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Cheng-Hsuan Tsai
Cheng-Hsuan Tsai
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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Zheng-Wei Chen
Zheng-Wei Chen
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital Yun-Lin Branch, Yun-Lin, Taiwan
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Bo-Ching Lee
Bo-Ching Lee
Department of Medical Imaging, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Che-Wei Liao
Che-Wei Liao
Department of Medicine, National Taiwan University Cancer Center, Taipei, Taiwan
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Yi-Yao Chang
Yi-Yao Chang
Cardiology Division of Cardiovascular Medical Center, Far Eastern Memorial Hospital, New Taipei City, Taiwan
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Yan-Rou Tsai
Yan-Rou Tsai
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Chia-Hung Chou
Chia-Hung Chou
Department of Obstetrics and Gynecology, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Vin-Cent Wu
Vin-Cent Wu
National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
Division of Nephrology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Division of Nephrology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
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Chi-Sheng Hung
Chi-Sheng Hung
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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Yen-Hung Lin
Yen-Hung Lin
Division of Cardiology, Department of Internal Medicine, National Taiwan University Hospital and National Taiwan University College of Medicine, Taipei, Taiwan
National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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National Taiwan University Hospital Primary Aldosteronism Center, Taipei, Taiwan
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interest to clinicians and researchers for decades. Aldosterone has increasingly attracted attention due to its influence on mitochondria, the dynamic organelles responsible for cellular energy production and numerous physiological processes. Mitochondria
Laura Marroqui
Laura Marroqui
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM) and Institute of Bioengineering, Miguel Hernández University of Elche, Alicante, Spain
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Eva Tudurí
Eva Tudurí
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM) and Institute of Bioengineering, Miguel Hernández University of Elche, Alicante, Spain
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Paloma Alonso-Magdalena
Paloma Alonso-Magdalena
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM) and Institute of Bioengineering, Miguel Hernández University of Elche, Alicante, Spain
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Iván Quesada
Iván Quesada
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM) and Institute of Bioengineering, Miguel Hernández University of Elche, Alicante, Spain
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Ángel Nadal
Ángel Nadal
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM) and Institute of Bioengineering, Miguel Hernández University of Elche, Alicante, Spain
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Reinaldo Sousa dos Santos
Reinaldo Sousa dos Santos
Centro de Investigación Biomédica en Red de Diabetes y Enfermedades Metabólicas Asociadas (CIBERDEM) and Institute of Bioengineering, Miguel Hernández University of Elche, Alicante, Spain
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metabolism by affecting multiple cellular events in different organelles, including endoplasmic reticulum (ER) and mitochondria ( Meyer et al. 2013 , Nadal et al. 2017 , Rajamani et al. 2017 ). Mitochondria are essential for the normal cellular
Sergio Di Meo
Sergio Di Meo
Department of Biology, University of Naples ‘Federico II’, Naples, Italy
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Susanna Iossa
Susanna Iossa
Department of Biology, University of Naples ‘Federico II’, Naples, Italy
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Paola Venditti
Paola Venditti
Department of Biology, University of Naples ‘Federico II’, Naples, Italy
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dysregulation, including IR. The theory also suggests that mitochondria are key players in the IR development, but there are different views about the mechanisms by which mitochondria contribute to IR pathogenesis. It has been proposed that a decrease in
Yuehui Zhang
Yuehui Zhang
Department of Obstetrics and Gynecology, Key Laboratory and Unit of Infertility in Chinese Medicine, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Harbin, China
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Min Hu
Min Hu
Department of Physiology/Endocrinology, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden
Department of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China
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Department of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China
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Wenyan Jia
Wenyan Jia
Department of Obstetrics and Gynecology, Key Laboratory and Unit of Infertility in Chinese Medicine, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Harbin, China
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Guoqi Liu
Guoqi Liu
Department of Obstetrics and Gynecology, Key Laboratory and Unit of Infertility in Chinese Medicine, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Harbin, China
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Jiao Zhang
Jiao Zhang
