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Gabriela Capllonch-Amer Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain

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Miquel Sbert-Roig Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain

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Bel M Galmés-Pascual Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain

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Ana M Proenza Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain
Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain

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Isabel Lladó Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain
Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain

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Magdalena Gianotti Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain
Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain

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Francisco J García-Palmer Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain
Grup Metabolisme Energètic i Nutrició, Centro de Investigación Biomédica en Red Fisiopatología de la Obesidad y la Nutrición (CIBERobn, Departament de Biologia Fonamental i Ciències de la Salut, Institut Universitari d'Investigació en Ciències de la Salut (IUNICS), Universitat de les Illes Balears, Ctra. Valldemossa, km 7,5. E-07122 Palma de Mallorca, Illes Balears, Spain

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Introduction Sexual dimorphism in mitochondrial functionality has been described in many rat tissues such as liver, adipose tissue, brain, and skeletal muscle ( Colom et al . 2007 a , b , Valle et al . 2007 a , b , Gómez-Pérez et al . 2008

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0008
Volume/Issue:
Volume 221: Issue 3
Free access
Stephen P Ashcroft School of Sport, Exercise and Rehabilitation Sciences, University of Birmingham, Birmingham, UK
MRC-Versus Arthritis Centre for Musculoskeletal Ageing Research, Clinical, Metabolic and Molecular Physiology, University of Nottingham, Royal Derby Hospital Centre, Derby, UK
Mitochondrial Metabolism and Ageing Laboratory, Garvan Institute of Medical Research, Sydney, New South Wales, Australia

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Gareth Fletcher Mitochondrial Metabolism and Ageing Laboratory, Garvan Institute of Medical Research, Sydney, New South Wales, Australia

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Ashleigh M Philp Mitochondrial Metabolism and Ageing Laboratory, Garvan Institute of Medical Research, Sydney, New South Wales, Australia
St Vincent’s Clinical School, UNSW Medicine, UNSW Sydney, New South Wales, Australia

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Carl Jenkinson Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
ANZAC Research Institute, University of Sydney, Sydney, New South Wales, Australia

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Shatarupa Das Centenary Institute, Royal Prince Alfred Hospital, Camperdown, New South Wales, Australia
UTS Centenary Centre for Inflammation, University Technology Sydney, New South Wales, Australia

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Philip M Hansbro Centenary Institute, Royal Prince Alfred Hospital, Camperdown, New South Wales, Australia
UTS Centenary Centre for Inflammation, University Technology Sydney, New South Wales, Australia

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Philip J Atherton MRC-Versus Arthritis Centre for Musculoskeletal Ageing Research, Clinical, Metabolic and Molecular Physiology, University of Nottingham, Royal Derby Hospital Centre, Derby, UK

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Andrew Philp Mitochondrial Metabolism and Ageing Laboratory, Garvan Institute of Medical Research, Sydney, New South Wales, Australia
St Vincent’s Clinical School, UNSW Medicine, UNSW Sydney, New South Wales, Australia

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of skeletal muscle function ( Girgis et al. 2013 ). Within human populations, multiple observational studies have reported a positive association between serum 25(OH)D, skeletal muscle strength and lower extremity function in older individuals

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-20-0233
Volume/Issue:
Volume 249: Issue 2
Restricted access
Yuriko Kitajima Department of Stem Cell Biology, Atomic Bomb Disease Institute, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki, Japan

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Yusuke Ono Department of Stem Cell Biology, Atomic Bomb Disease Institute, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki, Japan

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. 2015 ). Thus, understanding the regulation of satellite cells is critical to elucidate the mechanisms in skeletal muscle maintenance. Several studies have reported that estrogens appear to favor muscle regeneration after injury ( Diel 2014 ). Estrogens

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-15-0476
Volume/Issue:
Volume 229: Issue 3
Free access
Juthamard Surapongchai Exercise Physiology Laboratory, Department of Physiology, Faculty of Science, Mahidol University, Bangkok, Thailand

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Mujalin Prasannarong Department of Physical Therapy, Faculty of Associated Medical Sciences, Chiang Mai University, Chiang Mai, Thailand

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Tepmanas Bupha-Intr Exercise Physiology Laboratory, Department of Physiology, Faculty of Science, Mahidol University, Bangkok, Thailand

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Vitoon Saengsirisuwan Exercise Physiology Laboratory, Department of Physiology, Faculty of Science, Mahidol University, Bangkok, Thailand

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Introduction Insulin resistance of skeletal muscle represents a major defect in the maintenance of euglycemia and is often accompanied by a variety of metabolic and cardiovascular abnormalities, including glucose intolerance, hyperinsulinemia

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0579
Volume/Issue:
Volume 232: Issue 3
Free access
Kok Lim Kua Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA

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Shanming Hu Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA

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Chunlin Wang Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA

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Jianrong Yao Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA

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Diana Dang Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA

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Alexander B Sawatzke Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA

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Jeffrey L Segar Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA

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Kai Wang Department of Biostatistics, College of Public Health, University of Iowa, Iowa City, Iowa, USA

