The estrogen receptor α-selective agonist propyl pyrazole triol improves glucose tolerance in ob/ob mice: potential molecular mechanisms

in Journal of Endocrinology
Authors:
L Lundholm Department of Biosciences and Nutrition, Karolinska Institute, Huddinge, Sweden

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G Bryzgalova Department of Molecular Medicine and Surgery, Karolinska Institute, Karolinska University Hospital, Stockholm, Sweden

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H Gao Department of Biosciences and Nutrition, Karolinska Institute, Huddinge, Sweden

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N Portwood Department of Molecular Medicine and Surgery, Karolinska Institute, Karolinska University Hospital, Stockholm, Sweden

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S Fält Department of Biosciences and Nutrition, Karolinska Institute, Huddinge, Sweden

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K D Berndt Department of Biosciences and Nutrition, Karolinska Institute, Huddinge, Sweden

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A Dicker Department of Medicine, Karolinska Institute, Karolinska University Hospital, Huddinge, Sweden

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D Galuska Department of Molecular Medicine and Surgery, Karolinska Institute, Stockholm, Sweden

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J R Zierath Department of Molecular Medicine and Surgery, Karolinska Institute, Stockholm, Sweden

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J-Å Gustafsson Department of Biosciences and Nutrition, Karolinska Institute, Huddinge, Sweden

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S Efendic Department of Molecular Medicine and Surgery, Karolinska Institute, Karolinska University Hospital, Stockholm, Sweden

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K Dahlman-Wright Department of Biosciences and Nutrition, Karolinska Institute, Huddinge, Sweden

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A Khan Department of Molecular Medicine and Surgery, Karolinska Institute, Karolinska University Hospital, Stockholm, Sweden

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Correspondence should be addressed to L Lundholm: lovisa.lundholm@su.se
DOI:
https://doi.org/10.1530/JOE-08-0192e
Volume/Issue:
Volume 243: Issue 2
Article Type:
Correction
Page Range:
X1
Online Publication Date:
Nov 2019
Copyright:
© 2019 Society for Endocrinology 2019
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The authors and journal apologise for an error in the above paper, which appeared in volume 199 part 2, pages 275–286. The error relates to Fig. 10, given on page 283. The corrected composite figure for Fig. 10 is given in full below:

Figure 10
Figure 10

Western blot analysis of Stat3, phosphorylated Stat3 on Tyr705 (Stat3P), and glucose-6-phosphatase (G6p) protein in liver of ob/ob mice treated with vehicle or PPT for 30 days (n = 5 mice/group). (A) Immunoblots for each respective protein showing the results for individual liver samples from the vehicle-treated or PPT-treated mice. (B) Mean ± s.e.m. relative abundance for each respective protein in liver of vehicle-treated (open bar) or PPT-treated (filled bar) ob/ob mice (n = 5 mice/group). P = 0.07, **P < 0.01 for PPT-treated versus vehicle-treated ob/ob mice.

Citation: Journal of Endocrinology 243, 2; 10.1530/JOE-08-0192e

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Print ISSN:
0022-0795
Online ISSN:
1479-6805
Copyright:
© 2019 Society for Endocrinology 2019
Article Type:
Correction
Print Publication Date:
01 Nov 2019
Online Publication Date:
Nov 2019
  • Figure 10
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    Figure 10

    Western blot analysis of Stat3, phosphorylated Stat3 on Tyr705 (Stat3P), and glucose-6-phosphatase (G6p) protein in liver of ob/ob mice treated with vehicle or PPT for 30 days (n = 5 mice/group). (A) Immunoblots for each respective protein showing the results for individual liver samples from the vehicle-treated or PPT-treated mice. (B) Mean ± s.e.m. relative abundance for each respective protein in liver of vehicle-treated (open bar) or PPT-treated (filled bar) ob/ob mice (n = 5 mice/group). P = 0.07, **P < 0.01 for PPT-treated versus vehicle-treated ob/ob mice.

Figure 10

Western blot analysis of Stat3, phosphorylated Stat3 on Tyr705 (Stat3P), and glucose-6-phosphatase (G6p) protein in liver of ob/ob mice treated with vehicle or PPT for 30 days (n = 5 mice/group). (A) Immunoblots for each respective protein showing the results for individual liver samples from the vehicle-treated or PPT-treated mice. (B) Mean ± s.e.m. relative abundance for each respective protein in liver of vehicle-treated (open bar) or PPT-treated (filled bar) ob/ob mice (n = 5 mice/group). P = 0.07, **P < 0.01 for PPT-treated versus vehicle-treated ob/ob mice.