decade including roles in cellular adhesion, migration and survival (see Elola et al . 2007 , Yang et al . 2008 for recent
Jing Lu
Jing Lu
Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
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Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
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Joshua Reese
Joshua Reese
Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
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Ying Zhou
Ying Zhou
Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
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Emmet Hirsch
Emmet Hirsch
Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
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Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
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P4BSA-induced inflammatory responses. Figure 4 Effects of PD98059 on P4BSA-stimulated activation of MEK1/2 in macrophages. RAW 264.7 cells were pretreated in the absence or presence of the MEK1/2 inhibitor PD98059 (2.0×10 −5 mol/l) for 1 h and then
Shisan Xu
Shisan Xu
Department of Biomedical Sciences, College of Veterinary Medicine and Life Science, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Fangjing Xie
Fangjing Xie
Department of Biomedical Sciences, College of Veterinary Medicine and Life Science, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Li Tian
Li Tian
Department of Biomedical Sciences, College of Veterinary Medicine and Life Science, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Samane Fallah
Samane Fallah
Department of Biomedical Sciences, College of Veterinary Medicine and Life Science, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Fatemeh Babaei
Fatemeh Babaei
Department of Chemistry, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Sinai H C Manno
Sinai H C Manno
Department of Biomedical Sciences, College of Veterinary Medicine and Life Science, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Francis A M Manno III
Francis A M Manno III
School of Biomedical Engineering, Faculty of Engineering, University of Sydney, Sydney, New South Wales, Australia
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Lina Zhu
Lina Zhu
Department of Biomedical Sciences, College of Veterinary Medicine and Life Science, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Kin Fung Wong
Kin Fung Wong
Department of Biomedical Engineering, Polytechnic University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Yimin Liang
Yimin Liang
Department of Chemistry, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Rajkumar Ramalingam
Rajkumar Ramalingam
Department of Chemistry, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Lei Sun
Lei Sun
Department of Biomedical Engineering, Polytechnic University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Xin Wang
Xin Wang
Department of Biomedical Sciences, College of Veterinary Medicine and Life Science, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Robert Plumb
Robert Plumb
Waters Technologies Corporation, Milford, Massachusetts, USA
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Lee Gethings
Lee Gethings
Waters Technologies Corporation, Milford, Massachusetts, USA
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Yun Wah Lam
Yun Wah Lam
Department of Chemistry, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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Shuk Han Cheng
Shuk Han Cheng
Department of Biomedical Sciences, College of Veterinary Medicine and Life Science, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
State Key Laboratory of Marine Pollution (SKLMP) at City University of Hong Kong, Hong Kong SAR, People’s Republic of China
Department of Materials Science and Engineering, College of Science and Engineering, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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State Key Laboratory of Marine Pollution (SKLMP) at City University of Hong Kong, Hong Kong SAR, People’s Republic of China
Department of Materials Science and Engineering, College of Science and Engineering, City University of Hong Kong, Hong Kong SAR, People’s Republic of China
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. Estrogen enhances immune and inflammatory responses in regenerating heart To elucidate the mechanism underlying the sexual difference in zebrafish heart regeneration, we performed comparative transcriptomic analyses on RNA extracted from female and male
Jiali Liu
Jiali Liu
Department of Clinical Laboratory, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
Leonard Davis School of Gerontology, University of Southern California, Los Angeles, California, USA
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Leonard Davis School of Gerontology, University of Southern California, Los Angeles, California, USA
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Yue Li
Yue Li
Department of Clinical Laboratory, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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Xiaoyan Zhou
Xiaoyan Zhou
Department of Clinical Laboratory, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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Xi Zhang
Xi Zhang
Department of Neurosurgery, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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Hao Meng
Hao Meng
Department of Clinical Laboratory, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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Sanyuan Liu
Sanyuan Liu
Department of Clinical Laboratory, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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Lei Zhang
Lei Zhang
Department of Clinical Laboratory, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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Juntao He
Juntao He
Department of Health Office, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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Qian He
Qian He
Department of Clinical Laboratory, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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Yan Geng
Yan Geng
Department of Clinical Laboratory, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, Shaanxi, China
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.01. Injection of CaMKIV involved in chronic inflammatory response To further investigate alleviation of the hepatosteatotic phenotype, we next analyzed key metabolic and inflammatory myokines in serum and their mRNA and protein expression in mice liver tissues
Cheng-Hsuan Tsai
Cheng-Hsuan Tsai
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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Zheng-Wei Chen
Zheng-Wei Chen
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital Yun-Lin Branch, Yun-Lin, Taiwan
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Bo-Ching Lee
Bo-Ching Lee
Department of Medical Imaging, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Che-Wei Liao
Che-Wei Liao
Department of Medicine, National Taiwan University Cancer Center, Taipei, Taiwan
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Yi-Yao Chang
Yi-Yao Chang
Cardiology Division of Cardiovascular Medical Center, Far Eastern Memorial Hospital, New Taipei City, Taiwan
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Yan-Rou Tsai
Yan-Rou Tsai
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Chia-Hung Chou
Chia-Hung Chou
Department of Obstetrics and Gynecology, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Vin-Cent Wu
Vin-Cent Wu
National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
Division of Nephrology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Division of Nephrology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Chi-Sheng Hung
Chi-Sheng Hung
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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Yen-Hung Lin
