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Xiao-Bing Cui Department of Physiology and Pharmacology, Renmin Hospital, Antioxidant and Gene Regulation Laboratory, University of Georgia, 501 D.W. Brooks Drive, Athens, Georgia 30602, USA

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Jun-Na Luan Department of Physiology and Pharmacology, Renmin Hospital, Antioxidant and Gene Regulation Laboratory, University of Georgia, 501 D.W. Brooks Drive, Athens, Georgia 30602, USA

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Jianping Ye Department of Physiology and Pharmacology, Renmin Hospital, Antioxidant and Gene Regulation Laboratory, University of Georgia, 501 D.W. Brooks Drive, Athens, Georgia 30602, USA

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Shi-You Chen Department of Physiology and Pharmacology, Renmin Hospital, Antioxidant and Gene Regulation Laboratory, University of Georgia, 501 D.W. Brooks Drive, Athens, Georgia 30602, USA
Department of Physiology and Pharmacology, Renmin Hospital, Antioxidant and Gene Regulation Laboratory, University of Georgia, 501 D.W. Brooks Drive, Athens, Georgia 30602, USA

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complications. Response gene to complement 32 (RGC32) is expressed in numerous human organs and tissues including placenta, kidney, liver, heart, and brain ( Badea et al . 1998 ). Functionally, RGC32 plays an important role in cell proliferation

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0548
Volume/Issue:
Volume 224: Issue 2
Free access
Mary C Gieske Department of Clinical Sciences and Biological Sciences, University of Kentucky, Lexington, Kentucky 40506, USA
Obstetrics and Gynecology, University of Kentucky, Lexington, Kentucky, USA
School of Biotechnology and Biomedical Sciences, Inje University, Kimhae, South Korea

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Gi Youn Na Department of Clinical Sciences and Biological Sciences, University of Kentucky, Lexington, Kentucky 40506, USA
Obstetrics and Gynecology, University of Kentucky, Lexington, Kentucky, USA
School of Biotechnology and Biomedical Sciences, Inje University, Kimhae, South Korea

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Yongbum Koo Department of Clinical Sciences and Biological Sciences, University of Kentucky, Lexington, Kentucky 40506, USA
Obstetrics and Gynecology, University of Kentucky, Lexington, Kentucky, USA
School of Biotechnology and Biomedical Sciences, Inje University, Kimhae, South Korea

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Misung Jo Department of Clinical Sciences and Biological Sciences, University of Kentucky, Lexington, Kentucky 40506, USA
Obstetrics and Gynecology, University of Kentucky, Lexington, Kentucky, USA
School of Biotechnology and Biomedical Sciences, Inje University, Kimhae, South Korea

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Thomas E Curry Jr Department of Clinical Sciences and Biological Sciences, University of Kentucky, Lexington, Kentucky 40506, USA
Obstetrics and Gynecology, University of Kentucky, Lexington, Kentucky, USA
School of Biotechnology and Biomedical Sciences, Inje University, Kimhae, South Korea

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Chemyong Ko Department of Clinical Sciences and Biological Sciences, University of Kentucky, Lexington, Kentucky 40506, USA
Obstetrics and Gynecology, University of Kentucky, Lexington, Kentucky, USA
School of Biotechnology and Biomedical Sciences, Inje University, Kimhae, South Korea

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-ovulatory effects ( Richards et al. 1998 ), and null mutants for P4 receptor (PR) or the cyclooxygenase-2 gene are infertile due to anovulatory syndromes ( Richards et al. 2002 ). PR is transcribed in granulosa cells of pre-ovulatory follicles in response to LH

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06218
Volume/Issue:
Volume 186: Issue 2
Free access
Aijun Zhang Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Douglas H Sieglaff Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Jean Philippe York Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Ji Ho Suh Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Stephen D Ayers Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Glenn E Winnier Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Alexei Kharitonenkov Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Christopher Pin Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA
Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Pumin Zhang Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Paul Webb Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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Xuefeng Xia Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA
Houston Methodist Research Institute, College of Arts and Sciences, Departments of Paediatrics, Children's Health Research Institute, Department of Molecular Physiology and Biophysics, The Third Affiliated Hospital of Guangzhou Medical University, Genomic Medicine Program, 6670 Bertner Ave, Houston, Texas 77030, USA

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for interdependency of TH and FGF21 signaling pathways. TH receptors (TRα and TRβ) are members of the nuclear hormone receptor family that regulate target genes in response to the main active form of TH, triiodothyronine (T 3 ). TRs usually work by

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0440
Volume/Issue:
Volume 224: Issue 3
Free access
Lovisa Lundholm
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Milica Putnik
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Michio Otsuki Department of Biosciences and Nutrition at Novum, Department of Medicine, Division of Endocrinology, Karolinska Institutet, SE-141 57 Huddinge, Sweden

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Sandra Andersson
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Claes Ohlsson Department of Biosciences and Nutrition at Novum, Department of Medicine, Division of Endocrinology, Karolinska Institutet, SE-141 57 Huddinge, Sweden

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Jan-Åke Gustafsson
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Karin Dahlman-Wright
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. 1996 , Nilsson et al . 2001 ). The receptors function as ligand-dependent transcription factors that bind to estrogen-response elements (EREs) or, for example, in association with fos and jun to activator protein 1 (AP-1) sites in target gene

