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Satoko Haeno Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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Naoyuki Maeda Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine
Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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Takeshi Yagi Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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Sachi Tahata Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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Michiko Sato Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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Kanako Sakaguchi Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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Taku Miyasho Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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Hiromi Ueda Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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Hiroshi Yokota Laboratory of Veterinary Biochemistry, Laboratory of Veterinary Pathology, Laboratory of Veterinary Anatomy, Laboratory Animal Nutrition, Japan Meat Science and Technology Institute, School of Veterinary Medicine

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the adrenal P450s, which are involved in corticosterone biosynthesis, have been reported ( Diamanti-Kandarakis et al . 2009 ). Zimmerman et al . (1991) is the only research paper to report an increase in adrenal weight and decrease in steroid

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0460
Volume/Issue:
Volume 221: Issue 2
Free access
Hong-Tao Zheng College of Veterinary Medicine, South China Agricultural University, Guangzhou, China

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Tao Fu College of Veterinary Medicine, South China Agricultural University, Guangzhou, China

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Hai-Yi Zhang College of Veterinary Medicine, South China Agricultural University, Guangzhou, China

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Zhen-Shan Yang College of Veterinary Medicine, South China Agricultural University, Guangzhou, China

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Zhan-Hong Zheng College of Veterinary Medicine, South China Agricultural University, Guangzhou, China

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Zeng-Ming Yang College of Veterinary Medicine, South China Agricultural University, Guangzhou, China

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1/Hsd11b2 are main enzymes for regulating local level of GCs (cortisol in humans; corticosterone in mice). Hsd11b1 catalyzes the conversion of inactive cortisone and 11-dehydrocorticosterone into active cortisol and corticosterone, respectively. In

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0349
Volume/Issue:
Volume 244: Issue 1
Restricted access
Adrián Báez-Ruiz Department of Physiology and Cellular Biology, Biomedical Research Institute, UNAM, DF, México
Department of Cellular Physiology, Faculty of Science, UASLP, San Luis Potosí, México

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Natalí N Guerrero-Vargas Department of Physiology and Cellular Biology, Biomedical Research Institute, UNAM, DF, México
Department of Anatomy, Faculty of Medicine, UNAM, DF, México

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Fernando Cázarez-Márquez Department of Physiology and Cellular Biology, Biomedical Research Institute, UNAM, DF, México

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Elizabeth Sabath Department of Physiology and Cellular Biology, Biomedical Research Institute, UNAM, DF, México

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María del Carmen Basualdo Department of Physiology and Cellular Biology, Biomedical Research Institute, UNAM, DF, México

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Roberto Salgado-Delgado Department of Cellular Physiology, Faculty of Science, UASLP, San Luis Potosí, México

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Carolina Escobar Department of Anatomy, Faculty of Medicine, UNAM, DF, México

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Ruud M Buijs Department of Physiology and Cellular Biology, Biomedical Research Institute, UNAM, DF, México

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, there are numerous data highlighting corticosterone and melatonin as important circadian regulated hormones maintaining metabolic homeostasis and energy balance ( Bedrosian et al. 2016 ). The daily peak of glucocorticoids is associated with the onset

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-17-0370
Volume/Issue:
Volume 235: Issue 3
Free access
Ken Takao Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Katsumi Iizuka Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Yanyan Liu Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Teruaki Sakurai Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Sodai Kubota Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan
Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan

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Saki Kubota-Okamoto Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan
Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan

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Toshinori Imaizumi Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Yoshihiro Takahashi Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Yermek Rakhat Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan
Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan

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Satoko Komori Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Tokuyuki Hirose Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Kenta Nonomura Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Takehiro Kato Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Masami Mizuno Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Tetsuya Suwa Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Yukio Horikawa Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Masakatsu Sone Division of Metabolism and Endocrinology, Department of Internal Medicine, St. Marianna University School of Medicine, Kawasaki, Japan

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Daisuke Yabe Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan
Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan
Division of Molecular and Metabolic Medicine, Department of Physiology and Cell Biology, Kobe University Graduate School of Medicine, Kobe, Japan

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through synthesis and secretion of steroid hormones such as corticosterone. Patients with adrenal cortex insufficiency show plasma hyponatremia, hyperkalemia, and hypoglycemia, indicating that steroid hormones are essential for survival ( Val & Martinez

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-20-0442
Volume/Issue:
Volume 248: Issue 3
Restricted access
Cherish Chong Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital, Boston, Massachusetts, USA

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Anis Hamid Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital, Boston, Massachusetts, USA

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Tham Yao Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital, Boston, Massachusetts, USA