Department of Acupuncture and Moxibustion, Second Affiliated Hospital, Heilongjiang University of Chinese Medicine, Harbin, China
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Bing Wang
Bing Wang
Department of Obstetrics and Gynecology, Key Laboratory and Unit of Infertility in Chinese Medicine, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Harbin, China
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Juan Li
Juan Li
Department of Physiology/Endocrinology, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden
Department of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China
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Department of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou, China
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Peng Cui
Peng Cui
Department of Physiology/Endocrinology, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden
Department of Gynecology, Shuguang Hospital affiliated to Shanghai University of Traditional Chinese Medicine, Shanghai, China
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Department of Gynecology, Shuguang Hospital affiliated to Shanghai University of Traditional Chinese Medicine, Shanghai, China
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Xin Li
Xin Li
Department of Physiology/Endocrinology, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden
Department of Gynecology, Obstetrics and Gynecology Hospital of Fudan University, Shanghai, China
Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Shanghai, China
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Department of Gynecology, Obstetrics and Gynecology Hospital of Fudan University, Shanghai, China
Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Shanghai, China
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Susanne Lager
Susanne Lager
Department of Women’s and Children’s Health, Uppsala University, Uppsala, Sweden
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Amanda Nancy Sferruzzi-Perri
Amanda Nancy Sferruzzi-Perri
Centre for Trophoblast Research, Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK
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Yanhua Han
Yanhua Han
Department of Obstetrics and Gynecology, Key Laboratory and Unit of Infertility in Chinese Medicine, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Harbin, China
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Songjiang Liu
Songjiang Liu
Department of Obstetrics and Gynecology, Key Laboratory and Unit of Infertility in Chinese Medicine, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Harbin, China
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Xiaoke Wu
Xiaoke Wu
Department of Obstetrics and Gynecology, Key Laboratory and Unit of Infertility in Chinese Medicine, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Harbin, China
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Mats Brännström
Mats Brännström
Department of Obstetrics and Gynecology, Sahlgrenska University Hospital, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden
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Linus R Shao
Linus R Shao
Department of Physiology/Endocrinology, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden
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Håkan Billig
Håkan Billig
Department of Physiology/Endocrinology, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden
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activation of ferroptosis in the gravid uterus and placenta contribute to HAIR-induced fetal loss in both animal models and humans. Mitochondria play a protective role in the regulation of glutathione-induced ferroptosis ( Gao et al. 2019 ). In women with
Kechun Tang
Kechun Tang
Department of Medicine, University of California, San Diego, La Jolla, California, USA
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Teresa Pasqua
Teresa Pasqua
Department of Medicine, University of California, San Diego, La Jolla, California, USA
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Angshuman Biswas
Angshuman Biswas
Department of Medicine, University of California, San Diego, La Jolla, California, USA
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Sumana Mahata
Sumana Mahata
Division of Biology & Biological Engineering, California Institute of Technology, Pasadena, California, USA
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Jennifer Tang
Jennifer Tang
Department of Medicine, University of California, San Diego, La Jolla, California, USA
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Alisa Tang
Alisa Tang
Department of Medicine, University of California, San Diego, La Jolla, California, USA
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Gautam K Bandyopadhyay
Gautam K Bandyopadhyay
Department of Medicine, University of California, San Diego, La Jolla, California, USA
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Amiya P Sinha-Hikim
Amiya P Sinha-Hikim
Charles Drew University of Medicine and Science, Los Angeles, California, USA
David Geffen School of Medicine, University of California-Los Angeles, Los Angeles, California, USA
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David Geffen School of Medicine, University of California-Los Angeles, Los Angeles, California, USA
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Nai-Wen Chi
Nai-Wen Chi
Department of Medicine, University of California, San Diego, La Jolla, California, USA
VA San Diego Healthcare System, San Diego, California, USA
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VA San Diego Healthcare System, San Diego, California, USA
