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M Kurtz Laboratory of Reproduction and Metabolism, UMYMFOR (CONICET‐UBA), CEFyBO‐CONICET, School of Medicine and

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E Capobianco Laboratory of Reproduction and Metabolism, UMYMFOR (CONICET‐UBA), CEFyBO‐CONICET, School of Medicine and

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V Careaga Laboratory of Reproduction and Metabolism, UMYMFOR (CONICET‐UBA), CEFyBO‐CONICET, School of Medicine and

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N Martinez Laboratory of Reproduction and Metabolism, UMYMFOR (CONICET‐UBA), CEFyBO‐CONICET, School of Medicine and

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M B Mazzucco Laboratory of Reproduction and Metabolism, UMYMFOR (CONICET‐UBA), CEFyBO‐CONICET, School of Medicine and

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M Maier Laboratory of Reproduction and Metabolism, UMYMFOR (CONICET‐UBA), CEFyBO‐CONICET, School of Medicine and

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A Jawerbaum Laboratory of Reproduction and Metabolism, UMYMFOR (CONICET‐UBA), CEFyBO‐CONICET, School of Medicine and

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2009 , Ali & Dornhorst 2011 ). Both glucose and lipid metabolic substrates transferred in excess from maternal circulation into the fetuses have been involved in the generation of a proinflammatory environment that challenges the development of the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0362
Volume/Issue:
Volume 220: Issue 3
Free access
Daiana Fornes Universidad de Buenos Aires, Facultad de Medicina, Buenos Aires, Argentina
CONICET – Universidad de Buenos Aires, Laboratory of Reproduction and Metabolism, CEFYBO, Buenos Aires, Argentina

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Florencia Heinecke Universidad de Buenos Aires, Facultad de Medicina, Buenos Aires, Argentina
CONICET – Universidad de Buenos Aires, Laboratory of Programming of Metabolic Anomalies, CEFYBO, Buenos Aires, Argentina

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Cintia Romina Gatti Universidad de Buenos Aires, Facultad de Medicina, Buenos Aires, Argentina
CONICET – Universidad de Buenos Aires, Laboratory of Reproduction and Metabolism, CEFYBO, Buenos Aires, Argentina

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Sabrina Lorena Roberti Universidad de Buenos Aires, Facultad de Medicina, Buenos Aires, Argentina
CONICET – Universidad de Buenos Aires, Laboratory of Reproduction and Metabolism, CEFYBO, Buenos Aires, Argentina

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Verónica White Universidad de Buenos Aires, Facultad de Medicina, Buenos Aires, Argentina
CONICET – Universidad de Buenos Aires, Laboratory of Programming of Metabolic Anomalies, CEFYBO, Buenos Aires, Argentina

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Alicia Jawerbaum Universidad de Buenos Aires, Facultad de Medicina, Buenos Aires, Argentina
CONICET – Universidad de Buenos Aires, Laboratory of Reproduction and Metabolism, CEFYBO, Buenos Aires, Argentina

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Evangelina Capobianco Universidad de Buenos Aires, Facultad de Medicina, Buenos Aires, Argentina
CONICET – Universidad de Buenos Aires, Laboratory of Reproduction and Metabolism, CEFYBO, Buenos Aires, Argentina

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of intrauterine growth is the placental supply of maternal nutrients and oxygen to the fetus. Placental lipid content depends on the maternal supply and placental de novo lipid synthesis ( Capobianco et al. 2005 , Lager & Powell 2012 ). The

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-21-0301
Volume/Issue:
Volume 254: Issue 1
Restricted access
Qiong Lv Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Rufei Gao Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China
Laboratory of Lipids and Glucose Metabolism, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Chuan Peng School of Public Health and Management, Chongqing Medical University, Chongqing, China

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Juan Yi Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Lulu Liu Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Shumin Yang Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Danting Li Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Jinbo Hu Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Ting Luo Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Mei Mei Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Ying Song Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Chaodong Wu Department of Nutrition and Food Science, Texas A&M University, College Station, Texas, USA

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Xiaoqiu Xiao School of Public Health and Management, Chongqing Medical University, Chongqing, China