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Amanda E Garza Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital, Boston, Massachusetts, USA

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Luminita H Pojoga Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital, Boston, Massachusetts, USA

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Gail K Adler Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital, Boston, Massachusetts, USA

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Jose R Romero Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital, Boston, Massachusetts, USA

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Gordon H Williams Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital, Boston, Massachusetts, USA

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Aldosterone Radioimmunoassay (RIA) kit (IBL International, Hamburg, Germany). Corticosterone was quantified with ImmuChem Corticosterone Double Antibody RIA kit (MP Biomedicals, Orangeburg, NY, USA). Statistical analysis Data are expressed as mean ±  s

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0452
Volume/Issue:
Volume 232: Issue 3
Free access
Marian Joëls Department of Translational Neuroscience, Brain Center Rudolf Magnus, University Medical Center Utrecht, University of Utrecht, Utrecht, The Netherlands
University of Groningen, University Medical Center Groningen, Groningen, The Netherlands

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of adrenocorticotropin hormone from the anterior pituitary into the circulation ( Herman et al. 2016 ). In the adrenal cortex, this causes the synthesis and release of cortisol (the predominant corticosteroid hormone in humans) or corticosterone (in

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-18-0226
Volume/Issue:
Volume 238: Issue 3
Free access
Lesley A Hill Department of Cellular and Physiological Sciences, University of British Columbia, Vancouver
Department of Obstetrics and Gynaecology, University of British Columbia, Vancouver, British Columbia, Canada

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Tamara S Bodnar Department of Cellular and Physiological Sciences, University of British Columbia, Vancouver

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Joanne Weinberg Department of Cellular and Physiological Sciences, University of British Columbia, Vancouver

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Geoffrey L Hammond Department of Cellular and Physiological Sciences, University of British Columbia, Vancouver
Department of Obstetrics and Gynaecology, University of British Columbia, Vancouver, British Columbia, Canada

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. Plasma CBG analysis Plasma corticosterone-binding capacity of CBG was measured using an established ligand-saturation assay ( Hammond & Lahteenmaki 1983 ). Briefly, samples were diluted (1:1000–1:3000) in phosphate-buffered saline and stripped of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0047
Volume/Issue:
Volume 230: Issue 2
Free access
B M McGowan Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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J S Minnion Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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K G Murphy Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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D Roy Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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S A Stanley Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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W S Dhillo Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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J V Gardiner Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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M A Ghatei Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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S R Bloom Department of Diabetes and Endocrinology, Section of Investigative Medicine, Molecular Genetics, Guy's and St Thomas' NHS Foundation Trust, Westminster Bridge Road, London SE1 7EH, London

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anterior pituitary, an effect that is potentiated by AVP ( Volpi et al . 2004 ). ACTH in turn stimulates corticosterone release from the adrenal cortex. A further pituitary hormone, prolactin, so-called because of its well-characterised role in lactation

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0603
Volume/Issue:
Volume 221: Issue 2
Free access
Lisa L Koorneef Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, The Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands

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Jan Kroon Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, The Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands

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Eva M G Viho Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, The Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands

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Lucas F Wahl Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, The Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands

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Kim M L Heckmans Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, The Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands

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Marloes M A R van Dorst Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, The Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands

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Menno Hoekstra Division of BioTherapeutics, Leiden Academic Centre for Drug Research, Leiden University, Leiden, The Netherlands

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René Houtman Pamgene International, Den Bosch, The Netherlands

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Hazel Hunt Corcept Therapeutics, Menlo Park, California, USA

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Onno C Meijer Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, The Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands

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and ad libitum access to food and water. In the acute corticosterone exposure experiments, mice received a synthetic chow diet (Research Diets, New Brunswick, NJ, USA) containing vehicle or CORT125281 (6, 20 or 60 mg/kg/day, Corcept Therapeutics

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0486
Volume/Issue:
Volume 246: Issue 1
Open access
Zachary Silver Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada

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Sam Abbott-Tate Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada

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Lindsay Hyland Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada

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Frances Sherratt Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada

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Barbara Woodside Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada

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Alfonso Abizaid Department of Neuroscience, Carleton University, Ottawa, Ontario, Canada

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exposure to corticosterone (CORT) results in increased food intake, weight gain, and adiposity ( Karatsoreos et al. 2010 , Sefton et al. 2016 ), as well as atrophy of the thymus and adrenal glands, hyperleptinemia, impaired glucose tolerance and

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-20-0579
Volume/Issue:
Volume 250: Issue 2
Restricted access