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Jay W Porter Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA

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Joe L Rowles III Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA
Division of Nutritional Sciences, University of Illinois at Urbana-Champaign, Urbana, Illinois, USA

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Justin A Fletcher Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA
Research Service-Harry S Truman Memorial VA Hospital, Columbia, Missouri, USA
University of Texas Southwestern Medical Center, Dallas, Texas, USA

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Terese M Zidon Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA

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Nathan C Winn Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA

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Leighton T McCabe Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA

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Young-Min Park Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA
University of Colorado Denver – Anschutz Medical Campus, Denver, Colorado, USA

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James W Perfield II Lilly Research Laboratories, Indianapolis, Indiana, USA

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John P Thyfault Department of Molecular and Integrative Physiology, University of Kansas Medical Center, Kansas City, Kansas, USA
Kansas City VA Medical Center, Kansas City, Missouri, USA

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R Scott Rector Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA
Research Service-Harry S Truman Memorial VA Hospital, Columbia, Missouri, USA

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Jaume Padilla Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA
Department of Child Health, University of Missouri, Columbia, Missouri, USA
Dalton Cardiovascular Research Center, University of Missouri, Columbia, Missouri, USA

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Victoria J Vieira-Potter Department of Nutrition and Exercise Physiology, University of Missouri, Columbia, Missouri, USA

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endocrine organ that is profoundly affected by exercise. Exercise lessens WAT inflammation and improves systemic metabolic health, even in the settings of obesity ( Vieira et al . 2009 a , Welly et al . 2016 ), although the mechanisms are not fully

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-17-0190
Volume/Issue:
Volume 235: Issue 2
Free access
Hiroharu Mifune Institute of Animal Experimentation, Kurume University School of Medicine, Kurume, Japan

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Yuji Tajiri Division of Endocrinology and Metabolism, Kurume University School of Medicine, Kurume, Japan

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Yusuke Sakai Institute of Animal Experimentation, Kurume University School of Medicine, Kurume, Japan

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Yukie Kawahara Department of Pharmacology, Kurume University School of Medicine, Kurume, Japan

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Kento Hara Division of Endocrinology and Metabolism, Kurume University School of Medicine, Kurume, Japan

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Takahiro Sato Molecular Genetics, Life Science Institute, Kurume University, Kurume, Japan

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Yoshihiro Nishi Department of Physiology, Kurume University School of Medicine, Kurume, Japan

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Akinori Nishi Department of Pharmacology, Kurume University School of Medicine, Kurume, Japan

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Ryouichi Mitsuzono Department of Exercise Physiology, Institute of Health and Sports Science, Kurume University, Kurume, Japan

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Tatsuyuki Kakuma Bostatistics Center, Kurume University, Kurume, Japan

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Masayasu Kojima Molecular Genetics, Life Science Institute, Kurume University, Kurume, Japan

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Introduction It is well known that exercise in itself has several benefits in terms of health and fitness ( Patterson & Levin 2008 , Haskell-Luevano et al. 2009 ), as well as neural and cognitive effects ( Cotman & Engesser-Cesar 2002 ), in

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0213
Volume/Issue:
Volume 244: Issue 1
Open access
Daiana Araujo Santana-Oliveira Laboratory of Morphometry, Metabolism and Cardiovascular Diseases, Biomedical Center, Institute of Biology, Rio de Janeiro State University, Rio de Janeiro, Brazil

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Henrique Souza-Tavares Laboratory of Morphometry, Metabolism and Cardiovascular Diseases, Biomedical Center, Institute of Biology, Rio de Janeiro State University, Rio de Janeiro, Brazil

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Aline Fernandes-da-Silva Laboratory of Morphometry, Metabolism and Cardiovascular Diseases, Biomedical Center, Institute of Biology, Rio de Janeiro State University, Rio de Janeiro, Brazil

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Flavia Maria Silva-Veiga Laboratory of Morphometry, Metabolism and Cardiovascular Diseases, Biomedical Center, Institute of Biology, Rio de Janeiro State University, Rio de Janeiro, Brazil