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Qifu Li Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China

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Introduction Excess hepatic deposition of lipids, especially triglyceride (TG), is one of the important features of nonalcoholic fatty liver disease (NAFLD). Over-nutrition, metabolic diseases and genetic factors were widely considered to

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-17-0028
Volume/Issue:
Volume 234: Issue 2
Free access
Mark A Exley Department of Medicine, Faculty of Medical and Human Sciences, Department of Endocrinology, Department of Medicine, Brigham and Women's Hospital, Thorn Bldg, 1405, Harvard Medical School, 75 Francis Street, Boston, Massachusetts 02115, USA
Department of Medicine, Faculty of Medical and Human Sciences, Department of Endocrinology, Department of Medicine, Brigham and Women's Hospital, Thorn Bldg, 1405, Harvard Medical School, 75 Francis Street, Boston, Massachusetts 02115, USA

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Laura Hand Department of Medicine, Faculty of Medical and Human Sciences, Department of Endocrinology, Department of Medicine, Brigham and Women's Hospital, Thorn Bldg, 1405, Harvard Medical School, 75 Francis Street, Boston, Massachusetts 02115, USA

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Donal O'Shea Department of Medicine, Faculty of Medical and Human Sciences, Department of Endocrinology, Department of Medicine, Brigham and Women's Hospital, Thorn Bldg, 1405, Harvard Medical School, 75 Francis Street, Boston, Massachusetts 02115, USA

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Lydia Lynch Department of Medicine, Faculty of Medical and Human Sciences, Department of Endocrinology, Department of Medicine, Brigham and Women's Hospital, Thorn Bldg, 1405, Harvard Medical School, 75 Francis Street, Boston, Massachusetts 02115, USA

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inflammation is triggered is not completely understood, but it is suggested that lipotoxicity ( Unger & Scherer 2010 ), endoplasmic reticulum (ER) stress due to excess lipid burden ( Ozcan et al . 2004 ), hypoxia due to decreased oxygen diffusion into enlarged

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-13-0516
Volume/Issue:
Volume 223: Issue 2
Free access
Affiong Ika Oqua Section of Cell Biology and Functional Genomics, Division of Diabetes, Endocrinology and Metabolism, Department of Metabolism, Digestion and Reproduction, Imperial College London, London, UK

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Yusman Manchanda Section of Cell Biology and Functional Genomics, Division of Diabetes, Endocrinology and Metabolism, Department of Metabolism, Digestion and Reproduction, Imperial College London, London, UK

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Emma Rose McGlone Department of Surgery and Cancer, Imperial College London, London, UK

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Ben Jones Section of Investigative Medicine, Division of Diabetes, Endocrinology and Metabolism, Department of Metabolism, Digestion and Reproduction, Imperial College London, London, UK

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Sarah Rouse Department of Life Sciences, Imperial College, London, UK

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Alejandra Tomas Section of Cell Biology and Functional Genomics, Division of Diabetes, Endocrinology and Metabolism, Department of Metabolism, Digestion and Reproduction, Imperial College London, London, UK

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Introduction Lipids are structurally diverse organic compounds crucial for the normal functioning of all cells, as they are key constituents of membranes essential for cellular structure that enable the compartmentalisation of tightly

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-23-0335
Volume/Issue:
Volume 261: Issue 3
Open access
Jung-Yoon Heo Department of Physiology, College of Medicine, Yeungnam University, Daegu, Korea
Smart-Aging Convergence Research Center, College of Medicine, Yeungnam University, Daegu, Korea

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Ji-Eun Kim Department of Physiology, College of Medicine, Yeungnam University, Daegu, Korea
Smart-Aging Convergence Research Center, College of Medicine, Yeungnam University, Daegu, Korea

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Yongwook Dan Weinberg College, Northwestern University, Evanston, Illinois, USA

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Yong-Woon Kim Department of Physiology, College of Medicine, Yeungnam University, Daegu, Korea

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Jong-Yeon Kim Department of Physiology, College of Medicine, Yeungnam University, Daegu, Korea

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Kyu Hyang Cho Department of Internal Medicine, College of Medicine, Yeungnam University, Daegu, Korea