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0277
Volume/Issue:
Volume 196: Issue 3
Free access
Asmaà Fritah Institut National de la Santé et de la Recherche Médicale, U673, Hôpital Saint-Antoine, Université Pierre et Marie Curie (UPMC-Paris 6), Paris, France

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Gérard Redeuilh Institut National de la Santé et de la Recherche Médicale, U673, Hôpital Saint-Antoine, Université Pierre et Marie Curie (UPMC-Paris 6), Paris, France

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Michèle Sabbah Institut National de la Santé et de la Recherche Médicale, U673, Hôpital Saint-Antoine, Université Pierre et Marie Curie (UPMC-Paris 6), Paris, France

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sequence called the oestrogen response element (ERE), which is usually located upstream of the promoter and displays enhancer properties. For regulation of gene transcription, ER has to interact with basal transcription factors and RNA polymerase II ( Jacq

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.07009
Volume/Issue:
Volume 191: Issue 3
Free access
Sofianos Andrikopoulos Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Barbara C Fam Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Anita Holdsworth Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Sherley Visinoni Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Zheng Ruan Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Maria Stathopoulos Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Anne W Thorburn Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Christos N Joannides Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Michael Cancilla Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Lois Balmer Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia
Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Joseph Proietto Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Grant Morahan Department of Medicine (AH), Walter and Eliza Hall Institute of Medical Research, Harry Perkins Institute of Medical Research, The School of Medical Sciences Edith Cowan University, Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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secretory response in NZO transgenic mice To determine whether complementing the defective Abcc8/Kcnj11 gene with the functional B6 gene could reverse the insulin secretory defect of the NZO mice, an IVTT to glucose was performed ( Fig. 5 ). As can be seen

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-15-0290
Volume/Issue:
Volume 228: Issue 1
Free access
Rohit Singhal Physiology and Biophysics, Arkansas Children's Nutrition Center, Departments of

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Kartik Shankar Physiology and Biophysics, Arkansas Children's Nutrition Center, Departments of
Physiology and Biophysics, Arkansas Children's Nutrition Center, Departments of

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Thomas M Badger Physiology and Biophysics, Arkansas Children's Nutrition Center, Departments of
Physiology and Biophysics, Arkansas Children's Nutrition Center, Departments of

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Martin J Ronis Physiology and Biophysics, Arkansas Children's Nutrition Center, Departments of
Physiology and Biophysics, Arkansas Children's Nutrition Center, Departments of

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characterization and fold changes of hepatic genes common to estradiol (E 2 ) and soy protein isolate (SPI) Gene symbol Gene title GO biological process term E 2 -fold change SPI-fold change RefSeq NM_031048 Lifr Leukemia inhibitory factor receptor Stress response

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0059
Volume/Issue:
Volume 202: Issue 1
Free access
Stephen G Matthews Department of Physiology, University of Toronto, Toronto, Ontario, Canada
Departments of Obstetrics & Gynaecology and Medicine, University of Toronto, Toronto, Ontario, Canada
Lunenfeld Tanenbaum Research Institute, Sinai Health System, Toronto, Ontario, Canada

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Patrick O McGowan Department of Physiology, University of Toronto, Toronto, Ontario, Canada
Department of Biological Sciences and Center for Environmental Epigenetics and Development, University of Toronto, Scarborough, Ontario, Canada
Department of Cell and Systems Biology, Department of Psychology, University of Toronto, Toronto, Ontario, Canada

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research) in the field of developmental origins of health and disease. Responses to environmental cues that lead to stable alterations in HPA function may initially be adaptive but in the long term, and if not appropriate for the ongoing environment, may

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-19-0057
Volume/Issue:
Volume 242: Issue 1
Free access
Joyce Emons
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Bas E Dutilh Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Eva Decker Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Heide Pirzer Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Carsten Sticht Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Norbert Gretz Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Gudrun Rappold Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Ewan R Cameron Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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James C Neil Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Gary S Stein Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Andre J van Wijnen Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Jan Maarten Wit
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Janine N Post Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Marcel Karperien Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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analysis of these growth plate specimens complemented with a detailed microarray and bioinformatic analysis and identified 394 differentially expressed genes, some of which are representative for processes that occur during growth plate maturation. We

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-10-0219
Volume/Issue:
Volume 209: Issue 2
Free access
Clyde J Wright Section of Neonatology, Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA

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Sarah McKenna Section of Neonatology, Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA

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Robyn De Dios Section of Neonatology, Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA

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Brit H Boehmer Section of Neonatology, Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA

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Leanna Nguyen Section of Neonatology, Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA

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Sankar Ghosh Department of Microbiology and Immunology, Vagelos College of Physicians and Surgeons, Columbia University, New York, New York, USA

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Jeryl Sandoval Section of Neonatology, Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA

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Paul J Rozance Section of Neonatology, Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado, USA

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studies complement the existing literature by interrogating the effect of adding cytokine exposure (TNFα) to STZ. We found that co-exposure significantly increased expression of NFκB target genes and cell death compared to STZ or TNFα exposure alone. These

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-21-0047
Volume/Issue:
Volume 249: Issue 3
Restricted access