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Andrew W Norris Stead Family Department of Pediatrics, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA
Department of Biochemistry, Carver College of Medicine, University of Iowa, Iowa City, Iowa, USA
Fraternal Order of Eagles Diabetes Research Center, University of Iowa, Iowa City, Iowa, USA

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hyperglycemia rapidly developed skeletal muscle insulin resistance while still in utero . The insulin signaling defects persisted through postnatal life, localized to skeletal muscle. Uteroplacental insufficiency and fetal hyperinsulinism did not have these

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0455
Volume/Issue:
Volume 242: Issue 1
Free access
J Kwakkel
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H C van Beeren
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M T Ackermans Department of Endocrinology and Metabolism, Laboratory of Endocrinology, Department of Clinical Chemistry, Academic Medical Center, University of Amsterdam, F5-165, Meibergdreef 9, 1105 AZ Amsterdam, The Netherlands

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M C Platvoet-ter Schiphorst
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E Fliers
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W M Wiersinga
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A Boelen
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prohormone thyroxine (T 4 ) into the active hormone T 3 by outer-ring deiodination. D2 is expressed in brain, pituitary, skeletal muscle, brown adipose tissue, and placenta and is present as an active dimer in the endoplasmic reticulum ( Kohrle 2000

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0118
Volume/Issue:
Volume 203: Issue 2
Free access
John-Paul Fuller-Jackson
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Belinda A Henry Metabolism, Diabetes and Obesity Program, Monash Biomedicine Discovery Institute, Department of Physiology, Monash University, Clayton, Victoria, Australia

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energy expenditure is reduced adaptive thermogenesis ( Rosenbaum et al . 2008 , Camps et al . 2015 , Henry et al . 2017 ), which occurs in both skeletal muscle and brown adipose tissue (BAT). To investigate the specific metabolic adaptations that

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-18-0090
Volume/Issue:
Volume 237: Issue 3
Free access
Ishita Bakshi Diabetes and Metabolism Division, Garvan Institute, Sydney, New South Wales, Australia

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Eurwin Suryana Diabetes and Metabolism Division, Garvan Institute, Sydney, New South Wales, Australia

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Lewin Small Diabetes and Metabolism Division, Garvan Institute, Sydney, New South Wales, Australia

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Lake-Ee Quek School of Mathematics and Statistics, University of Sydney, Charles Perkins Centre, Sydney, New South Wales, Australia

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Amanda E Brandon Diabetes and Metabolism Division, Garvan Institute, Sydney, New South Wales, Australia
Sydney Medical School, Charles Perkins Centre, University of Sydney, Sydney, New South Wales, Australia

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Nigel Turner Department of Pharmacology, School of Medical Sciences, University of New South Wales, Sydney, New South Wales, Australia

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Gregory J Cooney Diabetes and Metabolism Division, Garvan Institute, Sydney, New South Wales, Australia
Sydney Medical School, Charles Perkins Centre, University of Sydney, Sydney, New South Wales, Australia

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Introduction Reduced levels of glycolysis and glycogen synthesis are a well characterised feature of skeletal muscle in type 2 diabetes ( Bouche et al . 2004 , Abdul-Ghani & DeFronzo 2010 ). In general, the flux through glycolysis is

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-17-0555
Volume/Issue:
Volume 237: Issue 2
Free access
Nele Cielen Laboratory of Respiratory Diseases, Department of Clinical and Experimental Medicine, KULeuven, Leuven, Belgium

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Nele Heulens Laboratory of Respiratory Diseases, Department of Clinical and Experimental Medicine, KULeuven, Leuven, Belgium

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Karen Maes Laboratory of Respiratory Diseases, Department of Clinical and Experimental Medicine, KULeuven, Leuven, Belgium

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Geert Carmeliet Laboratory of Clinical and Experimental Endocrinology, Department of Clinical and Experimental Medicine, KULeuven, Leuven, Belgium

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Chantal Mathieu Laboratory of Clinical and Experimental Endocrinology, Department of Clinical and Experimental Medicine, KULeuven, Leuven, Belgium

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Wim Janssens Laboratory of Respiratory Diseases, Department of Clinical and Experimental Medicine, KULeuven, Leuven, Belgium

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Ghislaine Gayan-Ramirez Laboratory of Respiratory Diseases, Department of Clinical and Experimental Medicine, KULeuven, Leuven, Belgium

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Introduction Chronic obstructive pulmonary disease (COPD) is not only a lung disease, but also associates with several comorbidities. Among them, skeletal muscle dysfunction is of major concern, as it contributes, independently of lung

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-15-0491
Volume/Issue:
Volume 229: Issue 2
Open access
Jonathan M Mudry Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery

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Julie Massart Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery

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Ferenc L M Szekeres Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery

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Anna Krook Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery
Section for Integrative Physiology, Section for Integrative Physiology, Department of Molecular Medicine and Surgery

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investigated gene. TWIST proteins play important roles in tissue differentiation. In skeletal muscle, TWIST1 blocks myogenesis via inhibition of MYOD transactivation ( Hamamori et al . 1997 ), which may lead to myotube dedifferentiation ( Hjiantoniou et al

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0474
Volume/Issue:
Volume 224: Issue 3
Free access