Yen-Hung Lin
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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mitochondrial dysfunction and the potential impacts on cardiovascular and metabolic dysfunction. Aldosterone activates the mineralocorticoid receptor (MR), which induces an increase in reactive oxygen species (ROS) and an inflammatory response, leading to
Anne Q Reuwer
Anne Q Reuwer
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Marco van Eijk
Marco van Eijk
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Felicia M Houttuijn-Bloemendaal
Felicia M Houttuijn-Bloemendaal
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Chris M van der Loos
Chris M van der Loos
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Nike Claessen
Nike Claessen
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Peter Teeling
Peter Teeling
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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John J P Kastelein
John J P Kastelein
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Jörg Hamann
Jörg Hamann
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Vincent Goffin
Vincent Goffin
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Jan H von der Thüsen
Jan H von der Thüsen
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Marcel Th B Twickler
Marcel Th B Twickler
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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Jan Aten
Jan Aten
Departments of, Vascular Medicine, Medical Biochemistry, Pathology, Experimental Immunology, INSERM, Department of Internal Medicine, Academic Medical Center, 1105 AZ Amsterdam, The Netherlands
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prolactin receptor pathway may contribute to the inflammatory response. Based on the available literature and our current results, we propose that prolactin plays the following role in the pathophysiology of atherosclerosis: increased prolactin receptor
Caterina Luana Pitasi
Caterina Luana Pitasi
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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Junjun Liu
Junjun Liu
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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Blandine Gausserès
Blandine Gausserès
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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Gaëlle Pommier
Gaëlle Pommier
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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Etienne Delangre
Etienne Delangre
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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Mathieu Armanet
Mathieu Armanet
Cell Therapy Unit, Saint-Louis hospital, AP-HP, University Paris-Diderot, Paris, France
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Pierre Cattan
Pierre Cattan
Cell Therapy Unit, Saint-Louis hospital, AP-HP, University Paris-Diderot, Paris, France
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Bruno Mégarbane
Bruno Mégarbane
INSERM UMRS1144, Université Paris-Descartes, Université Paris-Diderot, Paris, France
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Anne-Sophie Hanak
Anne-Sophie Hanak
INSERM UMRS1144, Université Paris-Descartes, Université Paris-Diderot, Paris, France
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Kamel Maouche
Kamel Maouche
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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Danielle Bailbé
Danielle Bailbé
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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Bernard Portha
Bernard Portha
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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Jamileh Movassat
Jamileh Movassat
Université Paris Diderot, BFA, UMR 8251, CNRS, Team ‘Biologie et Pathologie du Pancréas Endocrine’, Paris, France
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tissues of patients with T2D also revealed the presence of immune cells within the endocrine pancreas ( Donath et al. 2008 ). Diabetes-associated glucolipotoxicity has been shown to induce an inflammatory response in the islets, notably by inducing
Gordon J Allan
Gordon J Allan
SIPBS, University of Strathclyde, Glasgow G1 1XW, UK
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James Beattie
James Beattie
SIPBS, University of Strathclyde, Glasgow G1 1XW, UK
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David J Flint
David J Flint
SIPBS, University of Strathclyde, Glasgow G1 1XW, UK
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characterized by excessive deposition of ECM, fibrotic diseases are also accompanied by an inflammatory response involving monocyte/macrophage activation, and thus attempts to control the progress of these diseases with anti-inflammatory therapies have been
Wailan Shan
Wailan Shan
Institute of Biomedicine, Department of Cellular Biology, Jinan University, Guangzhou, China
National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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Shiyin Lu
Shiyin Lu
Institute of Biomedicine, Department of Cellular Biology, Jinan University, Guangzhou, China
National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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Biqian Ou
Biqian Ou
Institute of Biomedicine, Department of Cellular Biology, Jinan University, Guangzhou, China
National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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Jia Feng
Jia Feng
Institute of Biomedicine, Department of Cellular Biology, Jinan University, Guangzhou, China
National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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Zixian Wang
Zixian Wang
Institute of Biomedicine, Department of Cellular Biology, Jinan University, Guangzhou, China
National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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Huixian Li
Huixian Li
Institute of Biomedicine, Department of Cellular Biology, Jinan University, Guangzhou, China
National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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Xiaohua Lu
Xiaohua Lu
The First Affiliated Hospital of Jinan University, Guangzhou, China
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Yi Ma
Yi Ma
Institute of Biomedicine, Department of Cellular Biology, Jinan University, Guangzhou, China
National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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National Engineering Research Center of Genetic Medicine, Key Laboratory of Bioengineering Medicine of Guangdong Province, Jinan University, Guangzhou, China
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NC or PA; ns, no significant difference. Data are presented as means ± s.d. PA, palmitic acid; PACAP, pituitary adenylate cyclase activating peptide. PACAP alleviated PA-induced oxidative stress, apoptosis and inflammatory responses in
Milos Mraz
Milos Mraz
Third Department of Medicine – Department of Endocrinology and Metabolism, General University Hospital, First Faculty of Medicine of Charles University in Prague, U nemocnice 1, 128 00 Prague 2, Czech Republic
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Martin Haluzik
Martin Haluzik
Third Department of Medicine – Department of Endocrinology and Metabolism, General University Hospital, First Faculty of Medicine of Charles University in Prague, U nemocnice 1, 128 00 Prague 2, Czech Republic
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generalized inflammatory response), obesity-induced inflammation exerts profound effects on metabolic pathways, playing one of the central roles in the development of IR ( Heilbronn & Campbell 2008 , Oliver et al . 2010 ). Although a connection between