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Nicholas J G Webster
Nicholas J G Webster
Department of Medicine, University of California, San Diego, La Jolla, California, USA
VA San Diego Healthcare System, San Diego, California, USA
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VA San Diego Healthcare System, San Diego, California, USA
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Angelo Corti
Angelo Corti
IRCCS San Raffaele Scientific Institute, San Raffaele Vita-Salute University, Milan, Italy
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Sushil K Mahata
Sushil K Mahata
Department of Medicine, University of California, San Diego, La Jolla, California, USA
VA San Diego Healthcare System, San Diego, California, USA
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VA San Diego Healthcare System, San Diego, California, USA
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cristae membrane surface area. To normalize the measurement, this area was divided by the outer membrane area per mitochondrion. The sum of the area of the mitochondria was divided by the area of the cytoplasm and multiplied by 100 to determine the
Emily J King
Emily J King
Baker Heart & Diabetes Institute, Melbourne, Victoria, Australia
Central Clinical School, Monash University, Melbourne, Victoria, Australia
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Central Clinical School, Monash University, Melbourne, Victoria, Australia
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Simon T Bond
Simon T Bond
Baker Heart & Diabetes Institute, Melbourne, Victoria, Australia
Central Clinical School, Monash University, Melbourne, Victoria, Australia
Baker Department of Cardiometabolic Health, The University of Melbourne, Melbourne, Australia
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Central Clinical School, Monash University, Melbourne, Victoria, Australia
Baker Department of Cardiometabolic Health, The University of Melbourne, Melbourne, Australia
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Christine Yang
Christine Yang
Baker Heart & Diabetes Institute, Melbourne, Victoria, Australia
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Yingying Liu
Yingying Liu
Baker Heart & Diabetes Institute, Melbourne, Victoria, Australia
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Anna C Calkin
Anna C Calkin
Baker Heart & Diabetes Institute, Melbourne, Victoria, Australia
Central Clinical School, Monash University, Melbourne, Victoria, Australia
Baker Department of Cardiometabolic Health, The University of Melbourne, Melbourne, Australia
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Central Clinical School, Monash University, Melbourne, Victoria, Australia
Baker Department of Cardiometabolic Health, The University of Melbourne, Melbourne, Australia
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Darren C Henstridge
Darren C Henstridge
Baker Heart & Diabetes Institute, Melbourne, Victoria, Australia
School of Health Sciences, University of Tasmania, Launceston, Tasmania, Australia
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School of Health Sciences, University of Tasmania, Launceston, Tasmania, Australia
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Brian G Drew
Brian G Drew
Baker Heart & Diabetes Institute, Melbourne, Victoria, Australia
Central Clinical School, Monash University, Melbourne, Victoria, Australia
Baker Department of Cardiometabolic Health, The University of Melbourne, Melbourne, Australia
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Central Clinical School, Monash University, Melbourne, Victoria, Australia
Baker Department of Cardiometabolic Health, The University of Melbourne, Melbourne, Australia
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their importance to cellular health, there are many intrinsic pathways that protect and repair mitochondria, so as to maintain high functionality. One such process is that of mitochondrial quality control, which includes processes such as mitochondrial
Sandra Zárate
Sandra Zárate
Instituto de Investigaciones Biomédicas (INBIOMED, UBA-CONICET), Buenos Aires, Argentina
Departamento de Histología, Embriología, Biología Celular y Genética, Facultad de Medicina, Universidad de Buenos Aires, Buenos Aires, Argentina
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Departamento de Histología, Embriología, Biología Celular y Genética, Facultad de Medicina, Universidad de Buenos Aires, Buenos Aires, Argentina
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Mariana Astiz
Mariana Astiz
Instituto de Investigaciones Bioquímicas de La Plata (INIBIOLP, UNLP-CONICET) and Cátedra de Bioquímica y Biología Molecular, Facultad de Medicina, Universidad Nacional de La Plata, La Plata, Argentina
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Natalia Magnani
Natalia Magnani
Instituto de Bioquímica y Medicina Molecular (IBIMOL, UBA-CONICET), Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina
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Mercedes Imsen
Mercedes Imsen
Instituto de Investigaciones Biomédicas (INBIOMED, UBA-CONICET), Buenos Aires, Argentina
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Florencia Merino
Florencia Merino
Instituto de Investigaciones Biomédicas (INBIOMED, UBA-CONICET), Buenos Aires, Argentina
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Silvia Álvarez
Silvia Álvarez
Instituto de Bioquímica y Medicina Molecular (IBIMOL, UBA-CONICET), Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina
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Analía Reinés
Analía Reinés