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Gustavo Casimiro-Lopes Department of Gymnastics, Physical Education and Sports Institute, Laboratory of Exercise Pathophysiology (LAFE), Rio de Janeiro State University, Rio de Janeiro, Brazil

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Patricia Cristina Lisboa Laboratory of Endocrine Physiology, Biology Institute, Rio de Janeiro State University, Rio de Janeiro, Rio de Janeiro, Brazil

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Carlos Alberto Mandarim-de-Lacerda Laboratory of Morphometry, Metabolism and Cardiovascular Diseases, Biomedical Center, Institute of Biology, Rio de Janeiro State University, Rio de Janeiro, Brazil

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Vanessa Souza-Mello Laboratory of Morphometry, Metabolism and Cardiovascular Diseases, Biomedical Center, Institute of Biology, Rio de Janeiro State University, Rio de Janeiro, Brazil

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. 2017 ). Therefore, lifestyle modifications that prevent dysbiosis and suppress diet-induced changes in the adipose tissue are relevant to obesity and metabolic alterations prevention. Exercise training is a successful and safe approach to preventing

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-23-0123
Volume/Issue:
Volume 259: Issue 1
Restricted access
Daniele Leão Ignacio School of Physical Education and Sports, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil
Faculdades Integradas IESGO, Formosa, Goiás, Brazil

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Rodrigo Soares Fortunato Institute of Biophysics Carlos Chagas Filho, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil

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Diego Silvestre School of Physical Education and Sports, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil

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Leonardo Matta Institute of Biophysics Carlos Chagas Filho, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil

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Andressa Lima de Vansconcelos Institute of Biophysics Carlos Chagas Filho, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil

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Denise Pires Carvalho Institute of Biophysics Carlos Chagas Filho, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil

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Antonio Galina Institute of Medical Biochemistry, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil

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João Pedro Werneck-de-Castro School of Physical Education and Sports, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil
Institute of Biophysics Carlos Chagas Filho, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil
Division of Endocrinology, Diabetes and Metabolism, University of Miami, Miller School of Medicine, Miami, Florida, USA

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João Paulo Cavalcanti-de-Albuquerque Institute of Biophysics Carlos Chagas Filho, Federal University of Rio de Janeiro, Rio de Janeiro, Brazil

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and could explain the fat gain observed in this model. Physical exercise is a non-pharmacological approach to treat metabolic diseases ( Colberg et al. 2010 ). Exercise-induced adaptations slow down age-related muscle-function decline, including

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-22-0057
Volume/Issue:
Volume 254: Issue 2
Restricted access
Amanda J Genders Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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Jujiao Kuang Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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Evelyn C Marin Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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Nicholas J Saner Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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Javier Botella Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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Macsue Jacques Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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Glenn K McConell Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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Victor A Andrade-Souza Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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Javier Chagolla Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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David J Bishop Institute for Health and Sport (iHeS), Victoria University, Melbourne, Victoria, Australia

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interventions that cause whole-body insulin resistance (e.g. HFD) also decrease mitochondrial respiration, using techniques that assess mitochondrial respiratory function in situ . Several studies have shown that exercise can reverse various aspects of the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-21-0242
Volume/Issue:
Volume 252: Issue 2
Restricted access
Brenton T Laing Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA

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Khoa Do Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA

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Tomoko Matsubara Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA

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David W Wert East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA

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Michael J Avery Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA

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Erin M Langdon Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA

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Donghai Zheng Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, Collage of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA

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Hu Huang Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, Collage of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA
Department of Physiology, East Carolina University, Greenville, North Carolina, USA

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preferentially affects POMC neurons in the ARC. This suggests that POMC neurons are the target and part of the mechanism of HFD-induced obesity and diabetes ( Li et al. 2012 ). Exercise therapy is a proven and effective clinical intervention for treating

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-15-0510
Volume/Issue:
Volume 229: Issue 2
Free access
Hyo Youl Moon
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Parkyong Song BioSignal Network Laboratory, Division of Molecular and Life Sciences, Lee Gil Ya Cancer and Diabetes Institute, School of Nano-Biotechnology and Chemical Engineering, Ulsan National Institute of Science and Technology, Engineering Building 104, 689-805 Ulsan, Republic of Korea