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Young Kyung Bae Department of Pathology, College of Medicine, Yeungnam University, Daegu, Korea

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Seung-Soon Im Department of Physiology, Keimyung University School of Medicine, Daegu, Korea

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Kwang-Hyeon Liu College of Pharmacy and Research Institute of Pharmaceutical Sciences, Kyungpook National University, Daegu, Korea

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In-Hwan Song Department of Anatomy, College of Medicine, Yeungnam University, Daegu, Korea

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Jae-Ryong Kim Smart-Aging Convergence Research Center, College of Medicine, Yeungnam University, Daegu, Korea
Department of Biochemistry and Molecular Biology, College of Medicine, Yeungnam University, Daegu, Korea

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In-Kyu Lee Department of Internal Medicine, School of Medicine, Kyungpook National University, Daegu, Korea

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So-Young Park Department of Physiology, College of Medicine, Yeungnam University, Daegu, Korea
Smart-Aging Convergence Research Center, College of Medicine, Yeungnam University, Daegu, Korea

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independent risk factor for the development of chronic kidney disease ( Laville 2011 , Odagiri et al. 2014 ). One of the proposed mechanisms for obesity-induced chronic kidney disease is lipid accumulation induced by abnormal lipid metabolism, reduced

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-17-0453
Volume/Issue:
Volume 237: Issue 2
Free access
Caroline E Geisler School of Animal and Comparative Biomedical Sciences, University of Arizona, Tucson, Arizona, USA

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Benjamin J Renquist School of Animal and Comparative Biomedical Sciences, University of Arizona, Tucson, Arizona, USA

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have similar histology and disease progression, AFLD also induces molecular and clinical changes that are attributed to high alcohol consumption, and not as a result of hepatic lipid accumulation ( Toshikuni et al. 2014 , Rasineni et al. 2016

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-16-0513
Volume/Issue:
Volume 234: Issue 1
Free access
Giselle Adriana Abruzzese Laboratorio de Fisio-patología Ovárica, Centro de Estudios Farmacológicos y Botánicos (CEFYBO), Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Facultad de Medicina, Universidad de Buenos Aires (UBA), Buenos Aires, Argentina

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Maria Florencia Heber Laboratorio de Fisio-patología Ovárica, Centro de Estudios Farmacológicos y Botánicos (CEFYBO), Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Facultad de Medicina, Universidad de Buenos Aires (UBA), Buenos Aires, Argentina

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Silvana Rocio Ferreira Laboratorio de Fisio-patología Ovárica, Centro de Estudios Farmacológicos y Botánicos (CEFYBO), Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Facultad de Medicina, Universidad de Buenos Aires (UBA), Buenos Aires, Argentina

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Leandro Martin Velez Laboratorio de Fisio-patología Ovárica, Centro de Estudios Farmacológicos y Botánicos (CEFYBO), Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Facultad de Medicina, Universidad de Buenos Aires (UBA), Buenos Aires, Argentina

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Roxana Reynoso Laboratorio de Endocrinología, Departamento de Fisiología, Facultad de Medicina, Universidad de Buenos Aires (UBA), Buenos Aires, Argentina

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Omar Pedro Pignataro Laboratorio de Endocrinología Molecular y Transducción de Señales, Instituto de Biología y Medicina Experimental, Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Universidad de Buenos Aires (UBA), Buenos Aires, Argentina

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Alicia Beatriz Motta Laboratorio de Fisio-patología Ovárica, Centro de Estudios Farmacológicos y Botánicos (CEFYBO), Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Facultad de Medicina, Universidad de Buenos Aires (UBA), Buenos Aires, Argentina

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tissues is unknown. The liver is involved in lipid synthesis, transportation and storage, as well as in glucose and insulin metabolism ( Paschos & Paletas 2009 ), all key factors in PCOS pathogenesis ( den Boer et al . 2004 , Paschos & Paletas 2009