Instituto de Biología Celular y Neurociencias ‘Prof. E. De Robertis’ (IBCN, UBA-CONICET), Facultad de Medicina and Cátedra de Farmacología, Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires, Buenos Aires, Argentina
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Adriana Seilicovich
Adriana Seilicovich
Instituto de Investigaciones Biomédicas (INBIOMED, UBA-CONICET), Buenos Aires, Argentina
Departamento de Histología, Embriología, Biología Celular y Genética, Facultad de Medicina, Universidad de Buenos Aires, Buenos Aires, Argentina
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Departamento de Histología, Embriología, Biología Celular y Genética, Facultad de Medicina, Universidad de Buenos Aires, Buenos Aires, Argentina
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Introduction Mitochondria are key organelles for cellular bioenergetics and survival. They are the main source of ATP production through oxidative phosphorylation by the mitochondrial respiratory chain and also the primary sites for cellular
R Mastrocola
R Mastrocola
Department of Experimental Medicine and Oncology, Section of General Pathology, Corso Raffaello 30, University of Turin, 10125 Turin, Italy
Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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F Restivo
F Restivo
Department of Experimental Medicine and Oncology, Section of General Pathology, Corso Raffaello 30, University of Turin, 10125 Turin, Italy
Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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I Vercellinatto
I Vercellinatto
Department of Experimental Medicine and Oncology, Section of General Pathology, Corso Raffaello 30, University of Turin, 10125 Turin, Italy
Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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O Danni
O Danni
Department of Experimental Medicine and Oncology, Section of General Pathology, Corso Raffaello 30, University of Turin, 10125 Turin, Italy
Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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E Brignardello
E Brignardello
Department of Experimental Medicine and Oncology, Section of General Pathology, Corso Raffaello 30, University of Turin, 10125 Turin, Italy
Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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M Aragno
M Aragno
Department of Experimental Medicine and Oncology, Section of General Pathology, Corso Raffaello 30, University of Turin, 10125 Turin, Italy
Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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G Boccuzzi
G Boccuzzi
Department of Experimental Medicine and Oncology, Section of General Pathology, Corso Raffaello 30, University of Turin, 10125 Turin, Italy
Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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Department of Clinical Pathophysiology, Via Genova 3, University of Turin, 10126 Turin, Italy
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role in brain damage ( Aragno et al. 2000 a , b , 2002 , Arvanitakis et al. 2004 ). Emerging evidence shows that the increased oxidative stress and consequent oxidative damage observed in hyperglycemic conditions begins in the mitochondria, which
Zhe-Zhen Liao
Zhe-Zhen Liao
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Xiao-Yan Qi
Xiao-Yan Qi
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Ya-Di Wang
Ya-Di Wang
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Jiao-Yang Li
Jiao-Yang Li
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Qian-Qian Gu
Qian-Qian Gu
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Can Hu
Can Hu
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Yin Hu
Yin Hu
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Heng Sun
Heng Sun
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Li Ran
Li Ran
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Jing Yang
Jing Yang
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Jiang-Hua Liu
Jiang-Hua Liu
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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Xin-Hua Xiao
Xin-Hua Xiao
Department of Metabolism and Endocrinology, The First Affiliated Hospital of University of South China, Hengyang, Hunan Province, China
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betatrophin in WAT beiging by employing a loss-of-function approach in mice. Here, we discovered that inhibition of betatrophin attenuated white fat mass and improve TG levels through stimulating beiging process and mitochondria biogenesis. The above process
Giuseppe Calamita
Giuseppe Calamita
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Maria Moreno
Maria Moreno
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Domenico Ferri
Domenico Ferri
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Elena Silvestri
Elena Silvestri
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Patrizia Roberti
Patrizia Roberti
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Luigi Schiavo
Luigi Schiavo
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Patrizia Gena
Patrizia Gena
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Maria Svelto
Maria Svelto
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Fernando Goglia
Fernando Goglia
Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Introduction Triiodothyronine (T3) exerts significant actions on energy metabolism, with mitochondria being a major target for its effects ( Soboll 1993 ). Extensive changes occur in the mitochondrial compartment in response either