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Cheol Soo Choi BioSignal Network Laboratory, Division of Molecular and Life Sciences, Lee Gil Ya Cancer and Diabetes Institute, School of Nano-Biotechnology and Chemical Engineering, Ulsan National Institute of Science and Technology, Engineering Building 104, 689-805 Ulsan, Republic of Korea

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Sung Ho Ryu BioSignal Network Laboratory, Division of Molecular and Life Sciences, Lee Gil Ya Cancer and Diabetes Institute, School of Nano-Biotechnology and Chemical Engineering, Ulsan National Institute of Science and Technology, Engineering Building 104, 689-805 Ulsan, Republic of Korea

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Pann-Ghill Suh
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is regulated by lifestyle modifications that aim to increase physical activity, induce weight reduction, and decrease energy intake ( Farrell & Larter 2006 ). Exercise is accompanied by metabolic adaptations that promote the delivery to, and oxidation

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0135
Volume/Issue:
Volume 218: Issue 3
Open access
Chunxia Yu Department of Physiology and Pathophysiology, School of Basic Medical Science, Tianjin Medical University, Tianjin, China

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Sujuan Liu Department of Anatomy and Embryology, School of Basic Medical Science, Tianjin Medical University, Tianjin, China

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Liqin Chen Tianjin Key Laboratory of Environment, Nutrition and Public Health, School of Public Health, Tianjin Medical University, Tianjin, China

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Jun Shen Tianjin Key Laboratory of Environment, Nutrition and Public Health, School of Public Health, Tianjin Medical University, Tianjin, China

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Yanmei Niu Department of Rehabilitation, School of Medical Technology, Tianjin Medical University, Tianjin, China

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Tianyi Wang Department of Physiology and Pathophysiology, School of Basic Medical Science, Tianjin Medical University, Tianjin, China

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Wanqi Zhang Tianjin Key Laboratory of Environment, Nutrition and Public Health, School of Public Health, Tianjin Medical University, Tianjin, China

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Li Fu Department of Physiology and Pathophysiology, School of Basic Medical Science, Tianjin Medical University, Tianjin, China
Department of Rehabilitation, School of Medical Technology, Tianjin Medical University, Tianjin, China

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ecosystem development and its stability can be influenced by an existing dynamic balance between intrinsic and extrinsic factors that in turn can impact host health, such as diet and exercise ( Monda et al. 2017 ). Food is the substrate for the growth of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0122
Volume/Issue:
Volume 243: Issue 2
Restricted access
Taylor Landry East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA
Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA

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Daniel Shookster East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA
Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA

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Alec Chaves East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA
Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA

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Katrina Free East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA
Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA

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Tony Nguyen East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA
Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA

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Hu Huang East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina, USA
Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA
Department of Physiology, East Carolina University, Greenville, North Carolina, USA

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Introduction The effects of exercise on food intake have been studied extensively, yet the reasons for frequently conflicting results are still incompletely understood ( Durrant et al. 1982 , Staten 1991 , King et al. 1996 , Pomerleau

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-21-0250
Volume/Issue:
Volume 252: Issue 3
Restricted access
Hari K Somineni Departments of Pharmacology and Toxicology, Pathology, Veterans Affairs Medical Center, Boonshoft School of Medicine, Wright State University, Dayton, Ohio 45435, USA

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Gregory P Boivin Departments of Pharmacology and Toxicology, Pathology, Veterans Affairs Medical Center, Boonshoft School of Medicine, Wright State University, Dayton, Ohio 45435, USA
Departments of Pharmacology and Toxicology, Pathology, Veterans Affairs Medical Center, Boonshoft School of Medicine, Wright State University, Dayton, Ohio 45435, USA

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Khalid M Elased Departments of Pharmacology and Toxicology, Pathology, Veterans Affairs Medical Center, Boonshoft School of Medicine, Wright State University, Dayton, Ohio 45435, USA

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). Retrospective clinical trials validated exercise training as an important non-pharmacological strategy to prevent diabetes and obesity ( Pan et al . 1997 ). The American Diabetes Association and European Association for the Study of Diabetes recommend

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0532
Volume/Issue:
Volume 221: Issue 2
Free access