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-15-0471
Volume/Issue:
Volume 230: Issue 1
Free access
Aline Cordeiro Biophysics Institute Carlos Chagas Filho, Federal University of Rio de Janeiro, Centro de Ciências da Saúde, Avenida Carlos Chagas Filho, 373, Bloco G, Cidade Universitária - Ilha do Fundão, Rio de Janeiro - RJ 21941-902, Brazil

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Luana Lopes Souza Biophysics Institute Carlos Chagas Filho, Federal University of Rio de Janeiro, Centro de Ciências da Saúde, Avenida Carlos Chagas Filho, 373, Bloco G, Cidade Universitária - Ilha do Fundão, Rio de Janeiro - RJ 21941-902, Brazil

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Marcelo Einicker-Lamas Biophysics Institute Carlos Chagas Filho, Federal University of Rio de Janeiro, Centro de Ciências da Saúde, Avenida Carlos Chagas Filho, 373, Bloco G, Cidade Universitária - Ilha do Fundão, Rio de Janeiro - RJ 21941-902, Brazil

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Carmen Cabanelas Pazos-Moura Biophysics Institute Carlos Chagas Filho, Federal University of Rio de Janeiro, Centro de Ciências da Saúde, Avenida Carlos Chagas Filho, 373, Bloco G, Cidade Universitária - Ilha do Fundão, Rio de Janeiro - RJ 21941-902, Brazil

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Thyroid hormone effects on lipid metabolism The thyroid hormones, triiodothyronine (T 3 ) and thyroxine (T 4 ), modulate several physiological processes in organisms and are critical in the growth, development, differentiation and maintenance of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0542
Volume/Issue:
Volume 216: Issue 3
Free access
Elias Rawish Institute of Experimental and Clinical Pharmacology and Toxicology, University of Lübeck, Lübeck, Germany
DZHK (German Centre for Cardiovascular Research), partner site Hamburg/Kiel/Lübeck, Lübeck, Germany
CBBM (Center of Brain, Behavior and Metabolism), Lübeck, Germany

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Laura Nickel Institute of Experimental and Clinical Pharmacology and Toxicology, University of Lübeck, Lübeck, Germany
DZHK (German Centre for Cardiovascular Research), partner site Hamburg/Kiel/Lübeck, Lübeck, Germany
CBBM (Center of Brain, Behavior and Metabolism), Lübeck, Germany

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Franziska Schuster Institute of Experimental and Clinical Pharmacology and Toxicology, University of Lübeck, Lübeck, Germany
CBBM (Center of Brain, Behavior and Metabolism), Lübeck, Germany

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Ines Stölting Institute of Experimental and Clinical Pharmacology and Toxicology, University of Lübeck, Lübeck, Germany

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Alex Frydrychowicz Department of Radiology and Nuclear Medicine, University-Hospital Schleswig Holstein and University of Lübeck, Lübeck, Germany

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Kathrin Saar Max Delbruck Center for Molecular Medicine in the Helmholtz Association, Berlin-Buch, Germany
DZHK (German Centre for Cardiovascular Research), partner site Berlin, Berlin, Germany

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Norbert Hübner Max Delbruck Center for Molecular Medicine in the Helmholtz Association, Berlin-Buch, Germany
DZHK (German Centre for Cardiovascular Research), partner site Berlin, Berlin, Germany

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Alaa Othman CBBM (Center of Brain, Behavior and Metabolism), Lübeck, Germany
Institute for Clinical Chemistry, University Hospital Zürich, Zurich, Switzerland

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Lars Kuerschner Life and Medical Sciences Institute (LIMES), University of Bonn, Bonn, Germany

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Walter Raasch Institute of Experimental and Clinical Pharmacology and Toxicology, University of Lübeck, Lübeck, Germany
DZHK (German Centre for Cardiovascular Research), partner site Hamburg/Kiel/Lübeck, Lübeck, Germany
CBBM (Center of Brain, Behavior and Metabolism), Lübeck, Germany

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development of both peripheral and central leptin resistance and obesity by impairing leptin penetration through the BBB and leptin signaling in the brain ( Banks et al. 2018 ). In addition to peripheral alterations in TG concentrations, cerebral lipid

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0319
Volume/Issue:
Volume 244: Issue 1
Restricted access